Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
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Breaking tolerance to thyroid antigens: changing concepts in thyroid autoimmunityThyroid Autoimmunity: Role of Anti-thyroid Antibodies in Thyroid and Extra-Thyroidal Diseases.Different fetal-neonatal outcomes in siblings born to a mother with Graves-Basedow disease after total thyroidectomy: a case series.Immunopathogenesis of thyroid eye disease: emerging paradigmsGraves' hyperthyroidism is antibody-mediated but is predominantly a Th1-type cytokine disease.Decrease in proportion of CD19+ CD24(hi) CD27+ B cells and impairment of their suppressive function in Graves' disease.Thyroid-stimulating autoantibodies in Graves disease preferentially recognize the free A subunit, not the thyrotropin holoreceptor.Preferential use of T-cell receptor V genes in human autoimmune thyroid disease.Autoimmune thyroid disease and pregnancy: relevance for the child.Identification of thyroid stimulating hormone receptor-specific T cells in Graves' disease thyroid using autoantigen-transfected Epstein-Barr virus-transformed B cell lines.Thyrotropin-blocking autoantibodies and thyroid-stimulating autoantibodies: potential mechanisms involved in the pendulum swinging from hypothyroidism to hyperthyroidism or vice versa.Analytical aspects of thyroid antibodies estimation.TSHR as a therapeutic target in Graves' disease.Serum 25-hydroxyvitamin D might be an independent prognostic factor for Graves disease recurrenceDendritic cells infected with adenovirus expressing the thyrotrophin receptor induce Graves' hyperthyroidism in BALB/c mice.The peripheral blood compartment in patients with Graves' disease: activated T lymphocytes and increased transitional and pre-naive mature B lymphocytes.Induction of hyperthyroidism in mice by intradermal immunization with DNA encoding the thyrotropin receptor.Adenovirus-mediated gene delivery of interleukin-10, but not transforming growth factor beta, ameliorates the induction of Graves' hyperthyroidism in BALB/c mice.Induction of hyperthyroxinemia in BALB/C but not in several other strains of mice.Low regulatory T cell and high IL-17 mRNA expression in a mouse Graves' disease model.Involvement of functional polymorphisms in the TNFA gene in the pathogenesis of autoimmune thyroid diseases and production of anti-thyrotropin receptor antibody.Distinct role of T helper Type 17 immune response for Graves' hyperthyroidism in mice with different genetic backgrounds.Clonal relationships between thyroid-stimulating hormone receptor-stimulating antibodies illustrate the effect of hypermutation on antibody function.
P2860
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P2860
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
description
1990 nî lūn-bûn
@nan
1990 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@ast
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@en
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@nl
type
label
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@ast
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@en
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@nl
prefLabel
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@ast
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@en
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@nl
P2093
P2860
P356
P1476
Thyroid-stimulating antibody activity between different immunoglobulin G subclasses.
@en
P2093
A P Weetman
C B Reimer
M E Yateman
N J Marshall
R C Williams
P2860
P304
P356
10.1172/JCI114768
P407
P577
1990-09-01T00:00:00Z