Drastic down-regulation of Krüppel-like factor 4 expression is critical in human gastric cancer development and progression. - Wikidata - wikimedia - TriplyDB

Drastic down-regulation of Krüppel-like factor 4 expression is critical in human gastric cancer development and progression.

Drastic down-regulation of Krüppel-like factor 4 expression is critical in human gastric cancer development and progression.

http://www.wikidata.org/entity/Q34408251

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Krüppel-like factor 4 regulates genetic stability in mouse embryonic fibroblasts Mammalian Krüppel-like factors in health and diseases Prognostic value and function of KLF4 in prostate cancer: RNAa and vector-mediated overexpression identify KLF4 as an inhibitor of tumor cell growth and migration The diverse functions of Krüppel-like factors 4 and 5 in epithelial biology and pathobiology Stem cells in gastric cancer MiR-7 promotes epithelial cell transformation by targeting the tumor suppressor KLF4 Transgenic and gene knockout mice in gastric cancer research Haploinsufficiency of Krüppel-like factor 4 promotes adenomatous polyposis coli dependent intestinal tumorigenesis Conditional deletion of the mouse Klf4 gene results in corneal epithelial fragility, stromal edema, and loss of conjunctival goblet cells Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injury On the statistical assessment of classifiers using DNA microarray data.The Transcription Regulator Krüppel-Like Factor 4 and Its Dual Roles of Oncogene in Glioblastoma and Tumor Suppressor in Neuroblastoma.Expression of Krüppel-like factor 4 in breast cancer tissues and its effects on the proliferation of breast cancer MDA-MB-231 cells.MiR-145 negatively regulates Warburg effect by silencing KLF4 and PTBP1 in bladder cancer cells KLF4 is a novel candidate tumor suppressor gene in pancreatic ductal carcinoma.KLF4 is regulated by RAS/RAF/MEK/ERK signaling through E2F1 and promotes melanoma cell growth.KLF6 inhibited oral cancer migration and invasion via downregulation of mesenchymal markers and inhibition of MMP-9 activities.KLF16 promotes proliferation in gastric cancer cells via regulating p21 and CDK4.Nitric oxide synthase II suppresses the growth and metastasis of human cancer regardless of its up-regulation of protumor factors.Genetic deletion of Klf4 in the mouse intestinal epithelium ameliorates dextran sodium sulfate-induced colitis by modulating the NF-κB pathway inflammatory response MCC-555-induced NAG-1 expression is mediated in part by KLF4.KLF4 inhibition of lung cancer cell invasion by suppression of SPARC expression.High KLF4 level in normal tissue predicts poor survival in colorectal cancer patients Krüppel-like factor 4 mediates lysophosphatidic acid-stimulated migration and proliferation of PC3M prostate cancer cells.Regulation of EMT by KLF4 in gastrointestinal cancer.Elevated Krüppel-like factor 4 transcription factor in canine mammary carcinoma A novel KLF4/LDHA signaling pathway regulates aerobic glycolysis in and progression of pancreatic cancer Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Krüppel-like factor 4 gene.Critical and reciprocal regulation of KLF4 and SLUG in transforming growth factor β-initiated prostate cancer epithelial-mesenchymal transition Dopamine, by acting through its D2 receptor, inhibits insulin-like growth factor-I (IGF-I)-induced gastric cancer cell proliferation via up-regulation of Krüppel-like factor 4 through down-regulation of IGF-IR and AKT phosphorylation Krüppel-like factor 4, a tumor suppressor in hepatocellular carcinoma cells reverts epithelial mesenchymal transition by suppressing slug expression.Association of Kruppel-like factor 4 expression with the prognosis of esophageal squamous cell carcinoma patients.KLF4 Suppresses Tumor Formation in Genetic and Pharmacological Mouse Models of Colonic Tumorigenesis.Novel cross talk of Kruppel-like factor 4 and beta-catenin regulates normal intestinal homeostasis and tumor repression Suppression of epithelial-mesenchymal transition in hepatocellular carcinoma cells by Krüppel-like factor 4.HDAC1 and Klf4 interplay critically regulates human myeloid leukemia cell proliferation KLF4 overexpression and apigenin treatment down regulated anti-apoptotic Bcl-2 proteins and matrix metalloproteinases to control growth of human malignant neuroblastoma SK-N-DZ and IMR-32 cells Klf4 is a transcriptional regulator of genes critical for EMT, including Jnk1 (Mapk8).Attenuation of krüppel-like factor 4 facilitates carcinogenesis by inducing g1/s phase arrest in clear cell renal cell carcinoma Mouse embryonic fibroblasts null for the Krüppel-like factor 4 gene are genetically unstable.

P2860

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P2860

Drastic down-regulation of Krüppel-like factor 4 expression is critical in human gastric cancer development and progression.

http://www.wikidata.org/entity/Q34408251

description

2005 nî lūn-bûn

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2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2005 թվականի ապրիլին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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type

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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Drastic down-regulation of Krü ...... r development and progression.

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0008-5472.CAN-04-3619

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Cancer Research

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Drastic down-regulation of Krü ...... r development and progression.

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Christian Schlunk

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Daoyan Wei

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James C Yao

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Keping Xie

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Liwei Wang

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Masashi Kanai

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Suyun Huang

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Tsung-Teh Wu

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Weida Gong

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2746-2754

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scholarly article

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10.1158/0008-5472.CAN-04-3619

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7

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65

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15805274

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