Multiple oncogenic mutations and clonal relationship in spatially distinct benign human epidermal tumors - Wikidata - wikimedia - TriplyDB

Multiple oncogenic mutations and clonal relationship in spatially distinct benign human epidermal tumors

Multiple oncogenic mutations and clonal relationship in spatially distinct benign human epidermal tumors

http://www.wikidata.org/entity/Q34375736

about

Field cancerisation in colorectal cancer: a new frontier or pastures past?High frequency of the X-chromosome inactivation in young female patients with high-grade glioma Early dissemination seeds metastasis in breast cancer.Genetic alterations in seborrheic keratoses.Systemic epidermal nevus with involvement of the oral mucosa due to FGFR3 mutation.Field cancerization in the intestinal epithelium of patients with Crohn's ileocolitis.Are oncogenes sufficient to cause human cancer?Cooperative effects of Akt-1 and Raf-1 on the induction of cellular senescence in doxorubicin or tamoxifen treated breast cancer cells.Mosaic RASopathies.The Conundrum of Genetic "Drivers" in Benign Conditions.Field cancerization in the GI tract.MiRNA-34 intrinsically links p53 tumor suppressor and Wnt signaling.Modeling tumor progression by the sequential introduction of genetic alterations into the genome of human normal cells.Activating FGFR3 mutations cause mild hyperplasia in human skin, but are insufficient to drive benign or malignant skin tumors.Circulating Tumor Cells: Who is the Killer?Squamous cell carcinomas in situ arising in seborrheic keratoses: an association with concomitant immunosuppression?Phosphorylated tubulin adaptor protein CRMP-2 as prognostic marker and candidate therapeutic target for NSCLC.Microarray analysis reveals increased expression of ΔNp63α in seborrhoeic keratosis.Genetic alterations driving metastatic colony formation are acquired outside of the primary tumour in melanoma.Somatic mosaicism: on the road to cancer.[Genetic basis of seborrheic keratosis and epidermal nevi].The categories of cutaneous mosaicism: A proposed classification.Postzygotic HRAS and KRAS mutations cause nevus sebaceous and Schimmelpenning syndrome.FGFR3, PIK3CA and RAS mutations in benign lichenoid keratosis.Clinical and Biological Characterization of Skin Pigmentation Diversity and Its Consequences on UV Impact Selection and adaptation during metastatic cancer progression

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Multiple oncogenic mutations and clonal relationship in spatially distinct benign human epidermal tumors

http://www.wikidata.org/entity/Q34375736

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Proceedings of the National Academy of Sciences of the United States of America

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Multiple oncogenic mutations a ...... benign human epidermal tumors

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Agustí Toll

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Alejandro Fernández-Casado

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Arndt Hartmann

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Christian Hafner

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Francesco Acquadro

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Juan C Cigudosa

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Julie E Burns

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Julie Earl

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Marinela Méndez-Pertuz

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Michael Landthaler

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P2860

Cellular signaling by fibroblast growth factor receptors

Amino acid substitution matrices from protein blocks

Core signaling pathways in human pancreatic cancers revealed by global genomic analyses

An integrated genomic analysis of human glioblastoma multiforme

A transforming mutation in the pleckstrin homology domain of AKT1 in cancer

A germline JAK2 SNP is associated with predisposition to the development of JAK2(V617F)-positive myeloproliferative neoplasms

The genomic landscapes of human breast and colorectal cancers

Epidermal growth factor receptor activation in glioblastoma through novel missense mutations in the extracellular domain

The molecular and genetic basis of fibroblast growth factor receptor 3 disorders: the achondroplasia family of skeletal dysplasias, Muenke craniosynostosis, and Crouzon syndrome with acanthosis nigricans

Frequent translocation t(4;14)(p16.3;q32.3) in multiple myeloma is associated with increased expression and activating mutations of fibroblast growth factor receptor 3

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20780-20785

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10.1073/PNAS.1008365107

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48

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107

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Miriam Marqués

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Francisco X Real

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2010-11-15T00:00:00Z

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47794349

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