Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux. - Wikidata - wikimedia - TriplyDB

Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux.

Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux.

http://www.wikidata.org/entity/Q34289365

about

Reactive oxygen species and mitochondria: A nexus of cellular homeostasis NLRP3 inflammasome and its inhibitors: a review Initiation and perpetuation of NLRP3 inflammasome activation and assembly Inflammasome control of viral infection The intersection of cell death and inflammasome activation The nucleic acid-sensing inflammasomes Mitochondria: diversity in the regulation of the NLRP3 inflammasome.Regulation of inflammasome activation.Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3 RIG-I/MAVS and STING signaling promote gut integrity during irradiation- and immune-mediated tissue injury RNase L activates the NLRP3 inflammasome during viral infections.Discriminating self from non-self in nucleic acid sensing.Loss of macrophage fatty acid oxidation does not potentiate systemic metabolic dysfunction.Mechanism and Regulation of NLRP3 Inflammasome Activation.Inflammasomes and its importance in viral infections.Endogenous Retroelements and the Host Innate Immune Sensors.Cytokines IL-17 and IL-22 in the host response to infection.Epidermal keratinocytes sense dsRNA via the NLRP3 inflammasome, mediating interleukin (IL)-1β and IL-18 release.Conditional, Genetically Encoded, Small Molecule-Regulated Inhibition of NFκB Signaling in RPE Cells.The RIP1-RIP3 complex initiates mitochondrial fission to fuel NLRP3.Pharmacological inhibition of the NLRP3 inflammasome as a potential target for multiple sclerosis induced central neuropathic pain.The regulation of NLRP3 inflammasome expression during the development of cardiac contractile dysfunction in chronic kidney disease.Evasion of Human Neutrophil-Mediated Host Defense during Toxoplasma gondii Infection.The Emerging Role of Inflammasomes as Central Mediators in Inflammatory Bladder Pathology.Reduced NLRP3 Gene Expression Limits the IL-1β Cleavage via Inflammasome in Monocytes from Severely Injured Trauma Patients.Modulation of three key innate immune pathways for the most common retinal degenerative diseases Pattern Recognition Receptors and the Host Cell Death Molecular Machinery

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P2860

Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux.

http://www.wikidata.org/entity/Q34289365

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年学术文章

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2014年学术文章

@zh-cn

2014年学术文章

@zh-hans

2014年学术文章

@zh-my

2014年学术文章

@zh-sg

2014年學術文章

@yue

name

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@ast

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@en

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@nl

type

label

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@ast

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@en

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@nl

prefLabel

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@ast

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@en

Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@nl

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JIMMUNOL.1400582

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Journal of Immunology

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Cytosolic double-stranded RNA ...... ermeabilization and K+ efflux.

@en

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Chakrabarti Arindam

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Gabriel Núñez

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Luigi Franchi

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Raúl Muñoz-Planillo

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Shizuo Akira

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Tatjana Eigenbrod

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Ulas Ozkurede

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Yun-Gi Kim

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P2860

Immune signaling by RIG-I-like receptors

Recognition of 5' triphosphate by RIG-I helicase requires short blunt double-stranded RNA as contained in panhandle of negative-strand virus.

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

The adaptor MAVS promotes NLRP3 mitochondrial localization and inflammasome activation

Apoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells.

Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors

A role for mitochondria in NLRP3 inflammasome activation

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4214-4222

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scholarly article

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10.4049/JIMMUNOL.1400582

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8

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193

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Robert H. Silverman

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2014-09-15T00:00:00Z

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265692210

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25225670

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4185247

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