Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD. - Wikidata - wikimedia - TriplyDB

Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD.

Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD.

http://www.wikidata.org/entity/Q34290920

about

Muscle satellite cell heterogeneity and self-renewal Myocardin is sufficient for a smooth muscle-like contractile phenotype E-box- and MEF-2-independent muscle-specific expression, positive autoregulation, and cross-activation of the chicken MyoD (CMD1) promoter reveal an indirect regulatory pathway Basonuclin: a keratinocyte protein with multiple paired zinc fingers The basic helix-loop-helix transcription factor Mist1 functions as a transcriptional repressor of myoD The MyoD-inducible p204 protein overcomes the inhibition of myoblast differentiation by Id proteins.p38 and extracellular signal-regulated kinases regulate the myogenic program at multiple steps Identification and cloning of TCF-1, a T lymphocyte-specific transcription factor containing a sequence-specific HMG box The p48 DNA-binding subunit of transcription factor PTF1 is a new exocrine pancreas-specific basic helix-loop-helix protein Molecular cloning and characterization of a human PAX-7 cDNA expressed in normal and neoplastic myocytes MyoD and the regulation of myogenesis by helix-loop-helix proteins The muscle regulatory gene, Myf-6, has a biphasic pattern of expression during early mouse development Gene regulatory logic of dopamine neuron differentiation Nuclear reprogramming in heterokaryons is rapid, extensive, and bidirectional A new description of cellular quiescence Induced neuronal cells: how to make and define a neuron The ETS Factor, ETV2: a Master Regulator for Vascular Endothelial Cell Development Potential of adipose-derived stem cells in muscular regenerative therapies Direct Reprogramming-The Future of Cardiac Regeneration?Molecular roadblocks for cellular reprogramming Cardiac regeneration therapy: connections to cardiac physiology Progress and obstacles towards generating hematopoietic stem cells from pluripotent stem cells MicroRNA-mediated regulation of differentiation and trans-differentiation in stem cells Induction of pluripotency by defined factors The role of single-cell analyses in understanding cell lineage commitment The cell cycle and pluripotency Role of heat shock proteins in stem cell behavior Differential modulation of cell cycle progression distinguishes members of the myogenic regulatory factor family of transcription factors Inducing brain regeneration from within: in vivo reprogramming of endogenous somatic cells into neurons MASH1/Ascl1a leads to GAP43 expression and axon regeneration in the adult CNS p53 suppresses muscle differentiation at the myogenin step in response to genotoxic stress Target hub proteins serve as master regulators of development in yeast.Coordinating cell proliferation and differentiation: Antagonism between cell cycle regulators and cell type-specific gene expression From pluripotency to myogenesis: a multistep process in the dish SWI/SNF-directed stem cell lineage specification: dynamic composition regulates specific stages of skeletal myogenesis Stabilization of MyoD by direct binding to p57(Kip2)TGF-β family signaling in stem cells Myocardin is a bifunctional switch for smooth versus skeletal muscle differentiation Signal-dependent incorporation of MyoD-BAF60c into Brg1-based SWI/SNF chromatin-remodelling complex Skin fibroblasts from patients with type 1 diabetes (T1D) can be chemically transdifferentiated into insulin-expressing clusters: a transgene-free approach

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Activation of muscle-specific genes in pigment, nerve, fat, liver, and fibroblast cell lines by forced expression of MyoD.

http://www.wikidata.org/entity/Q34290920

description

1989 nî lūn-bûn

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1989 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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1989 թվականի հուլիսին հրատարակված գիտական հոդված

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1989年の論文

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1989年論文

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1989年論文

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1989年論文

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1989年論文

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1989年論文

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1989年论文

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Activation of muscle-specific ...... by forced expression of MyoD.

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Activation of muscle-specific ...... by forced expression of MyoD.

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Activation of muscle-specific ...... by forced expression of MyoD.

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Proceedings of the National Academy of Sciences of the United States of America

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Adam MA

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Davis RL

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Lassar AB

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Miller AD

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Tapscott SJ

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Thayer MJ

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Weintraub H

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P2860

MyoD1: a nuclear phosphoprotein requiring a Myc homology region to convert fibroblasts to myoblasts

A new DNA binding and dimerization motif in immunoglobulin enhancer binding, daughterless, MyoD, and myc proteins

A novel human muscle factor related to but distinct from MyoD1 induces myogenic conversion in 10T1/2 fibroblasts

Recombinant genomes which express chloramphenicol acetyltransferase in mammalian cells

Expression of a single transfected cDNA converts fibroblasts to myoblasts

Myogenic lineage determination and differentiation: evidence for a regulatory gene pathway.

Transfection of a DNA locus that mediates the conversion of 10T1/2 fibroblasts to myoblasts.

Multiple new phenotypes induced in 10T1/2 and 3T3 cells treated with 5-azacytidine.

Induction of muscle genes in neural cells.

The muscle creatine kinase gene is regulated by multiple upstream elements, including a muscle-specific enhancer

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