Dysfunctional behavioral modulation of corticostriatal communication in the R6/2 mouse model of Huntington's disease
about
Corticostriatal Dysfunction in Huntington's Disease: The BasicsRole of cerebral cortex in the neuropathology of Huntington's diseaseCorrelations of behavioral deficits with brain pathology assessed through longitudinal MRI and histopathology in the R6/1 mouse model of Huntington's diseaseA small molecule p75NTR ligand normalizes signalling and reduces Huntington's disease phenotypes in R6/2 and BACHD mice.Behavior modulates effective connectivity between cortex and striatum.Cortical efferents lacking mutant huntingtin improve striatal neuronal activity and behavior in a conditional mouse model of Huntington's diseaseQuantitative Electroencephalographic Analysis Provides an Early-Stage Indicator of Disease Onset and Progression in the zQ175 Knock-In Mouse Model of Huntington's Disease.β oscillation during slow wave sleep and rapid eye movement sleep in the electroencephalogram of a transgenic mouse model of Huntington's diseaseQuantitative Electroencephalographic Biomarkers in Preclinical and Human Studies of Huntington's Disease: Are They Fit-for-Purpose for Treatment Development?Corticostriatal connectivity and its role in diseaseChronic Paroxetine Treatment Prevents the Emergence of Abnormal Electroencephalogram Oscillations in Huntington's Disease Mice.Cognitive dysfunction in Huntington's disease: mechanisms and therapeutic strategies beyond BDNF.Transneuronal propagation of mutant huntingtin contributes to non-cell autonomous pathology in neurons.Dysregulation of Corticostriatal Connectivity in Huntington's Disease: A Role for Dopamine ModulationDysregulation of corticostriatal ascorbate release and glutamate uptake in transgenic models of Huntington's disease.Relationship between subthalamic nucleus neuronal activity and electrocorticogram is altered in the R6/2 mouse model of Huntington's disease.Striatal Network Models of Huntington's Disease Dysfunction Phenotypes.Longitudinal analysis of the electroencephalogram and sleep phenotype in the R6/2 mouse model of Huntington's disease.Progressive sleep and electroencephalogram changes in mice carrying the Huntington's disease mutation.Dysregulated corticostriatal activity in open-field behavior and the head-twitch response induced by the hallucinogen 2,5-dimethoxy-4-iodoamphetamine.
P2860
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P2860
Dysfunctional behavioral modulation of corticostriatal communication in the R6/2 mouse model of Huntington's disease
description
2012 nî lūn-bûn
@nan
2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
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name
Dysfunctional behavioral modul ...... model of Huntington's disease
@ast
Dysfunctional behavioral modul ...... model of Huntington's disease
@en
Dysfunctional behavioral modul ...... model of Huntington's disease
@nl
type
label
Dysfunctional behavioral modul ...... model of Huntington's disease
@ast
Dysfunctional behavioral modul ...... model of Huntington's disease
@en
Dysfunctional behavioral modul ...... model of Huntington's disease
@nl
prefLabel
Dysfunctional behavioral modul ...... model of Huntington's disease
@ast
Dysfunctional behavioral modul ...... model of Huntington's disease
@en
Dysfunctional behavioral modul ...... model of Huntington's disease
@nl
P2093
P2860
P1433
P1476
Dysfunctional behavioral modul ...... model of Huntington's disease
@en
P2093
Desirée Cossyleon
George V Rebec
Lauren J Walker
Scott J Barton
Wajeeha A Hussain
P2860
P304
P356
10.1371/JOURNAL.PONE.0047026
P407
P50
P577
2012-10-04T00:00:00Z