The sequence complementarity between HIV-1 5' splice site SD4 and U1 snRNA determines the steady-state level of an unstable env pre-mRNA.
about
Human SAP18 mediates assembly of a splicing regulatory multiprotein complex via its ubiquitin-like foldA bidirectional SF2/ASF- and SRp40-dependent splicing enhancer regulates human immunodeficiency virus type 1 rev, env, vpu, and nef gene expressionSplicing of human immunodeficiency virus RNA is position-dependent suggesting sequential removal of introns from the 5' endExtended base pair complementarity between U1 snRNA and the 5' splice site does not inhibit splicing in higher eukaryotes, but rather increases 5' splice site recognitionA suboptimal 5' splice site downstream of HIV-1 splice site A1 is required for unspliced viral mRNA accumulation and efficient virus replicationThe hepatitis B virus PRE contains a splicing regulatory element.A targeted bypass screen identifies Ynl187p, Prp42p, Snu71p, and Cbp80p for stable U1 snRNP/Pre-mRNA interaction.Evolutionary conservation of a molecular machinery for export and expression of mRNAs with retained intronsSerine- and arginine-rich proteins 55 and 75 (SRp55 and SRp75) induce production of HIV-1 vpr mRNA by inhibiting the 5'-splice site of exon 3A rev1-vpu polymorphism unique to HIV-1 subtype A and C strains impairs envelope glycoprotein expression from rev-vpu-env cassettes and reduces virion infectivity in pseudotyping assays.The transcriptional transactivator Tat selectively regulates viral splicing.SC35 and heterogeneous nuclear ribonucleoprotein A/B proteins bind to a juxtaposed exonic splicing enhancer/exonic splicing silencer element to regulate HIV-1 tat exon 2 splicing.Correct mRNA processing at a mutant TT splice donor in FANCC ameliorates the clinical phenotype in patients and is enhanced by delivery of suppressor U1 snRNAs.Genomic HEXploring allows landscaping of novel potential splicing regulatory elements.Analysis of the role of Caenorhabditis elegans GC-AG introns in regulated splicing.Excessive RNA splicing and inhibition of HIV-1 replication induced by modified U1 small nuclear RNAs.Regulation of alternative RNA splicing by exon definition and exon sequences in viral and mammalian gene expressionThe retrovirus RNA trafficking granule: from birth to maturity.The strength of the HIV-1 3' splice sites affects Rev function.Balanced splicing at the Tat-specific HIV-1 3'ss A3 is critical for HIV-1 replicationPosition-dependent splicing activation and repression by SR and hnRNP proteins rely on common mechanisms.HIV-1 infection induces changes in expression of cellular splicing factors that regulate alternative viral splicing and virus production in macrophages.Negative and positive mRNA splicing elements act competitively to regulate human immunodeficiency virus type 1 vif gene expression.Tra2-mediated recognition of HIV-1 5' splice site D3 as a key factor in the processing of vpr mRNAHuman immunodeficiency virus type 1 hnRNP A/B-dependent exonic splicing silencer ESSV antagonizes binding of U2AF65 to viral polypyrimidine tracts.Characterization of novel inhibitors of HIV-1 replication that function via alteration of viral RNA processing and rev functionHIV-1 Vpr N-terminal tagging affects alternative splicing of the viral genome.Alternative splicing: regulation of HIV-1 multiplication as a target for therapeutic action.Analysis of Competing HIV-1 Splice Donor Sites Uncovers a Tight Cluster of Splicing Regulatory Elements within Exon 2/2b.A functional conserved intronic G run in HIV-1 intron 3 is critical to counteract APOBEC3G-mediated host restriction.Succession of splicing regulatory elements determines cryptic 5΄ss functionality.Frequent gain and loss of intronic splicing regulatory elements during the evolution of vertebrates.An intronic G run within HIV-1 intron 2 is critical for splicing regulation of vif mRNA.The hnRNP A1 protein regulates HIV-1 tat splicing via a novel intron silencer element.Limited complementarity between U1 snRNA and a retroviral 5' splice site permits its attenuation via RNA secondary structure.In vitro and in silico analysis reveals an efficient algorithm to predict the splicing consequences of mutations at the 5' splice sites.Modulating role of RNA structure in alternative splicing of a critical exon in the spinal muscular atrophy genes.A novel splice donor site in the gag-pol gene is required for HIV-1 RNA stability.A novel approach to describe a U1 snRNA binding site.A purine-rich element in foamy virus pol regulates env splicing and gag/pol expression.
P2860
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P2860
The sequence complementarity between HIV-1 5' splice site SD4 and U1 snRNA determines the steady-state level of an unstable env pre-mRNA.
description
2001 nî lūn-bûn
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2001 թուականի Մարտին հրատարակուած գիտական յօդուած
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2001 թվականի մարտին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
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name
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@ast
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@en
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@nl
type
label
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@ast
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@en
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@nl
prefLabel
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@ast
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@en
The sequence complementarity b ...... l of an unstable env pre-mRNA.
@nl
P2093
P2860
P1433
P1476
The sequence complementarity b ...... el of an unstable env pre-mRNA
@en
P2093
J Krummheuer
P2860
P304
P356
10.1017/S1355838201001212
P407
P50
P577
2001-03-01T00:00:00Z