Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling. - Wikidata - wikimedia - TriplyDB

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

http://www.wikidata.org/entity/Q34291629

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Mu opioid receptors on primary afferent nav1.8 neurons contribute to opiate-induced analgesia: insight from conditional knockout mice Rapid Extraction and Identification of Maitotoxin and Ciguatoxin-Like Toxins from Caribbean and Pacific Gambierdiscus Using a New Functional Bioassay An Updated Review of Ciguatera Fish Poisoning: Clinical, Epidemiological, Environmental, and Public Health Management.Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1.Transient receptor potential (TRP) channels: a clinical perspective Ionic mechanisms of spinal neuronal cold hypersensitivity in ciguatera.Ciguatoxin reduces regenerative capacity of axotomized peripheral neurons and delays functional recovery in pre-exposed mice after peripheral nerve injury.Acute Exposure to Pacific Ciguatoxin Reduces Electroencephalogram Activity and Disrupts Neurotransmitter Metabolic Pathways in Motor Cortex.Excitation and modulation of TRPA1, TRPV1, and TRPM8 channel-expressing sensory neurons by the pruritogen chloroquine Assessment of gene expression of intracellular calcium channels, pumps and exchangers with epidermal growth factor-induced epithelial-mesenchymal transition in a breast cancer cell line.An animal model of oxaliplatin-induced cold allodynia reveals a crucial role for Nav1.6 in peripheral pain pathways Normal and abnormal coding of somatosensory stimuli causing pain.Neuropathic pruritus.The role of sodium channels in painful diabetic and idiopathic neuropathy.Molecular sensors and modulators of thermoreception.Cold Temperature Encoding by Cutaneous TRPA1 and TRPM8-Carrying Fibers in the Mouse.Ladder-Shaped Ion Channel Ligands: Current State of Knowledge.TRP channel blamed for burning cold after a tropical fish meal.Comprehensive thermal preference phenotyping in mice using a novel automated circular gradient assay.Ciguatoxins Evoke Potent CGRP Release by Activation of Voltage-Gated Sodium Channel Subtypes NaV1.9, NaV1.7 and NaV1.1.Taking a translational turn.Biotechnological and Pharmacological Applications of Biotoxins and Other Bioactive Molecules from Dinoflagellates.TRPA1 channels: molecular sentinels of cellular stress and tissue damage.Neurotoxicity and reactive astrogliosis in the anterior cingulate cortex in acute ciguatera poisoning.Sodium channels and pain: from toxins to therapies.Regulation of Pain and Itch by TRP Channels.Pain-Causing Venom Peptides: Insights into Sensory Neuron Pharmacology.Brain mechanisms of abnormal temperature perception in cold allodynia induced by ciguatoxin.Tectus niloticus (Tegulidae, Gastropod) as a Novel Vector of Ciguatera Poisoning: Clinical Characterization and Follow-Up of a Mass Poisoning Event in Nuku Hiva Island (French Polynesia).[Ciguatera: a multifaceted and puzzling disorder].

P2860

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P2860

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

http://www.wikidata.org/entity/Q34291629

description

2012 nî lūn-bûn

@nan

2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2012年の論文

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2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@ast

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@en

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@nl

type

label

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@ast

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@en

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@nl

prefLabel

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@ast

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@en

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@nl

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The EMBO Journal

P1476

Ciguatoxins activate specific cold pain pathways to elicit burning pain from cooling.

@en

P2093

Anastasia Sharov

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Andreas Hess

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John N Wood

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Marina Sergejeva

http://www.w3.org/2001/XMLSchema#string

Matthias Engel

http://www.w3.org/2001/XMLSchema#string

Mirjam Eberhardt

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Peter J Cabot

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Rachel Hinsbey

http://www.w3.org/2001/XMLSchema#string

Simon Sattler

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Viktorie Vlachová

http://www.w3.org/2001/XMLSchema#string

P2860

A genome-wide Drosophila screen for heat nociception identifies α2δ3 as an evolutionarily conserved pain gene.

Synthetic ciguatoxins selectively activate Nav1.8-derived chimeric sodium channels expressed in HEK293 cells

TRPA1 mediates the inflammatory actions of environmental irritants and proalgesic agents

The menthol receptor TRPM8 is the principal detector of environmental cold

TRPM8 is required for cold sensation in mice

ANKTM1, a TRP-like channel expressed in nociceptive neurons, is activated by cold temperatures

A TRP channel that senses cold stimuli and menthol

A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

TRPA1 contributes to cold hypersensitivity

Identification of a cold receptor reveals a general role for TRP channels in thermosensation

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3795-3808

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scholarly article

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10.1038/EMBOJ.2012.207

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19

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31

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Richard J. Lewis

Angelika Lampert

Katharina Zimmermann

Lindon S Collins

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2012-07-31T00:00:00Z

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230592066

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22850668

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3463840

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