Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion. - Wikidata - wikimedia - TriplyDB

Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.

Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.

http://www.wikidata.org/entity/Q34315819

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Expression of complement components differs between kidney allografts from living and deceased donors Alternative complement pathway deficiency ameliorates chronic smoke-induced functional and morphological ocular injury Pericytes and immune cells contribute to complement activation in tubulointerstitial fibrosis.Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17 Pathogenesis of acute kidney injury: foundation for clinical practice.Immune mechanisms and novel pharmacological therapies of acute kidney injury.Soluble thrombomodulin protects ischemic kidneys Complement regulation in renal disease models Review: Complement and its regulatory proteins in kidney diseases.Smoke exposure causes endoplasmic reticulum stress and lipid accumulation in retinal pigment epithelium through oxidative stress and complement activation.Activation of complement factor B contributes to murine and human myocardial ischemia/reperfusion injury.Amelioration of acute kidney injury in lipopolysaccharide-induced systemic inflammatory response syndrome by an aldose reductase inhibitor, fidarestat.Tissue-specific deletion of Crry from mouse proximal tubular epithelial cells increases susceptibility to renal ischemia-reperfusion injury.A comparative transcriptome analysis identifying FGF23 regulated genes in the kidney of a mouse CKD model.Defective complement inhibitory function predisposes to renal disease.Both IL-1β and TNF-α regulate NGAL expression in polymorphonuclear granulocytes of chronic hemodialysis patients.An allelic variant of Crry in the murine Sle1c lupus susceptibility interval is not impaired in its ability to regulate complement activation.B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.Binding of factor H to tubular epithelial cells limits interstitial complement activation in ischemic injury The complement inhibitors Crry and factor H are critical for preventing autologous complement activation on renal tubular epithelial cells.Loss of CD11b exacerbates murine complement-mediated tubulointerstitial nephritis.Cellular pathophysiology of ischemic acute kidney injury Complement alternative pathway activation in the autologous phase of nephrotoxic serum nephritis.Complement activation and toll-like receptor-2 signaling contribute to cytokine production after renal ischemia/reperfusion.Mammalian DNA is an endogenous danger signal that stimulates local synthesis and release of complement factor B.C3a and C5a promote renal ischemia-reperfusion injury.New concepts of complement in allorecognition and graft rejection New Insights on Complement Inhibitor CD59 in Mouse Laser-Induced Choroidal Neovascularization: Mislocalization After Injury and Targeted Delivery for Protein Replacement.Annexin A2 Enhances Complement Activation by Inhibiting Factor H Triggers of inflammation after renal ischemia/reperfusion.The immunomodulatory role of carbon monoxide during transplantation.Blocking properdin, the alternative pathway, and anaphylatoxin receptors ameliorates renal ischemia-reperfusion injury in decay-accelerating factor and CD59 double-knockout mice Aloperine protects mice against ischemia reperfusion (IR)-induced renal injury by regulating PI3K/AKT/mTOR signaling and AP-1 activity.The role of complement and Toll-like receptors in organ transplantation.Collectin-11 detects stress-induced L-fucose pattern to trigger renal epithelial injury Membrane protein Crry maintains homeostasis of the complement system.Distinct roles for the complement regulators factor H and Crry in protection of the kidney from injury.The innate immune response in ischemic acute kidney injury.The alternative complement pathway revisited.Discrimination between host and pathogens by the complement system.

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P2860

Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.

http://www.wikidata.org/entity/Q34315819

description

2006 nî lūn-bûn

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2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2006 թվականի հունվարին հրատարակված գիտական հոդված

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2006年の論文

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Altered renal tubular expressi ...... on after ischemia/reperfusion.

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Altered renal tubular expressi ...... on after ischemia/reperfusion.

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Altered renal tubular expressi ...... on after ischemia/reperfusion.

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Altered renal tubular expressi ...... on after ischemia/reperfusion.

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Journal of Clinical Investigation

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Altered renal tubular expressi ...... ion after ischemia/reperfusion

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Damian M Kraus

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Danica Ljubanović

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Gregory Proctor

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Hector Molina

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Joshua M Thurman

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Nicholas P Barry

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V Michael Holers

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P2860

Dissociation of spectrin-ankyrin complex as a basis for loss of Na-K-ATPase polarity after ischemia

Gentamicin causes endocytosis of Na/Pi cotransporter protein (NaPi-2)

Time course of complement activation and inhibitor expression after ischemic injury of rat myocardium

Rat glomerular epithelial cells produce and bear factor H on their surface that is up-regulated under complement attack

Genomic structure, functional comparison, and tissue distribution of mouse Cd59a and Cd59b

A critical role for murine complement regulator crry in fetomaternal tolerance

Expression of a soluble complement inhibitor protects transgenic mice from antibody-induced acute renal failure.

Mutations in human complement regulator, membrane cofactor protein (CD46), predispose to development of familial hemolytic uremic syndrome.

Mutations in CD46, a complement regulatory protein, predispose to atypical HUS.

Cell biology and molecular mechanisms of injury in ischemic acute renal failure.

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10.1172/JCI24521

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