B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion. - Wikidata - wikimedia - TriplyDB

B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.

B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.

http://www.wikidata.org/entity/Q35097477

about

Role of Natural Autoantibodies and Natural IgM Anti-Leucocyte Autoantibodies in Health and Disease Mesenchymal Stromal Cell Therapy in Ischemia/Reperfusion Injury The Complement System and Antibody-Mediated Transplant Rejection Inflammation in AKI: Current Understanding, Key Questions, and Knowledge Gaps.Therapeutic Targets of Human AKI: Harmonizing Human and Animal AKI Role of leukocytes in the pathogenesis of acute kidney injury Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17 Therapeutic translation in acute kidney injury: the epithelial/endothelial axis B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction.Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complement Association between the perioperative antioxidative ability of platelets and early post-transplant function of kidney allografts: a pilot study.Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.Acute Liver Injury Is Independent of B Cells or Immunoglobulin M.IgM exacerbates glomerular disease progression in complement-induced glomerulopathy Endothelial pentraxin 3 contributes to murine ischemic acute kidney injury.B Cells, Antibodies, and More.Molecular characterization of the early B cell response to pulmonary Cryptococcus neoformans infection.IgM contributes to glomerular injury in FSGS Blocking properdin, the alternative pathway, and anaphylatoxin receptors ameliorates renal ischemia-reperfusion injury in decay-accelerating factor and CD59 double-knockout mice Modulating proximal cell signaling by targeting Btk ameliorates humoral autoimmunity and end-organ disease in murine lupus.Bax and Bak have critical roles in ischemic acute kidney injury in global and proximal tubule-specific knockout mouse models.Renal ischemia-reperfusion injury amplifies the humoral immune response B1a cells play a pathogenic role in the development of autoimmune arthritis.Cyclosporine induces endothelial cell release of complement-activating microparticles.The role of the complement system in acute kidney injury.Impact of mannose-binding lectin deficiency on radiocontrast-induced renal dysfunction.The complement system in ischemia-reperfusion injuries Mesenchymal stromal cell therapy in conditions of renal ischaemia/reperfusion.Phenotype and Influx Kinetics of Leukocytes and Inflammatory Cytokine Production in Kidney Ischemia/Reperfusion Injury.Cell Therapy in Kidney Transplantation: Focus on Regulatory T Cells.Role of Natural IgM Autoantibodies (IgM-NAA) and IgM Anti-Leukocyte Antibodies (IgM-ALA) in Regulating Inflammation.Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.Complement factor H protects mice from ischemic acute kidney injury but is not critical for controlling complement activation by glomerular IgM.B Cells in Transplantation: of rat, mouse and man.

P2860

Q26745015-7ED18ED8-5529-47A4-A08C-40297DE55F44 Q26799905-8E272842-A3BB-49EC-BAB0-5321B16C6199 Q28087773-D89F086A-9DCE-4A83-A143-0AFC819EFE5A Q30278352-EAD62C7F-9E01-4558-BF7B-C241E5995AF3 Q30278440-A301E74A-2270-4FC9-BBB0-864C84460BED Q30418652-EB8FE0F5-97CC-4D73-A79C-E3E903F7DD4F Q30422418-133920D2-6C1F-401A-9E08-EFC78A816BB1 Q30583254-EDCDE28C-B33A-40CA-B2A5-F20C0CA0EA36 Q33704093-9E74A548-A262-431A-BA5A-493B140344A8 Q33814826-6F475A8B-188F-4291-844E-C5EFE2EC5AA1 Q35680459-7D0CB906-AB47-4F2E-B56A-268D465BF15C Q35685695-4B0CB7B4-CA7F-4120-BEBD-CDD82797355D Q35788209-C7D9CBD1-02A9-49C7-89CC-C5F6269F67AF Q36016859-CCF07835-5CB1-40C9-A121-0342979259B4 Q36403585-A1C4F83D-26DF-4469-8CC5-6E9F6F338C45 Q36432935-7825638A-F39F-4EF7-8D19-FF7E226D4AF4 Q36456615-4655BFB2-CFDC-4851-80D6-88E51E59FE89 Q36638347-BDEA8AE3-BBCA-4BF6-875A-4B23ED28D986 Q36718295-687EDE3E-6BB5-440C-8E14-119A55471EFA Q36907698-E37665E5-E950-41F5-8D6D-1EA949DE3A9A Q36940938-D7DCB598-FA97-4882-9049-2CFC2EC5A5C6 Q36977339-19E86867-F6D1-4FBF-AE13-F35C857B9931 Q37190057-270D8C77-430F-493D-BA69-7014D930AB88 Q37260192-1CC5672F-5E74-443F-BD89-30DF04758848 Q37275159-A3E9FF4D-D46E-4CB7-AD34-30239145798C Q37413246-8AA5DE1D-C083-4ABE-AE57-FD0B07D7219D Q38042217-DD2F6E6F-DAEF-4058-83F4-EACD95F99A45 Q38187110-F25F4533-3434-4A02-89F8-28486A8B48ED Q38815356-032F527D-6D82-4A66-A66B-5A4F9C47DD99 Q39279238-AA0C523B-CE8E-4977-A306-023146D7859B Q39429814-49703CBD-EA39-485B-A9B0-AD326F456096 Q46310923-8908E728-F07F-40D3-B0BF-725FAC017897 Q49497379-2849AC41-DA81-4F67-BAF0-980D4DB7928F Q50105925-13327E5D-6D89-4D27-8E62-CFD958539A11

P2860

B cell subsets contribute to renal injury and renal protection after ischemia/reperfusion.

http://www.wikidata.org/entity/Q35097477

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@ast

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@en

type

label

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@ast

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@en

prefLabel

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@ast

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@en

P1433

Q35097477-D76204F5-F868-4767-851A-261CC36923FA

P1476

Q35097477-D014FAEE-2B86-4366-9088-9ADC133CF29F

P2093

Q35097477-0044D2E6-3F57-4695-A871-FADAEFA1B340

Q35097477-40EE7663-8499-4E44-9D11-657D713C912C

Q35097477-57A8BEDE-4BD4-4CB0-8010-0BB4DDA3EF3A

Q35097477-63B62AEB-729D-4C55-8D6F-ACF1B0860E95

Q35097477-7A21DB5A-3C5C-4D98-88C6-7972A79C368A

Q35097477-7A365464-F0FD-4945-9956-7BDCBDDB0419

Q35097477-9CD767BE-FC44-4760-A918-B82B2A247D43

Q35097477-A3960861-0708-4785-BFB8-822C238DDC93

Q35097477-B13A4B0F-F90E-43EC-A218-D09E7B1AABF5

Q35097477-EEA6975A-0749-4BCD-9E5F-2FAD0C9CAAAA

P2860

Q35097477-07C37101-2C37-4E20-A3E5-6785ABA10837

Q35097477-0D269014-7A79-462C-A1E6-E485CEE2EF82

Q35097477-13EF402F-44DB-46F4-A3B8-2EF718CFB475

Q35097477-204D5290-4C82-49F3-9AFF-57E3ABD3653C

Q35097477-23BA9393-D719-40AC-BA15-FC970FFF8DAF

Q35097477-3F2BB9C7-2C76-4011-A646-5FE48FDD3E81

Q35097477-3F32DDA3-F9F3-45BC-896E-7A6BC7ECB0A5

Q35097477-5A0AFBCA-FA0E-4285-802E-5C220E10530F

Q35097477-651406C6-087A-48CF-8CEE-3D366570E1F3

Q35097477-69DB5B39-C026-4677-9235-BF7623EB8121

P304

Q35097477-6219F427-19D8-4A3C-B279-92EBBFF63AB1

P31

Q35097477-AF05AFB6-F763-4A09-ABB9-3581A6065FC5

P356

Q35097477-06787202-104C-4EDE-984B-4DD09D5806BF

P407

Q35097477-D4BE23AC-F718-4E61-BFBA-B7AC1C94541D

P433

Q35097477-9B94A36F-2637-4D09-BC30-82C73388AE86

P478

Q35097477-077DC997-28F2-4F4F-A749-DB1C000A2789

P577

Q35097477-CFB5545C-DA1D-466B-9EDC-D5A3AAFD785A

P5875

Q35097477-194A86CD-1D5A-463E-9A4E-82782792D096

P698

Q35097477-E66D9ECA-CEBD-4822-B6E4-9F6C2D289709

P932

Q35097477-3ED05154-8F2F-4921-8A99-391588A024C2

P356

JIMMUNOL.0903239

P1433

Journal of Immunology

P1476

B cell subsets contribute to r ...... on after ischemia/reperfusion.

@en

P2093

Brandon Renner

http://www.w3.org/2001/XMLSchema#string

Claudia R Amura

http://www.w3.org/2001/XMLSchema#string

Danica Ljubanovic

http://www.w3.org/2001/XMLSchema#string

Derek Strassheim

http://www.w3.org/2001/XMLSchema#string

Joshua M Thurman

http://www.w3.org/2001/XMLSchema#string

Kazue Takahashi

http://www.w3.org/2001/XMLSchema#string

Liudmila Kulik

http://www.w3.org/2001/XMLSchema#string

Magdalena J Glogowska

http://www.w3.org/2001/XMLSchema#string

Michael C Carroll

http://www.w3.org/2001/XMLSchema#string

V Michael Holers

http://www.w3.org/2001/XMLSchema#string

P2860

Mannose-binding lectin-deficient mice display defective apoptotic cell clearance but no autoimmune phenotype

Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity.

Targeted and restricted complement activation on acrosome-reacted spermatozoa.

Altered renal tubular expression of the complement inhibitor Crry permits complement activation after ischemia/reperfusion.

Ischemic acute renal failure: an inflammatory disease?

Triggers of inflammation after renal ischemia/reperfusion.

Regulatory B cells as inhibitors of immune responses and inflammation.

Role of CD5+ B-1 cells in EAE pathogenesis.

Predominant role for C5b-9 in renal ischemia/reperfusion injury.

Injury in renal ischemia-reperfusion is independent from immunoglobulins and T lymphocytes.

P304

4393-4400

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4049/JIMMUNOL.0903239

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

185

http://www.w3.org/2001/XMLSchema#string

P577

2010-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

46110702

http://www.w3.org/2001/XMLSchema#string

P698

20810984

http://www.w3.org/2001/XMLSchema#string

P932

3133676

http://www.w3.org/2001/XMLSchema#string