Mycobacterium ulcerans triggers T-cell immunity followed by local and regional but not systemic immunosuppression.
about
The pathogenic mechanism of the Mycobacterium ulcerans virulence factor, mycolactone, depends on blockade of protein translocation into the ERGenetic Variation in Autophagy-Related Genes Influences the Risk and Phenotype of Buruli UlcerHeterogeneous Family of Cyclomodulins: Smart Weapons That Allow Bacteria to Hijack the Eukaryotic Cell Cycle and Promote Infections.Cellular immunity confers transient protection in experimental Buruli ulcer following BCG or mycolactone-negative Mycobacterium ulcerans vaccination.Buruli ulcer.Photodegradation of the Mycobacterium ulcerans toxin, mycolactones: considerations for handling and storageCorticosteroid-induced immunosuppression ultimately does not compromise the efficacy of antibiotherapy in murine Mycobacterium ulcerans infection.Microbiological, histological, immunological, and toxin response to antibiotic treatment in the mouse model of Mycobacterium ulcerans diseaseStructure-activity relationship studies on the macrolide exotoxin mycolactone of Mycobacterium ulcerans.Phage therapy is effective against infection by Mycobacterium ulcerans in a murine footpad modelCombined inflammatory and metabolic defects reflected by reduced serum protein levels in patients with Buruli ulcer diseaseTreating Mycobacterium ulcerans disease (Buruli ulcer): from surgery to antibiotics, is the pill mightier than the knife?Local and regional re-establishment of cellular immunity during curative antibiotherapy of murine Mycobacterium ulcerans infectionSpontaneous Healing of Mycobacterium ulcerans Lesions in the Guinea Pig ModelAnalysis of Mycobacterium ulcerans-specific T-cell cytokines for diagnosis of Buruli ulcer disease and as potential indicator for disease progression.An Optimized Method for Extracting Bacterial RNA from Mouse Skin Tissue Colonized by Mycobacterium ulceransHistory, biology and chemistry of Mycobacterium ulcerans infections (Buruli ulcer disease).FVB/N Mice Spontaneously Heal Ulcerative Lesions Induced by Mycobacterium ulcerans and Switch M. ulcerans into a Low Mycolactone Producer.Susceptibility to Mycobacterium ulcerans Disease (Buruli ulcer) Is Associated with IFNG and iNOS Gene Polymorphisms.[Buruli ulcer: a dynamic transversal research model performed through the international network of Pasteur Institutes].Membrane perturbing properties of toxin mycolactone from Mycobacterium ulcerans.
P2860
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P2860
Mycobacterium ulcerans triggers T-cell immunity followed by local and regional but not systemic immunosuppression.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@ast
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@en
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@nl
type
label
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@ast
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@en
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@nl
prefLabel
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@ast
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@en
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@nl
P2093
P2860
P50
P356
P1476
Mycobacterium ulcerans trigger ...... ot systemic immunosuppression.
@en
P2093
Daniela R Pereira
Françoise Portaels
Jorge Pedrosa
Manuel T Silva
Teresa G Martins
Wayne M Meyers
P2860
P304
P356
10.1128/IAI.00820-10
P407
P577
2010-10-25T00:00:00Z