Loss of eyes in zebrafish caused by mutation of chokh/rx3. - Wikidata - wikimedia - TriplyDB

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

http://www.wikidata.org/entity/Q34250379

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Subfunctionalization of duplicated zebrafish pax6 genes by cis-regulatory divergence Eye morphogenesis and patterning of the optic vesicle Forward genetic analysis of visual behavior in zebrafish.Glucocorticoids play a key role in circadian cell cycle rhythms Xenopus mutant reveals necessity of rax for specifying the eye field which otherwise forms tissue with telencephalic and diencephalic character.Eph/Ephrin signalling maintains eye field segregation from adjacent neural plate territories during forebrain morphogenesis.Sunscreen for fish: co-option of UV light protection for camouflage.Cell-autonomous requirement for rx function in the mammalian retina and posterior pituitary.B1 SOX coordinate cell specification with patterning and morphogenesis in the early zebrafish embryo The Rx-like homeobox gene (Rx-L) is necessary for normal photoreceptor development.Regulation of photoreceptor gene expression by the retinal homeobox (Rx) gene product.Neurogenesis in the fish retina.Regulation of retinal homeobox gene transcription by cooperative activity among cis-elements.Genes and signaling networks regulated during zebrafish optic vesicle morphogenesis The cavefish genome reveals candidate genes for eye loss.Generation of mRx-Cre transgenic mouse line for efficient conditional gene deletion in early retinal progenitors.The Retinal Homeobox (Rx) gene is necessary for retinal regeneration.Mutant Prpf31 causes pre-mRNA splicing defects and rod photoreceptor cell degeneration in a zebrafish model for Retinitis pigmentosa.The visual system of zebrafish and its use to model human ocular diseases.Development of extraocular muscles requires early signals from periocular neural crest and the developing eye.Expression of FoxE4 and Rx visualizes the timing and dynamics of critical processes taking place during initial stages of vertebrate eye development.OFF ganglion cells cannot drive the optokinetic reflex in zebrafish Zebrafish: a model system for the study of eye genetics.Molecular mechanisms of optic vesicle development: complexities, ambiguities and controversies Have we achieved a unified model of photoreceptor cell fate specification in vertebrates?Properties of the Visible Light Phototaxis and UV Avoidance Behaviors in the Larval Zebrafish.Confirmation of RAX gene involvement in human anophthalmia.Eye evolution: common use and independent recruitment of genetic components.Retinoic Acid Protects and Rescues the Development of Zebrafish Embryonic Retinal Photoreceptor Cells from Exposure to Paclobutrazol Brain regionalization: of signaling centers and boundaries.The genetics of ocular disorders: insights from the zebrafish.Genetic regulation of vertebrate eye development.The ascidian pigmented sensory organs: structures and developmental programs.Watching eyes take shape.Rax is a selector gene for mediobasal hypothalamic cell types.Conserved genetic pathways associated with microphthalmia, anophthalmia, and coloboma.Development of the Vertebrate Eye and Retina.Cavefish and the basis for eye loss.A zebrafish LMO4 ortholog limits the size of the forebrain and eyes through negative regulation of six3b and rx3.Neural retina identity is specified by lens-derived BMP signals.

P2860

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P2860

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

http://www.wikidata.org/entity/Q34250379

description

2003 nî lūn-bûn

@nan

2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2003年の論文

@ja

2003年論文

@yue

2003年論文

@zh-hant

2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

@wuu

name

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@ast

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

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Loss of eyes in zebrafish caused by mutation of chokh/rx3.

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type

label

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

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Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@en

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@nl

prefLabel

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@ast

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@en

Loss of eyes in zebrafish caused by mutation of chokh/rx3.

@nl

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P1476

Loss of eyes in zebrafish caused by mutation of chokh/rx3

@en

P2093

Felix Loosli

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Herwig Baier

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Wendy Staub

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P2860

Six3 inactivation reveals its essential role for the formation and patterning of the vertebrate eye

Repressor activity of Headless/Tcf3 is essential for vertebrate head formation.

The Rx homeobox gene is essential for vertebrate eye development.

Pax6 lights-up the way for eye development.

The genetic control of eye development and its implications for the evolution of the various eye-types.

The zebrafish eye: developmental and genetic analysis.

A mutation in the Gsk3-binding domain of zebrafish Masterblind/Axin1 leads to a fate transformation of telencephalon and eyes to diencephalon

Six3 overexpression initiates the formation of ectopic retina

A radiation hybrid map of the zebrafish genome.

Retinal ganglion cell genesis requires lakritz, a Zebrafish atonal Homolog.

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9

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4

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Joachim Wittbrodt

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10590661

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