about
Safety of targeting tumor endothelial cell antigensT Cell Epitope Peptide Therapy for Allergic DiseasesImmunoregulatory T cell epitope peptides: the new frontier in allergy therapyMolecular and immunological characterization of hr44, a human ocular component immunologically cross-reactive with antigen Ov39 of Onchocerca volvulusT cells that are autoreactive to beta2-glycoprotein I in patients with antiphospholipid syndrome and healthy individualsAntigen mimicry in autoimmune disease sharing of amino acid residues critical for pathogenic T cell activationPeripheral T-cell tolerance induced in naive and primed mice by subcutaneous injection of peptides from the major cat allergen Fel d IHealthy individuals have Goodpasture autoantigen-reactive T cells.The etiology of paraneoplastic autoimmunity.PPD and hsp65 induced monoarthritis initiates spontaneous recurrent flares in Lewis rats.Polymorphism in the LMP2 gene influences susceptibility to extraspinal disease in HLA-B27 positive individuals with ankylosing spondylitis.Binding of myelin basic protein peptides to human histocompatibility leukocyte antigen class II molecules and their recognition by T cells from multiple sclerosis patientsProperties of HLA class II molecules divergently associated with Goodpasture's disease.Role of heat shock proteins in protection from and pathogenesis of infectious diseases.T helper cell recognition of muscle acetylcholine receptor in myasthenia gravis. Epitopes on the gamma and delta subunits.Intra- and intermolecular spreading of autoimmunity involving the nuclear self-antigens La (SS-B) and Ro (SS-A).Increased T cell reactivity to amyloid beta protein in older humans and patients with Alzheimer diseaseSpecific T-cell tolerance may be preceded by a primary response.Myasthenia gravis. CD4+ T epitopes on the embryonic gamma subunit of human muscle acetylcholine receptorT cell proliferative response induced by DNA topoisomerase I in patients with systemic sclerosis and healthy donorsCritical role of macrophages in the marginal zone in the suppression of immune responses to apoptotic cell-associated antigens.Certain HLA-DR5 and -DR6 major histocompatibility complex class II alleles are associated with a CD8 lymphocytic host response to human immunodeficiency virus type 1 characterized by low lymphocyte viral strain heterogeneity and slow disease progresT cell recognition of self-human histocompatibility leukocyte antigens (HLA)-DR peptides in context of syngeneic HLA-DR moleculesImmunogenicity and tolerogenicity of self-major histocompatibility complex peptides.An evaluation of the potential to use tumor-associated antigens as targets for antitumor T cell therapy using transgenic mice expressing a retroviral tumor antigen in normal lymphoid tissues.Initiation of autoimmunity to the p53 tumor suppressor protein by complexes of p53 and SV40 large T antigen.Autoantigens targeted in systemic lupus erythematosus are clustered in two populations of surface structures on apoptotic keratinocytes.DNA-dependent protein kinase is one of a subset of autoantigens specifically cleaved early during apoptosisUnresponsiveness to a self-peptide of mouse lysozyme owing to hindrance of T cell receptor-major histocompatibility complex/peptide interaction caused by flanking epitopic residuesA predictable sequential determinant spreading cascade invariably accompanies progression of experimental autoimmune encephalomyelitis: a basis for peptide-specific therapy after onset of clinical diseaseInactivation of T cell receptor peptide-specific CD4 regulatory T cells induces chronic experimental autoimmune encephalomyelitis (EAE).Hierarchical self-tolerance to T cell determinants within the ubiquitous nuclear self-antigen La (SS-B) permits induction of systemic autoimmunity in normal mice.The stimulation of low-affinity, nontolerized clones by heteroclitic antigen analogues causes the breaking of tolerance established to an immunodominant T cell epitope.T cell receptor complementarity determining region 3 length analysis reveals the absence of a characteristic public T cell repertoire in neonatal tolerance. The response in the "tolerant" mouse within the residual repertoire is quantitatively similaAbortive proliferation of rare T cells induced by direct or indirect antigen presentation by rare B cells in vivo.Endogenous myelin basic protein inactivates the high avidity T cell repertoireThe presentation of self-peptides: tolerance and competition.The cell biology of antigen processing.Tolerance to a self-protein involves its immunodominant but does not involve its subdominant determinantsGene vaccination for the induction of immune tolerance.
P2860
Q26748863-CC9245F2-4221-4557-BDE3-EE3BCBFF5D7FQ26771989-14F159B8-4388-4C6A-A5B1-8CB16D5BD4ABQ26822702-26E02167-E870-441F-B846-183914E44967Q28118109-6B648E5F-802C-4AEA-BEFF-855C68A6370EQ28143036-5D549328-55CF-40B6-B371-B2BAE3A14545Q30449280-B7CFC71D-78F7-4382-AB0A-D6D2DC638E39Q30454050-8BC36C2F-A88F-422A-85CD-347172D13718Q33316272-933B71A8-DBCE-4DE0-846D-12E751AA5610Q33393705-8303D238-8914-462B-A2C3-7DD7B373BC7DQ33566939-EB9932DE-52FB-4917-86DA-805F6A47C0A4Q33567242-12B1368D-4941-4499-83E2-F2F56DCE9194Q33890993-E75CF5A1-F334-4A4B-A3CD-E82E1ACD7AD8Q33911738-277CF99E-2AF6-4C4D-9FE6-B5234ACD8B1AQ33975393-3953F188-0CBC-40DF-93BD-AC4C9C64B732Q34172481-8174D5DA-F121-4036-866B-FE0696AB6605Q34744384-B63022DA-539E-4649-B996-6A90FACF3318Q35169132-10D90084-E009-423F-B1DF-F01F292580BFQ35507250-D2244AEF-3833-4657-AEDC-F65A39260457Q35607371-E9A62F5E-C380-4087-AA4D-674B07AB6BB1Q35751372-5D834649-F6EA-4409-B197-CF2D6D42CE75Q35901015-64093733-FBCA-498F-AEC2-1D69F9182E6EQ35917416-4981AE92-3662-4656-9298-6A61AF10148AQ36231401-467B003C-0D13-441D-A2F9-253B3F83B4EDQ36353829-A1B79B38-DD8B-4D4D-8F1D-46DA42FC5CBCQ36361846-A2CBC6FD-5E4B-4992-A6BC-2A3BD764FDCFQ36362919-33A263FC-AAED-41FA-83D9-56FCEA4522EAQ36363024-C6F7547F-86D4-45A7-A9B8-FDE1A00815B8Q36365577-776CB6F4-7FFE-436F-8BF6-27D9A89AF178Q36366238-38D4BA20-D8FC-458E-9DAD-F927BB8CDA56Q36366545-95820B0F-B756-40D9-B6F9-BEB322B3DE91Q36367554-BCE1D2AA-46DD-408F-8DB5-1CEECF7A748FQ36367698-EBAB210A-359B-457A-A9E5-E0E30F5D252EQ36375353-CF872CA6-D8C4-4137-B4EC-B9CD75CE6F6DQ36375972-5E8896B6-A5E4-4819-A9C8-6F4A348EA773Q36400871-88D7DF20-C659-4F64-AB4C-D4A7D1669DFAQ36401074-F5F64FCD-F9B0-491F-AC09-0F27DA04EB28Q36676941-3099D523-6ADA-4B83-85BC-18D5BBF46CB6Q36707417-7D274B21-81CA-4852-9C3C-ABA6764714E6Q36778058-979AA8AE-E4E9-4C0C-A920-BF31345EE4E7Q36958305-4DBC8B1E-2614-492E-9BA1-4B1BB168BC49
P2860
description
1989 nî lūn-bûn
@nan
1989 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
name
How some T cells escape tolerance induction.
@ast
How some T cells escape tolerance induction.
@en
How some T cells escape tolerance induction.
@nl
type
label
How some T cells escape tolerance induction.
@ast
How some T cells escape tolerance induction.
@en
How some T cells escape tolerance induction.
@nl
prefLabel
How some T cells escape tolerance induction.
@ast
How some T cells escape tolerance induction.
@en
How some T cells escape tolerance induction.
@nl
P356
P1433
P1476
How some T cells escape tolerance induction.
@en
P2093
P2888
P304
P356
10.1038/342183A0
P407
P577
1989-11-01T00:00:00Z
P6179
1041463657