Morphine modulation of thrombospondin levels in astrocytes and its implications for neurite outgrowth and synapse formation - Wikidata - wikimedia - TriplyDB

Morphine modulation of thrombospondin levels in astrocytes and its implications for neurite outgrowth and synapse formation

Morphine modulation of thrombospondin levels in astrocytes and its implications for neurite outgrowth and synapse formation

http://www.wikidata.org/entity/Q34352249

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Morphine enhances HIV-1SF162-mediated neuron death and delays recovery of injured neurites Predicting Neuroinflammation in Morphine Tolerance for Tolerance Therapy from Immunostaining Images of Rat Spinal Cord Astrocyte-induced synaptogenesis is mediated by transforming growth factor β signaling through modulation of D-serine levels in cerebral cortex neurons Astrocytic transforming growth factor-beta signaling reduces subacute neuroinflammation after stroke in mice Opiate drug use and the pathophysiology of neuroAIDS.Molecular mechanisms of opioid receptor-dependent signaling and behavior.Astrocyte-secreted thrombospondin-1 modulates synapse and spine defects in the fragile X mouse model.Postnatal activation of TLR4 in astrocytes promotes excitatory synaptogenesis in hippocampal neurons.Thrombospondins and synaptogenesis GSK3β-activation is a point of convergence for HIV-1 and opiate-mediated interactive neurotoxicity Acute and chronic mu opioids differentially regulate thrombospondins 1 and 2 isoforms in astrocytes.Spinal Astrocytic Thrombospondin-4 Induced by Excitatory Neuronal Signaling Mediates Pain After Facet Capsule Injury.Thrombospondin 1 promotes synaptic formation in bone marrow-derived neuron-like cells.Thrombospondin-1 modified bone marrow mesenchymal stem cells (BMSCs) promote neurite outgrowth and functional recovery in rats with spinal cord injury.The Regulatory Effects of Transforming Growth Factor-β on Nerve Regeneration.Morphine-Mediated Brain Region-Specific Astrocytosis Involves the ER Stress-Autophagy Axis.Opiate Drugs with Abuse Liability Hijack the Endogenous Opioid System to Disrupt Neuronal and Glial Maturation in the Central Nervous System.Thrombospondin-1 secreted by human umbilical cord blood-derived mesenchymal stem cells rescues neurons from synaptic dysfunction in Alzheimer's disease model.

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Morphine modulation of thrombospondin levels in astrocytes and its implications for neurite outgrowth and synapse formation

http://www.wikidata.org/entity/Q34352249

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Morphine modulation of thrombo ...... utgrowth and synapse formation

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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JBC.M110.109827

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Journal of Biological Chemistry

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Morphine modulation of thrombo ...... utgrowth and synapse formation

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Akbar M Siddiqui

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Alexi Kiss

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Andrew L Leinweber

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Carmine J Coscia

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Hiroko Ikeda

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James D Churchill

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Kiyoshi Yamada

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Kuniyasu Ochiai

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Mariana M Belcheva

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Maureen J Donlin

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P2860

Synaptic plasticity and addiction

Mu and kappa opioid receptors activate ERK/MAPK via different protein kinase C isoforms and secondary messengers in astrocytes

Astrocytes promote myelination in response to electrical impulses

Mu-opioid receptors modulate the stability of dendritic spines

Physiological astrocytic calcium levels stimulate glutamate release to modulate adjacent neurons

Kappa opioids promote the proliferation of astrocytes via Gbetagamma and beta-arrestin 2-dependent MAPK-mediated pathways

Mitogenic signaling via endogenous kappa-opioid receptors in C6 glioma cells: evidence for the involvement of protein kinase C and the mitogen-activated protein kinase signaling cascade

Sciatic nerve ligation-induced proliferation of spinal cord astrocytes is mediated by kappa opioid activation of p38 mitogen-activated protein kinase

Kappa opioid receptor activation of p38 MAPK is GRK3- and arrestin-dependent in neurons and astrocytes

A new mathematical model for relative quantification in real-time RT-PCR

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38415-38427

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10.1074/JBC.M110.109827

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49

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285

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Lubov A Ezerskiy

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2010-10-02T00:00:00Z

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20889977

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