Comparative analysis of parvalbumin and SERCA2a cardiac myocyte gene transfer in a large animal model of diastolic dysfunction. - Wikidata - wikimedia - TriplyDB

Comparative analysis of parvalbumin and SERCA2a cardiac myocyte gene transfer in a large animal model of diastolic dysfunction.

Comparative analysis of parvalbumin and SERCA2a cardiac myocyte gene transfer in a large animal model of diastolic dysfunction.

http://www.wikidata.org/entity/Q34321370

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Model-specific selection of molecular targets for heart failure gene therapy Gene therapy targets in heart failure: the path to translation Calcium mishandling in diastolic dysfunction: mechanisms and potential therapies Heart failure management: the present and the future.Noncanonical EF-hand motif strategically delays Ca2+ buffering to enhance cardiac performance Gene delivery approaches to heart failure treatment.Genetic maneuvers to ameliorate ventricular function in heart failure: therapeutic potential and future implications.Gene therapy in heart failure.Early hemodynamic and biochemical changes in overloaded swine ventricle Alterations in ryanodine receptors and related proteins in heart failure Designing heart performance by gene transfer.Gene therapy to treat cardiovascular disease.microRNA-21 promotes cardiac fibrosis and development of heart failure with preserved left ventricular ejection fraction by up-regulating Bcl-2.Molecular cardiology in translation: gene, cell and chemical-based experimental therapeutics for the failing heart.Cardiac gene therapy in large animals: bridge from bench to bedside.Gene and cytokine therapy for heart failure: molecular mechanisms in the improvement of cardiac function.Therapeutic potential of genes in cardiac repair.Gene therapy for heart disease: molecular targets, vectors and modes of delivery to myocardium.Cardiac-directed parvalbumin transgene expression in mice shows marked heart rate dependence of delayed Ca2+ buffering action.Parvalbumin isoforms differentially accelerate cardiac myocyte relaxation kinetics in an animal model of diastolic dysfunction.β-adrenergic receptor blockade reduces endoplasmic reticulum stress and normalizes calcium handling in a coronary embolization model of heart failure in canines.

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P2860

Comparative analysis of parvalbumin and SERCA2a cardiac myocyte gene transfer in a large animal model of diastolic dysfunction.

http://www.wikidata.org/entity/Q34321370

description

2004 nî lūn-bûn

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2004 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2004年の論文

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2004年論文

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2004年論文

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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AJPHEART.01137.2003

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American Journal of Physiology - Heart and Circulatory Physiology

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Comparative analysis of parval ...... odel of diastolic dysfunction.

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Andrea R Borton

http://www.w3.org/2001/XMLSchema#string

Faris P Albayya

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Jennifer C Hirsch

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Joseph M Metzger

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Mark W Russell

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Richard G Ohye

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P2860

Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a.

Cardiac excitation-contraction coupling

Targeted ablation of the phospholamban gene is associated with markedly enhanced myocardial contractility and loss of beta-agonist stimulation

The effect of isoproterenol on phospholamban-deficient mouse hearts with altered thyroid conditions

Targeted overexpression of the sarcoplasmic reticulum Ca2+-ATPase increases cardiac contractility in transgenic mouse hearts

In vivo acceleration of heart relaxation performance by parvalbumin gene delivery

Interactions between phospholamban and beta-adrenergic drive may lead to cardiomyopathy and early mortality.

Signaling pathways for cardiac hypertrophy and failure.

Prospects for gene therapy for heart failure.

Targeting phospholamban by gene transfer in human heart failure.

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10.1152/AJPHEART.01137.2003

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6

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