BTBR Ob/Ob mutant mice model progressive diabetic nephropathy. - Wikidata - wikimedia - TriplyDB

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

http://www.wikidata.org/entity/Q34455365

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Recent Progress in Stem Cell Therapy for Diabetic Nephropathy An Update on the Use of Animal Models in Diabetic Nephropathy Research New experimental models of diabetic nephropathy in mice models of type 2 diabetes: efforts to replicate human nephropathy Mesenchymal Stem Cells as Therapeutic Candidates for Halting the Progression of Diabetic Nephropathy Long term metabolic syndrome induced by a high fat high fructose diet leads to minimal renal injury in C57BL/6 mice Diabetic nephropathy in a nonobese mouse model of type 2 diabetes mellitus.Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease.Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.CCR2 antagonist CCX140-B provides renal and glycemic benefits in diabetic transgenic human CCR2 knockin mice.MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.Microplate-based platform for combined chromatin and DNA methylation immunoprecipitation assays Fluorofenidone offers improved renoprotection at early interventions during the course of diabetic nephropathy in db/db mice via multiple pathways Endogenous fructose production and fructokinase activation mediate renal injury in diabetic nephropathy.Innate immune detection of flagellin positively and negatively regulates salmonella infection.Knockout of the TauT gene predisposes C57BL/6 mice to streptozotocin-induced diabetic nephropathy.Diffuse glomerular nodular lesions in diabetic pigs carrying a dominant-negative mutant hepatocyte nuclear factor 1-alpha, an inheritant diabetic gene in humans.Serum amyloid A and inflammation in diabetic kidney disease and podocytes.Chronic ingestion of high dosed Phikud Navakot extraction induces mesangiolysis in rats with alteration of AQP1 and Hsp60 expressions.Investigating mechanisms of chronic kidney disease in mouse models Effects of CP-900691, a novel peroxisome proliferator-activated receptor α, agonist on diabetic nephropathy in the BTBR ob/ob mouse Semaphorin3a promotes advanced diabetic nephropathy BTBR ob/ob mice as a novel diabetic neuropathy model: Neurological characterization and gene expression analyses Impaired Coronary and Renal Vascular Function in Spontaneously Type 2 Diabetic Leptin-Deficient Mice.Interstitial pericytes decrease in aged mouse kidneys.Cells of renin lineage express hypoxia inducible factor 2α following experimental ureteral obstruction.Integrin α1/Akita double-knockout mice on a Balb/c background develop advanced features of human diabetic nephropathy.Diet-Induced Podocyte Dysfunction in Drosophila and Mammals.Mechanism of Altered Metformin Distribution in Nonalcoholic Steatohepatitis Gender-specific differences in diabetic neuropathy in BTBR ob/ob mice Balanced regulation of the CCN family of matricellular proteins: a novel approach to the prevention and treatment of fibrosis and cancer.B7-1 Is Not Induced in Podocytes of Human and Experimental Diabetic Nephropathy.Advances in murine models of diabetic nephropathy.Reversibility of structural and functional damage in a model of advanced diabetic nephropathy Dipeptidyl peptidase-4 inhibition by Saxagliptin prevents inflammation and renal injury by targeting the Nlrp3/ASC inflammasome.Cyclodextrin protects podocytes in diabetic kidney disease.Noninvasive imaging of retinal morphology and microvasculature in obese mice using optical coherence tomography and optical microangiography.The effects of dual PPARα/γ agonism compared with ACE inhibition in the BTBRob/ob mouse model of diabetes and diabetic nephropathy.Leptin as a link between the immune system and kidney-related diseases: leading actor or just a coadjuvant?Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.The SGLT2 inhibitor empagliflozin ameliorates early features of diabetic nephropathy in BTBR ob/ob type 2 diabetic mice with and without hypertension.

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P2860

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

http://www.wikidata.org/entity/Q34455365

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@ast

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@en

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@nl

type

label

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@ast

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@en

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@nl

prefLabel

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@ast

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@en

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

@nl

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Journal of the American Society of Nephrology

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BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

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Alan D Attie

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Anja Mühlfeld

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Bardia Askari

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Charles E Alpers

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Jeffrey W Pippin

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Jolanta Kowalewska

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Kelly L Hudkins

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Mariko Koelling

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Mark P Keller

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Mary E Rabaglia

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P2860

A genome-wide association study identifies a novel major locus for glycemic control in type 1 diabetes, as measured by both A1C and glucose

Lifetime risk for diabetes mellitus in the United States

Mac-2-binding glycoproteins. Putative ligands for a cytosolic beta-galactoside lectin

K/DOQI clinical practice guidelines for chronic kidney disease: evaluation, classification, and stratification

Reduction in podocyte density as a pathologic feature in early diabetic nephropathy in rodents: prevention by lipoic acid treatment.

Rodent models of nephropathy associated with type II diabetes.

Mouse models of diabetic nephropathy.

Leptin resistance and obesity.

Pathophysiologic implications of reduced podocyte number in a rat model of progressive glomerular injury.

Outcome studies in diabetic nephropathy.

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9

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3013527

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