Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin.

Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin.

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http://www.wikidata.org/entity/Q34401965
Induction of cellular and molecular immunomodulatory pathways by vitamin A and flavonoidsApigenin Attenuates Adriamycin-Induced Cardiomyocyte Apoptosis via the PI3K/AKT/mTOR Pathway.Quercetin-induced apoptosis prevents EBV infection.Retinoid derivative Tp80 exhibits anti-hepatitis C virus activity through restoration of GI-GPx expression.Y-Box Binding Protein 1 Stabilizes Hepatitis C Virus NS5A via Phosphorylation-Mediated Interaction with NS5A To Regulate Viral Propagation.Prenatal caffeine exposure induces a poor quality of articular cartilage in male adult offspring rats via cholesterol accumulation in cartilage.An extract from Taxodium distichum targets hemagglutinin- and neuraminidase-related activities of influenza virus in vitro.GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.The acetyl-CoA carboxylase enzyme: a target for cancer therapy?A comprehensive overview of hepatoprotective natural compounds: mechanism of action and clinical perspectives.The role of PTEN - HCV core interaction in hepatitis C virus replication.Anti-Cancer Effect of Quercetin in Xenograft Models with EBV-Associated Human Gastric Carcinoma.Application of recombinant Pediococcus acidilactici BD16 (fcs⁺/ech⁺) in malolactic fermentation.Quercetin ameliorates dysregulation of lipid metabolism genes via the PI3K/AKT pathway in a diet-induced mouse model of nonalcoholic fatty liver disease.Plant-derived antivirals against hepatitis c virus infection.Synergistic effect of phytochemicals on cholesterol metabolism and lipid accumulation in HepG2 cells.Phytochemicals That Influence Gut Microbiota as Prophylactics and for the Treatment of Obesity and Inflammatory Diseases.
P2860

Modulation of PI3K-LXRα-dependent lipogenesis mediated by oxidative/nitrosative stress contributes to inhibition of HCV replication by quercetin.

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http://www.wikidata.org/entity/Q34401965
description
2014 nî lūn-bûn
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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
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2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@ast
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@en
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@nl
type
label
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@ast
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@en
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@nl
prefLabel
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@ast
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@en
Modulation of PI3K-LXRα-depend ...... HCV replication by quercetin.
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P433
P478
P50
P577
P5875
P6179
P698
P356
P1433
P1476
Modulation of PI3K-LXRα-depend ...... f HCV replication by quercetin
@en
P2093
P2860
P2888
P304
P31
P356
10.1038/LABINVEST.2013.156
P433
P478
P50
P577
P5875
P6179
P698