Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy. - Wikidata - wikimedia - TriplyDB

Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.

Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.

http://www.wikidata.org/entity/Q34427632

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Uric Acid as a Marker of Kidney Disease: Review of the Current Literature Diabetic Kidney Disease: Pathophysiology and Therapeutic Targets Diabetic nephropathy - complications and treatment.Aldose reductase expression as a risk factor for cataract The Short-Term Remission of Diabetic Nephropathy After Roux-en-Y Gastric Bypass in Chinese Patients of T2DM with Obesity Lower urinary pH is useful for predicting renovascular disorder onset in patients with diabetes.Effect of dietary fructose on portal and systemic serum fructose levels in rats and in KHK-/- and GLUT5-/- mice.Mini-review: diabetic renal complications, a potential stinky remedy.Hyperfiltration and uricosuria in adolescents with type 1 diabetes.Fructose and uric acid in diabetic nephropathy The human brain produces fructose from glucose.Protective role of fructokinase blockade in the pathogenesis of acute kidney injury in mice.The sweetest thing: blocking fructose metabolism to prevent acute kidney injury?Effects of exogenous desmopressin on a model of heat stress nephropathy in mice.Serum uric acid and acute kidney injury: A mini review.Renoprotective effect of topiroxostat via antioxidant activity in puromycin aminonucleoside nephrosis rats Aging-associated renal disease in mice is fructokinase dependent.Sweet debate: fructose versus glucose in diabetic kidney disease.Hyperglycemia-Induced Oxidative-Nitrosative Stress Induces Inflammation and Neurodegeneration via Augmented Tuberous Sclerosis Complex-2 (TSC-2) Activation in Neuronal Cells.Role of fructose and fructokinase in acute dehydration-induced vasopressin gene expression and secretion in mice.Mitochondrial energetics in the kidney.Fructose metabolism and metabolic disease.Deletion of pro-angiogenic factor vasohibin-2 ameliorates glomerular alterations in a mouse diabetic nephropathy model.Ketohexokinase C blockade ameliorates fructose-induced metabolic dysfunction in fructose-sensitive mice.Mechanisms for the establishment and maintenance of pregnancy: Synergies from scientific collaborations.Curcumin protects against fructose-induced podocyte insulin signaling impairment through upregulation of miR-206.A Higher Fructose Intake Is Associated with Greater Albuminuria in Subjects with Type 2 Diabetes Mellitus

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P2860

Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.

http://www.wikidata.org/entity/Q34427632

description

2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Endogenous fructose production ...... njury in diabetic nephropathy.

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Endogenous fructose production ...... njury in diabetic nephropathy.

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Endogenous fructose production ...... njury in diabetic nephropathy.

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Endogenous fructose production ...... njury in diabetic nephropathy.

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Journal of the American Society of Nephrology

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Endogenous fructose production ...... njury in diabetic nephropathy.

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A Ahsan Ejaz

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Ana Andres-Hernando

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Aruna Asipu

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Bernardo Rodriguez-Iturbe

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Carlos A Roncal-Jimenez

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Christina Cicerchi

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Christopher J Rivard

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David J Orlicky

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Gabriela E Garcia

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J Mark Petrash

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P2860

Opposing effects of fructokinase C and A isoforms on fructose-induced metabolic syndrome in mice

Ketohexokinase: expression and localization of the principal fructose-metabolizing enzyme

Effects of losartan on renal and cardiovascular outcomes in patients with type 2 diabetes and nephropathy

Dietary fructose causes tubulointerstitial injury in the normal rat kidney

The tubulointerstitium in progressive diabetic kidney disease: more than an aftermath of glomerular injury?

Aldo-keto reductases in the eye.

Diabetic nephropathy is accelerated by farnesoid X receptor deficiency and inhibited by farnesoid X receptor activation in a type 1 diabetes model

Connecting tubule glomerular feedback antagonizes tubuloglomerular feedback in vivo.

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

Uric acid stimulates fructokinase and accelerates fructose metabolism in the development of fatty liver.

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2526-2538

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scholarly article

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10.1681/ASN.2013080901

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11

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25

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David T. Bonthron

Christine Diggle

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2014-05-29T00:00:00Z

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24876114

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4214522

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