Increased brain monocarboxylic acid transport and utilization in type 1 diabetes - Wikidata - wikimedia - TriplyDB

Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

http://www.wikidata.org/entity/Q34370777

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The Contribution of Blood Lactate to Brain Energy Metabolism in Humans Measured by Dynamic 13C Nuclear Magnetic Resonance Spectroscopy Acetate transport and utilization in the rat brain.Brain fuel metabolism, aging, and Alzheimer's disease Energy Metabolism of the Brain, Including the Cooperation between Astrocytes and Neurons, Especially in the Context of Glycogen Metabolism Brain glucose metabolism during hypoglycemia in type 1 diabetes: insights from functional and metabolic neuroimaging studies Metabolic Alterations Associated to Brain Dysfunction in Diabetes Influence of VMH fuel sensing on hypoglycemic responses Diabetes, insulin-mediated glucose metabolism and Sertoli/blood-testis barrier function Receptor tyrosine kinase (RTK) mediated tyrosine phosphor-proteome from Drosophila S2 (ErbB1) cells reveals novel signaling networks Changes in human brain glutamate concentration during hypoglycemia: insights into cerebral adaptations in hypoglycemia-associated autonomic failure in type 1 diabetes Optimized [1-(13)C]glucose infusion protocol for 13C magnetic resonance spectroscopy at 3T of human brain glucose metabolism under euglycemic and hypoglycemic conditions Recurrent moderate hypoglycemia ameliorates brain damage and cognitive dysfunction induced by severe hypoglycemia.13C MRS studies of neuroenergetics and neurotransmitter cycling in humans Neuroendocrine responses to hypoglycemia.Alterations in function and expression of ABC transporters at blood-brain barrier under diabetes and the clinical significances.Differential energetic response of brain vs. skeletal muscle upon glycemic variations in healthy humans.Windowed stochastic proton decoupling for in vivo (13)C magnetic resonance spectroscopy with reduced RF power deposition.Brain activation during working memory is altered in patients with type 1 diabetes during hypoglycemia.Increased brain transport and metabolism of acetate in hypoglycemia unawareness Brain glycogen content and metabolism in subjects with type 1 diabetes and hypoglycemia unawareness.Hypoglycemia in type 1 diabetes.Effect of acute hypoglycemia on human cerebral glucose metabolism measured by ¹³C magnetic resonance spectroscopy Increased GABAergic output in the ventromedial hypothalamus contributes to impaired hypoglycemic counterregulation in diabetic rats Cortical substrate oxidation during hyperketonemia in the fasted anesthetized rat in vivo.In vivo detection of 13C isotopomer turnover in the human brain by sequential infusion of 13C labeled substrates Patients with type 1 diabetes exhibit altered cerebral metabolism during hypoglycemia.Defective counterregulation and hypoglycemia unawareness in diabetes: mechanisms and emerging treatments.Increased brain uptake and oxidation of acetate in heavy drinkers.Bringing light to the dark side of insulin: a journey across the blood-brain barrier.The mechanisms that underlie glucose sensing during hypoglycaemia in diabetes.Increased brain lactate concentrations without increased lactate oxidation during hypoglycemia in type 1 diabetic individuals.Medium-chain fatty acids improve cognitive function in intensively treated type 1 diabetic patients and support in vitro synaptic transmission during acute hypoglycemia.Recurrent antecedent hypoglycemia alters neuronal oxidative metabolism in vivo.Lactate-induced release of GABA in the ventromedial hypothalamus contributes to counterregulatory failure in recurrent hypoglycemia and diabetes.Effects of hypoglycaemia on neuronal metabolism in the adult brain: role of alternative substrates to glucose.Glycemic extremes in youth with T1DM: the structural and functional integrity of the developing brain.The Janus face of PAMAM dendrimers used to potentially cure nonenzymatic modifications of biomacromolecules in metabolic disorders-a critical review of the pros and cons.Effects of diabetes on brain metabolism--is brain glycogen a significant player?Monocarboxylate transporters: new players in body weight regulation.Non-linear actions of physiological agents: Finite disarrangements elicit fitness benefits.

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

http://www.wikidata.org/entity/Q34370777

description

2006 nî lūn-bûn

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2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2006 թվականի ապրիլին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Increased brain monocarboxylic acid transport and utilization in type 1 diabetes

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Douglas L Rothman

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Graeme F Mason

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Vincent Lebon

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P2860

Brain metabolism during fasting

In vivo 1H-[13C]-NMR spectroscopy of cerebral metabolism.

Astroglial contribution to brain energy metabolism in humans revealed by 13C nuclear magnetic resonance spectroscopy: elucidation of the dominant pathway for neurotransmitter glutamate repletion and measurement of astrocytic oxidative metabolism

Alterations in brain glucose uptake and hypoglycaemia unawareness.

Hyperglycemia and blood-brain barrier glucose transport.

Determination of the rate of the glutamate/glutamine cycle in the human brain by in vivo 13C NMR.

Banting Lecture. Hypoglycemia: the limiting factor in the management of IDDM.

Brain glucose uptake and unawareness of hypoglycemia in patients with insulin-dependent diabetes mellitus.

Regulation of glucose and ketone-body metabolism in brain of anaesthetized rats.

Ketone-body utilization by adult and suckling rat brain in vivo.

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type-1 diabetes

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