The AGE-receptor in the pathogenesis of diabetic complications.
about
Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cellsAdvanced glycation end products in foods and a practical guide to their reduction in the dietRole of advanced glycation end products in cellular signalingPeriodontitis and diabetes: a two-way relationshipThe effect of an AGE-rich dietary extract on the activation of NF-κB depends on the cell model used.[Protein glycation as a pathological mechanism in diabetes].Dissociation of functional status from accrual of CML and RAGE in the aged mouse brain.Advanced glycation end products induce a prothrombotic phenotype in mice via interaction with platelet CD36.Association analysis of ADPRT1, AKR1B1, RAGE, GFPT2 and PAI-1 gene polymorphisms with chronic renal insufficiency among Asian Indians with type-2 diabetes.Advanced glycation endproduct (AGE) accumulation and AGE receptor (RAGE) up-regulation contribute to the onset of diabetic cardiomyopathy.Ursodeoxycholic acid ameliorates fructose-induced metabolic syndrome in rats.Covalent binding antibodies suppress advanced glycation: on the innate tier of adaptive immunity.Advanced glycation in health and disease: role of the modern environment.The maillard reaction in eye diseases.Involvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategiesAdvanced glycation end product (AGE) receptor 1 suppresses cell oxidant stress and activation signaling via EGF receptorGlycated albumin produced in diabetic hyperglycemia promotes monocyte secretion of inflammatory cytokines and bacterial adherence to epithelial cells.The Effect of Systemic Delivery of Aminoguanidine versus Doxycycline on the Resorptive Phase of Alveolar Bone Following modified Widman Flap in Diabetic Rats: A Histopathological and Scanning Electron Microscope (SEM) studyRetinal proteins associated with redox regulation and protein folding play central roles in response to high glucose conditions.Chronic administration of sildenafil improves erectile function in a rat model of chronic renal failure.Genetic deficiency of neuronal RAGE protects against AGE-induced synaptic injury.Significance of proximal tubular metabolism of advanced glycation end products in kidney diseases.Low-molecular weight advanced glycation end products: markers of tissue AGE accumulation and more?LDL biochemical modifications: a link between atherosclerosis and aging.PPAR Agonists and Cardiovascular Disease in Diabetes.An emerging role of glucagon-like peptide-1 in preventing advanced-glycation-end-product-mediated damages in diabetesAdvanced glycation end products: Key players in skin aging?Multispecificity of immunoglobulin M antibodies raised against advanced glycation end products: involvement of electronegative potential of antigensActivation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetesEnzymatic and non-enzymatic functions of the lysyl oxidase family in boneThe Association of -429T>C and -374T>A Polymorphisms in the RAGE Gene with Polycystic Ovary Syndrome.AGE restriction in diabetes mellitus: a paradigm shift.Cellular signaling and potential new treatment targets in diabetic retinopathy.Determination of Free-Form and Peptide Bound Pyrraline in the Commercial Drinks Enriched with Different Protein HydrolysatesReactive immunization suppresses advanced glycation and mitigates diabetic nephropathyZerumbone, a Phytochemical of Subtropical Ginger, Protects against Hyperglycemia-Induced Retinal Damage in Experimental Diabetic RatsUse of human vascular tissue microarrays for measurement of advanced glycation endproductsIdentifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly personsAdvanced glycation end product receptor-1 transgenic mice are resistant to inflammation, oxidative stress, and post-injury intimal hyperplasia.Priming of neutrophil oxidative burst in diabetes requires preassembly of the NADPH oxidase.
P2860
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P2860
The AGE-receptor in the pathogenesis of diabetic complications.
description
2001 nî lūn-bûn
@nan
2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
The AGE-receptor in the pathogenesis of diabetic complications.
@ast
The AGE-receptor in the pathogenesis of diabetic complications.
@en
The AGE-receptor in the pathogenesis of diabetic complications.
@nl
type
label
The AGE-receptor in the pathogenesis of diabetic complications.
@ast
The AGE-receptor in the pathogenesis of diabetic complications.
@en
The AGE-receptor in the pathogenesis of diabetic complications.
@nl
prefLabel
The AGE-receptor in the pathogenesis of diabetic complications.
@ast
The AGE-receptor in the pathogenesis of diabetic complications.
@en
The AGE-receptor in the pathogenesis of diabetic complications.
@nl
P2860
P356
P1476
The AGE-receptor in the pathogenesis of diabetic complications.
@en
P2093
Vlassara H
P2860
P304
P356
10.1002/DMRR.233
P577
2001-11-01T00:00:00Z