Distinct poly(I-C) and virus-activated signaling pathways leading to interferon-beta production in hepatocytes.
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Hepatitis B virus polymerase blocks pattern recognition receptor signaling via interaction with DDX3: implications for immune evasionHepatitis C virus NS2 protease inhibits host cell antiviral response by inhibiting IKKε and TBK1 functionsAntiviral activities of ISG20 in positive-strand RNA virus infectionsReplication of subgenomic hepatitis C virus replicons in mouse fibroblasts is facilitated by deletion of interferon regulatory factor 3 and expression of liver-specific microRNA 122A role for the human nucleotide-binding domain, leucine-rich repeat-containing family member NLRC5 in antiviral responsesGB virus B disrupts RIG-I signaling by NS3/4A-mediated cleavage of the adaptor protein MAVSImmune Activation in the Liver by Nucleic AcidsNatural Products as Tools for Defining How Cellular Metabolism Influences Cellular Immune and Inflammatory Function during Chronic InfectionToll-like receptors in antiviral innate immunityInnate immune responses in hepatitis C virus infectionToll like receptor 3 plays a critical role in the progression and severity of acetaminophen-induced hepatotoxicityTargeted induction of interferon-λ in humanized chimeric mouse liver abrogates hepatotropic virus infectionVirus-induced type I IFN stimulates generation of immunoproteasomes at the site of infectionHepatitis C Virus Replication in Transfected and Serum-Infected Cultured Human Fetal HepatocytesDisruption of innate immunity due to mitochondrial targeting of a picornaviral protease precursorUbiquitination and proteasomal degradation of interferon regulatory factor-3 induced by Npro from a cytopathic bovine viral diarrhea virusHepatitis C Virus Infection Induces the Beta Interferon Signaling Pathway in Immortalized Human HepatocytesAcute hepatitis C virus infection: A chronic problemHepatitis C and Innate Immunity: Recent AdvancesToll-Like Receptor 3 Mediates Establishment of an Antiviral State against Hepatitis C Virus in Hepatoma CellsAntiviral activity and host gene induction by tamarin and marmoset interferon-α and interferon-γ in the GBV-B primary hepatocyte culture modelThe regulatory domain of the RIG-I family ATPase LGP2 senses double-stranded RNARO 90-7501 enhances TLR3 and RLR agonist induced antiviral responseThe ubiquitin-like protein PLIC-1 or ubiquilin 1 inhibits TLR3-Trif signalingIdentification of Adjuvantic Activity of Amphotericin B in a Novel, Multiplexed, Poly-TLR/NLR High-Throughput Screen.Differential utilization of NF-kappaB RELA and RELB in response to extracellular versus intracellular polyIC stimulation in HT1080 cells.Poly I:C enhances cycloheximide-induced apoptosis of tumor cells through TLR3 pathwayDifferential in vitro effects of intravenous versus oral formulations of silibinin on the HCV life cycle and inflammation.Andes virus nucleocapsid protein interrupts protein kinase R dimerization to counteract host interference in viral protein synthesis.Networks inferred from biochemical data reveal profound differences in toll-like receptor and inflammatory signaling between normal and transformed hepatocytes.Hepatitis C Virus Indirectly Disrupts DNA Damage-Induced p53 Responses by Activating Protein Kinase R.Viral induction of the zinc finger antiviral protein is IRF3-dependent but NF-kappaB-independent.RIG-I, MDA5 and TLR3 synergistically play an important role in restriction of dengue virus infection.Deubiquitinating and interferon antagonism activities of coronavirus papain-like proteases.Human neuronal cells possess functional cytoplasmic and TLR-mediated innate immune pathways influenced by phosphatidylinositol-3 kinase signaling.Interferon lambda alleles predict innate antiviral immune responses and hepatitis C virus permissivenessCharacterization of HCV interactions with Toll-like receptors and RIG-I in liver cells.Inhibition of hepatitis B virus replication by MyD88 involves accelerated degradation of pregenomic RNA and nuclear retention of pre-S/S RNAs.Induction and evasion of innate antiviral responses by hepatitis C virus.Disruption of TLR3 signaling due to cleavage of TRIF by the hepatitis A virus protease-polymerase processing intermediate, 3CD.
P2860
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P2860
Distinct poly(I-C) and virus-activated signaling pathways leading to interferon-beta production in hepatocytes.
description
2005 nî lūn-bûn
@nan
2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Distinct poly
@nl
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@ast
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@en
type
label
Distinct poly
@nl
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@ast
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@en
prefLabel
Distinct poly
@nl
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@ast
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@en
P2093
P356
P1476
Distinct poly(I-C) and virus-a ...... eta production in hepatocytes.
@en
P2093
Nobuyuki Kato
Stanley M Lemon
Zihong Chen
P304
16739-16747
P356
10.1074/JBC.M414139200
P407
P50
P577
2005-02-28T00:00:00Z