Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling - Wikidata - wikimedia - TriplyDB

Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

http://www.wikidata.org/entity/Q34387254

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CB2 Cannabinoid Receptor As Potential Target against Alzheimer's Disease cAMP/PKA-CREB-BDNF signaling pathway in hippocampus mediates cyclooxygenase 2-induced learning/memory deficits of rats subjected to chronic unpredictable mild stress.Endocannabinoids in synaptic plasticity and neuroprotection.Synaptic and cognitive improvements by inhibition of 2-AG metabolism are through upregulation of microRNA-188-3p in a mouse model of Alzheimer's disease.Cannabinoid receptor CB2 is involved in tetrahydrocannabinol-induced anti-inflammation against lipopolysaccharide in MG-63 cells Inhibition of monoacylglycerol lipase prevents chronic traumatic encephalopathy-like neuropathology in a mouse model of repetitive mild closed head injury.Subclinical depressive symptoms and continued cannabis use: predictors of negative outcomes in first episode psychosis 4'-O-methylhonokiol increases levels of 2-arachidonoyl glycerol in mouse brain via selective inhibition of its COX-2-mediated oxygenation.Fine-tuning of synaptic upscaling at excitatory synapses by endocannabinoid signaling is mediated via the CB1 receptor.Wnt-5a-regulated miR-101b controls COX2 expression in hippocampal neurons Endocannabinoids in cerebrovascular regulation.Hydrogel scaffolds promote neural gene expression and structural reorganization in human astrocyte cultures.Cannabinoids for treatment of Alzheimer's disease: moving toward the clinic.Behavioral sequelae of astrocyte dysfunction: focus on animal models of schizophrenia.Cannabinoids in Neurodegenerative Disorders and Stroke/Brain Trauma: From Preclinical Models to Clinical Applications.Pros and Cons of Medical Cannabis use by People with Chronic Brain Disorders.Polyunsaturated fatty acids and endocannabinoids in health and disease.Lipidomics profile of a NAPE-PLD KO mouse provides evidence of a broader role of this enzyme in lipid metabolism in the brain.Effect of norbinaltorphimine on ∆⁹-tetrahydrocannabinol (THC)-induced taste avoidance in adolescent and adult Sprague-Dawley rats.NF-κB-regulated microRNA-574-5p underlies synaptic and cognitive impairment in response to atmospheric PM2.5 aspiration.Broad impact of deleting endogenous cannabinoid hydrolyzing enzymes and the CB1 cannabinoid receptor on the endogenous cannabinoid-related lipidome in eight regions of the mouse brain.Clinical studies and anti-inflammatory mechanisms of treatments.System-specific activity in response to Δ9 -tetrahydrocannabinol: a functional magnetic resonance imaging study in awake male rats.The endocannabinoid system in guarding against fear, anxiety and stress.THC Treatment Alters Glutamate Receptor Gene Expression in Human Stem Cell-Derived Neurons.Alleviation of Neuropathology by Inhibition of Monoacylglycerol Lipase in APP Transgenic Mice Lacking CB2 Receptors.Chronic activation of CB2 cannabinoid receptors in the hippocampus increases excitatory synaptic transmission.Δ9-Tetrahydrocannabinol changes the brain lipidome and transcriptome differentially in the adolescent and the adult.Delineating the Efficacy of a Cannabis-Based Medicine at Advanced Stages of Dementia in a Murine Model.Memory: a boost for medical marijuana?Therapeutic endocannabinoid augmentation for mood and anxiety disorders: comparative profiling of FAAH, MAGL and dual inhibitors.Alantolactone plays neuroprotective roles in traumatic brain injury in rats via anti-inflammatory, anti-oxidative and anti-apoptosis pathways.Studies on Prostaglandin-Endoperoxide Synthase 1: Lower Levels in Schizophrenia and After Treatment with Antipsychotic Drugs in Conjunction with Aspirin.Review of the neurological benefits of phytocannabinoids.Uncovering the psychoactivity of a cannabinoid from liverworts associated with a legal high Painkiller kills the bad effects of pot

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

http://www.wikidata.org/entity/Q34387254

description

2013 nî lūn-bûn

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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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type

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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prefLabel

Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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J.CELL.2013.10.042

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Δ9-THC-caused synaptic and memory impairments are mediated through COX-2 signaling

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Chu Chen

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Hao Sun

http://www.w3.org/2001/XMLSchema#string

Hongwei Yang

http://www.w3.org/2001/XMLSchema#string

Jian Zhang

http://www.w3.org/2001/XMLSchema#string

Ni Fan

http://www.w3.org/2001/XMLSchema#string

Rongqing Chen

http://www.w3.org/2001/XMLSchema#string

Ya-Ping Tang

http://www.w3.org/2001/XMLSchema#string

Yan Wu

http://www.w3.org/2001/XMLSchema#string

Yunping Song

http://www.w3.org/2001/XMLSchema#string

P2860

Reduced expression of glutamate receptors and phosphorylation of CREB are responsible for in vivo Delta9-THC exposure-impaired hippocampal synaptic plasticity

Alzheimer's disease; taking the edge off with cannabinoids?

CB1 cannabinoid receptors and on-demand defense against excitotoxicity

The cannabinoid receptor agonist WIN 55,212-2 regulates glutamate transmission in rat cerebral cortex: an in vivo and in vitro study

Endocannabinoids modulate N-type calcium channels and G-protein-coupled inwardly rectifying potassium channels via CB1 cannabinoid receptors heterologously expressed in mammalian neurons

Cannabinoid modulation of hippocampal long-term memory is mediated by mTOR signaling

International Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid receptors and their ligands: beyond CB₁ and CB₂

Genetic dissection of behavioural and autonomic effects of Delta(9)-tetrahydrocannabinol in mice.

Addicting drugs utilize a synergistic molecular mechanism in common requiring adenosine and Gi-beta gamma dimers

COX-2, but not COX-1, activity is necessary for the induction of perforant path long-term potentiation and spatial learning in vivo

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