Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation. - Wikidata - wikimedia - TriplyDB

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

http://www.wikidata.org/entity/Q34321351

about

Characterization of systemic and pneumonic murine models of plague infection using a conditionally virulent strain.Pathophysiological consequences and benefits of HFE mutations: 20 years of research.Metal limitation and toxicity at the interface between host and pathogen Hepcidin as a Major Component of Renal Antibacterial Defenses against Uropathogenic Escherichia coli Q fever hepatitis and endocarditis in the context of haemochromatosis.Pulmonary Iron Homeostasis in Hepcidin Knockout Mice.Computerized image analysis of iron-stained macrophages.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

http://www.wikidata.org/entity/Q34321351

description

2012 nî lūn-bûn

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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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type

label

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

@nl

prefLabel

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

@ast

Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Hfe deficiency impairs pulmonary neutrophil recruitment in response to inflammation.

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Jens Stolte

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Karolina Benesova

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Katharina Waldow

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Maja Vujić Spasić

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Marcus A Mall

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Martina U Muckenthaler

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Sebastian M Schaefer

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Tomi Baehr-Ivacevic

http://www.w3.org/2001/XMLSchema#string

Ursula Klingmueller

http://www.w3.org/2001/XMLSchema#string

Zhe Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

Hepcidin regulates cellular iron efflux by binding to ferroportin and inducing its internalization

A novel MHC class I-like gene is mutated in patients with hereditary haemochromatosis

The C282Y mutation causing hereditary hemochromatosis does not produce a null allele

Increased airway epithelial Na+ absorption produces cystic fibrosis-like lung disease in mice

HFE gene knockout produces mouse model of hereditary hemochromatosis

Two to tango: regulation of Mammalian iron metabolism

Relative expression software tool (REST©) for group-wise comparison and statistical analysis of relative expression results in real-time PCR

Lipocalin 2 mediates an innate immune response to bacterial infection by sequestrating iron

Sequential recruitment of neutrophils into lung and bronchoalveolar lavage fluid in LPS-induced acute lung injury.

Anti-inflammatory cytokines.

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