MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol
about
Induction and activation of human Th17 by targeting antigens to dendritic cells via dectin-1.CD1d-restricted NKT cells modulate placental and uterine leukocyte populations during chlamydial infection in mice.Conjunctival scarring in trachoma is associated with the HLA-C ligand of KIR and is exacerbated by heterozygosity at KIR2DL2/KIR2DL3.Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infectionMaternal and fetal roles in bacterially induced preterm labor in the mouse.Vaginal chlamydial clearance following primary or secondary infection in mice occurs independently of TNF-α.Perforin-2 restricts growth of Chlamydia trachomatis in macrophagesCD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection.IL-23 induces IL-22 and IL-17 production in response to Chlamydia muridarum genital tract infection, but the absence of these cytokines does not influence disease pathogenesis.A Protective Vaccine against Chlamydia Genital Infection Using Vault Nanoparticles without an Added Adjuvant.Natural killer (NK) cells in antibacterial innate immunity: angels or devils?The role of innate immune signals in immunity to Brucella abortus.MyD88 signaling in CD4 T cells promotes IFN-γ production and hematopoietic progenitor cell expansion in response to intracellular bacterial infection.A Chlamydia-Specific TCR-Transgenic Mouse Demonstrates Th1 Polyfunctionality with Enhanced Effector Function.Inflammatory monocytes require type I interferon receptor signaling to activate NK cells via IL-18 during a mucosal viral infection.Chlamydia-induced ReA: immune imbalances and persistent pathogens.Loss of MyD88 leads to more aggressive TRAMP prostate cancer and influences tumor infiltrating lymphocytes.Toll-like receptor 3 (TLR3) promotes the resolution of Chlamydia muridarum genital tract infection in congenic C57BL/6N mice.
P2860
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P2860
MyD88 deficiency leads to decreased NK cell gamma interferon production and T cell recruitment during Chlamydia muridarum genital tract infection, but a predominant Th1 response and enhanced monocytic inflammation are associated with infection resol
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
MyD88 deficiency leads to decr ...... ssociated with infection resol
@ast
MyD88 deficiency leads to decr ...... ssociated with infection resol
@en
MyD88 deficiency leads to decr ...... ssociated with infection resol
@nl
type
label
MyD88 deficiency leads to decr ...... ssociated with infection resol
@ast
MyD88 deficiency leads to decr ...... ssociated with infection resol
@en
MyD88 deficiency leads to decr ...... ssociated with infection resol
@nl
prefLabel
MyD88 deficiency leads to decr ...... ssociated with infection resol
@ast
MyD88 deficiency leads to decr ...... ssociated with infection resol
@en
MyD88 deficiency leads to decr ...... ssociated with infection resol
@nl
P2093
P2860
P356
P1476
MyD88 deficiency leads to decr ...... ssociated with infection resol
@en
P2093
Anna Goodwin
Charles W Andrews
Daniel Prantner
James Sikes
Jessica N Snowden
Lauren Frazer
Toni Darville
Uma M Nagarajan
P2860
P304
P356
10.1128/IAI.00843-10
P407
P577
2010-11-15T00:00:00Z