MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol - Wikidata - wikimedia - TriplyDB

MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol

MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol

http://www.wikidata.org/entity/Q34484704

about

Induction and activation of human Th17 by targeting antigens to dendritic cells via dectin-1.CD1d-restricted NKT cells modulate placental and uterine leukocyte populations during chlamydial infection in mice.Conjunctival scarring in trachoma is associated with the HLA-C ligand of KIR and is exacerbated by heterozygosity at KIR2DL2/KIR2DL3.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infection Maternal and fetal roles in bacterially induced preterm labor in the mouse.Vaginal chlamydial clearance following primary or secondary infection in mice occurs independently of TNF-α.Perforin-2 restricts growth of Chlamydia trachomatis in macrophages CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection.IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.A Protective Vaccine against Chlamydia Genital Infection Using Vault Nanoparticles without an Added Adjuvant.Natural killer (NK) cells in antibacterial innate immunity: angels or devils?The role of innate immune signals in immunity to Brucella abortus.MyD88 signaling in CD4 T cells promotes IFN-γ production and hematopoietic progenitor cell expansion in response to intracellular bacterial infection.A Chlamydia-Specific TCR-Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function.Inflammatory monocytes require type I interferon receptor signaling to activate NK cells via IL-18 during a mucosal viral infection.Chlamydia-induced ReA: immune imbalances and persistent pathogens.Loss of MyD88 leads to more aggressive TRAMP prostate cancer and influences tumor infiltrating lymphocytes.Toll-like receptor 3 (TLR3) promotes the resolution of Chlamydia muridarum genital tract infection in congenic C57BL/6N mice.

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P2860

MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol

http://www.wikidata.org/entity/Q34484704

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

MyD88 deficiency leads to decr ...... ssociated with infection resol

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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Infection and Immunity

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MyD88 deficiency leads to decr ...... ssociated with infection resol

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P2093

Anna Goodwin

http://www.w3.org/2001/XMLSchema#string

Charles W Andrews

http://www.w3.org/2001/XMLSchema#string

Daniel Prantner

http://www.w3.org/2001/XMLSchema#string

James Sikes

http://www.w3.org/2001/XMLSchema#string

Jessica N Snowden

http://www.w3.org/2001/XMLSchema#string

Lauren Frazer

http://www.w3.org/2001/XMLSchema#string

Toni Darville

http://www.w3.org/2001/XMLSchema#string

Uma M Nagarajan

http://www.w3.org/2001/XMLSchema#string

P2860

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

A MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.

Role of NK cells in early host response to chlamydial genital infection

Stimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infection

Early local cytokine profiles in strains of mice with different outcomes from chlamydial genital tract infection

Mouse strain-dependent chemokine regulation of the genital tract T helper cell type 1 immune response.

Coclustering of CD4 (L3T4) molecule with the T-cell receptor is induced by specific direct interaction of helper T cells and antigen-presenting cells

Interleukin-17 is required for T helper 1 cell immunity and host resistance to the intracellular pathogen Francisella tularensis.

Initial route of antigen administration alters the T-cell cytokine profile produced in response to the mouse pneumonitis biovar of Chlamydia trachomatis following genital infection

Mouse strain-dependent variation in the course and outcome of chlamydial genital tract infection is associated with differences in host response.

P304

486-498

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scholarly article

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10.1128/IAI.00843-10

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1

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79

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2010-11-15T00:00:00Z

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21078858

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3019903

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