Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice - Wikidata - wikimedia - TriplyDB

Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice

Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice

http://www.wikidata.org/entity/Q34441342

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Mitochondria Thymidine Phosphorylase Participates in Platelet Signaling and Promotes Thrombosis Inherited mitochondrial DNA depletion.Modeling pathogenic mutations of human twinkle in Drosophila suggests an apoptosis role in response to mitochondrial defects.Long term expression of Drosophila melanogaster nucleoside kinase in thymidine kinase 2-deficient mice with no lethal effects caused by nucleotide pool imbalances.Mitochondrial Diseases Part II: Mouse models of OXPHOS deficiencies caused by defects in regulatory factors and other components required for mitochondrial function.Mitochondrial ATP transporter Ant2 depletion impairs erythropoiesis and B lymphopoiesis The role of thymidine phosphorylase, an angiogenic enzyme, in tumor progression.A discovery resource of rare copy number variations in individuals with autism spectrum disorder.Mouse models of mitochondrial DNA defects and their relevance for human disease.Unbalanced deoxynucleotide pools cause mitochondrial DNA instability in thymidine phosphorylase-deficient mice Two transgenic mouse models for β-subunit components of succinate-CoA ligase yielding pleiotropic metabolic alterations Screen for abnormal mitochondrial phenotypes in mouse embryonic stem cells identifies a model for succinyl-CoA ligase deficiency and mtDNA depletion.Mitochondrial Neurogastrointestinal Encephalomyopathy Caused by Thymidine Phosphorylase Enzyme Deficiency: From Pathogenesis to Emerging Therapeutic Options.Functional analysis of pyrimidine biosynthesis enzymes using the anticancer drug 5-fluorouracil in Caenorhabditis elegans.Nutritional Interventions for Mitochondrial OXPHOS Deficiencies: Mechanisms and Model Systems.Mitochondrial dysfunction and ovarian aging.Relationship between biodistribution of a novel thymidine phosphorylase (TP) imaging probe and TP expression levels in normal mice.Immunohistochemical detection of platelet-derived endothelial cell growth factor/thymidine phosphorylase and angiopoietins in ameloblastic tumors.Capecitabine reverses tumor escape from anti-VEGF through the eliminating CD11bhigh/Gr1high myeloid cells.

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Targeted deletion of both thymidine phosphorylase and uridine phosphorylase and consequent disorders in mice

http://www.wikidata.org/entity/Q34441342

description

2002 nî lūn-bûn

@nan

2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2002年の論文

@ja

2002年論文

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2002年論文

@zh-hant

2002年論文

@zh-hk

2002年論文

@zh-mo

2002年論文

@zh-tw

2002年论文

@wuu

name

Targeted deletion of both thym ...... d consequent disorders in mice

@ast

Targeted deletion of both thym ...... d consequent disorders in mice

@en

Targeted deletion of both thym ...... d consequent disorders in mice

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type

label

Targeted deletion of both thym ...... d consequent disorders in mice

@ast

Targeted deletion of both thym ...... d consequent disorders in mice

@en

Targeted deletion of both thym ...... d consequent disorders in mice

@nl

prefLabel

Targeted deletion of both thym ...... d consequent disorders in mice

@ast

Targeted deletion of both thym ...... d consequent disorders in mice

@en

Targeted deletion of both thym ...... d consequent disorders in mice

@nl

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P356

MCB.22.14.5212-5221.2002

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Molecular and Cellular Biology

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Targeted deletion of both thym ...... d consequent disorders in mice

@en

P2093

Atsuo Nakayama

http://www.w3.org/2001/XMLSchema#string

Fuyuki Ishikawa

http://www.w3.org/2001/XMLSchema#string

Hideo Toyoshima

http://www.w3.org/2001/XMLSchema#string

Hideo Utsumi

http://www.w3.org/2001/XMLSchema#string

Hiroaki Kawaguchi

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Hiroaki Tsujimoto

http://www.w3.org/2001/XMLSchema#string

Hiroki Yoshida

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Hiromitsu Hara

http://www.w3.org/2001/XMLSchema#string

Itsuro Higuchi

http://www.w3.org/2001/XMLSchema#string

Junko Hirato

http://www.w3.org/2001/XMLSchema#string

P2860

Thymidine phosphorylase gene mutations in MNGIE, a human mitochondrial disorder

Fatal infantile cardioencephalomyopathy with COX deficiency and mutations in SCO2, a COX assembly gene

Effects of platelet-derived endothelial cell growth factor/thymidine phosphorylase, substrate, and products in a three-dimensional model of angiogenesis

Platelet-derived endothelial cell growth factor has thymidine phosphorylase activity

Mutations in SCO2 are associated with a distinct form of hypertrophic cardiomyopathy and cytochrome c oxidase deficiency

Identification of angiogenic activity and the cloning and expression of platelet-derived endothelial cell growth factor

Mitochondrial neurogastrointestinal encephalomyopathy (MNGIE): clinical, biochemical, and genetic features of an autosomal recessive mitochondrial disorder

Cloning and characterization of full-length mouse thymidine kinase 2: the N-terminal sequence directs import of the precursor protein into mitochondria.

MNGIE: from nuclear DNA to mitochondrial DNA.

[Mitochondrial neurogastrointestinal encephalomyopathy presenting with protein-losing gastroenteropathy and serum copper deficiency: a case report]

P304

5212-5221

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scholarly article

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10.1128/MCB.22.14.5212-5221.2002

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P407

P433

14

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22

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P50

Hideto Kuribayashi

P577

2002-07-01T00:00:00Z

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12077348

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139774

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