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REX-1 expression and p38 MAPK activation status can determine proliferation/differentiation fates in human mesenchymal stem cellsActivation and function of the MAPKs and their substrates, the MAPK-activated protein kinasesPhosphorylation of Hsl1 by Hog1 leads to a G2 arrest essential for cell survival at high osmolarityHog1 mediates cell-cycle arrest in G1 phase by the dual targeting of Sic1.Cell cycle progression and de novo centriole assembly after centrosomal removal in untransformed human cells.Inhibition of p38 MAPK signaling augments skin tumorigenesis via NOX2 driven ROS generation.Downregulation of P38 phosphorylation correlates with low-grade differentiation and proliferation of lung squamous cell carcinoma.Potassium channel ether à go-go1 is aberrantly expressed in human liposarcoma and promotes tumorigenesisActivation of ERK induces phosphorylation of MAPK phosphatase-7, a JNK specific phosphatase, at Ser-446.Effect of phosphatidyl inositol 3-kinase, extracellular signal-regulated kinases 1/2, and p38 mitogen-activated protein kinase inhibition on osteogenic differentiation of muscle-derived stem cells.p38 MAP kinase inhibition enables proliferation of adult mammalian cardiomyocytes.Negative feedback regulation of MKK6 mRNA stability by p38alpha mitogen-activated protein kinaseDealing with osmostress through MAP kinase activation.Loss of oncogenic H-ras-induced cell cycle arrest and p38 mitogen-activated protein kinase activation by disruption of Gadd45a.Correlation between p38 mitogen-activated protein kinase and human telomerase reverse transcriptase in sarcomas.The p57 CDKi integrates stress signals into cell-cycle progression to promote cell survival upon stress.Deletion of p38-alpha mitogen-activated protein kinase within the intestinal epithelium promotes colon tumorigenesis.p38α regulates actin cytoskeleton and cytokinesis in hepatocytes during development and agingThe p38 mitogen-activated protein kinase signaling cascade in CD4 T cells.Cardiac myocyte cell cycle control in development, disease, and regeneration.Functional p38 MAPK identified by biomarker profiling of pancreatic cancer restrains growth through JNK inhibition and correlates with improved survival.Identification of Poor-outcome Biliopancreatic Carcinoma Patients With Two-marker Signature Based on ATF6α and p-p38 "STARD Compliant".The transcriptional repressor HBP1 is a target of the p38 mitogen-activated protein kinase pathway in cell cycle regulation.Xp38gamma/SAPK3 promotes meiotic G(2)/M transition in Xenopus oocytes and activates Cdc25C.p38(MAPK): stress responses from molecular mechanisms to therapeutics.The p38 and Hog1 SAPKs control cell cycle progression in response to environmental stresses.Estrogen potentiates reactive oxygen species (ROS) tolerance to initiate carcinogenesis and promote cancer malignant transformation.Insulin Exhibits an Antiproliferative and Hypertrophic Effect in First Trimester Human Extravillous Trophoblasts.Aristolochic acid-induced apoptosis and G2 cell cycle arrest depends on ROS generation and MAP kinases activation.Obatoclax induces G1/G0-phase arrest via p38/p21(waf1/Cip1) signaling pathway in human esophageal cancer cells.Delayed cell cycle progression in selenoprotein W-depleted cells is regulated by a mitogen-activated protein kinase kinase 4-p38/c-Jun NH2-terminal kinase-p53 pathway.Phosphorylation of FOXO3a on Ser-7 by p38 promotes its nuclear localization in response to doxorubicin.p38 MAPK contributes to the growth inhibition of leukemic tumor cells mediated by human umbilical cord mesenchymal stem cells.Modulation of the p38 MAPK (mitogen-activated protein kinase) pathway through Bcr/Abl: implications in the cellular response to Ara-C.A novel function of p38-regulated/activated kinase in endothelial cell migration and tumor angiogenesis.High WAVE3 expression correlates with proliferation, migration and invasion in human ovarian cancer.OSU-DY7, a novel D-tyrosinol derivative, mediates cytotoxicity in chronic lymphocytic leukaemia and Burkitt lymphoma through p38 mitogen-activated protein kinase pathway.TGF-β1 induces endothelial cell apoptosis by shifting VEGF activation of p38(MAPK) from the prosurvival p38β to proapoptotic p38α.Signaling pathways implicated in the stimulation of beta-cell proliferation by extracellular matrix.A TLR9-dependent checkpoint governs B cell responses to DNA-containing antigens.
P2860
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P2860
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Cell cycle regulation by p38 MAP kinases.
@ast
Cell cycle regulation by p38 MAP kinases.
@en
Cell cycle regulation by p38 MAP kinases.
@nl
type
label
Cell cycle regulation by p38 MAP kinases.
@ast
Cell cycle regulation by p38 MAP kinases.
@en
Cell cycle regulation by p38 MAP kinases.
@nl
prefLabel
Cell cycle regulation by p38 MAP kinases.
@ast
Cell cycle regulation by p38 MAP kinases.
@en
Cell cycle regulation by p38 MAP kinases.
@nl
P1433
P1476
Cell cycle regulation by p38 MAP kinases.
@en
P2093
A R Nebreda
C Ambrosino
P356
10.1016/S0248-4900(01)01124-8
P577
2001-09-01T00:00:00Z