An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury - Wikidata - wikimedia - TriplyDB

An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury

An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury

http://www.wikidata.org/entity/Q34485850

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The Nucleotide Oligomerization Domain-Like Receptors in Kidney Injury Inflammasomes in the Pathophysiology of Kidney Diseases ROS-Mediated NLRP3 Inflammasome Activation in Brain, Heart, Kidney, and Testis Ischemia/Reperfusion Injury How the Innate Immune System Senses Trouble and Causes Trouble Inflammation in AKI: Current Understanding, Key Questions, and Knowledge Gaps.Sisters in arms: myeloid and tubular epithelial cells shape renal innate immunity.Chrysophanol inhibits NALP3 inflammasome activation and ameliorates cerebral ischemia/reperfusion in mice Bone mesenchymal stem cells ameliorate ischemia/reperfusion-induced damage in renal epithelial cells via microRNA-223 NLRP3 inflammasome mediates albumin-induced renal tubular injury through impaired mitochondrial function.Quercetin and allopurinol ameliorate kidney injury in STZ-treated rats with regulation of renal NLRP3 inflammasome activation and lipid accumulation.The emerging role of the inflammasome in kidney diseases Deletion of the innate immune NLRP3 receptor abolishes cardiac ischemic preconditioning and is associated with decreased Il-6/STAT3 signaling.Inflammasome activation mediates inflammation and outcome in humans and mice with pneumococcal meningitis.NLRP3 inflammasome: from a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases.NLRP3 protein deficiency exacerbates hyperoxia-induced lethality through Stat3 protein signaling independent of interleukin-1β NLRP3 mediates NF-κB activation and cytokine induction in microbially induced and sterile inflammation Role of NLRP3 Inflammasomes for Rhabdomyolysis-induced Acute Kidney Injury An update on the role of the inflammasomes in the pathogenesis of kidney diseases.Innate immunity in donor procurement.Inflammasome-Independent NLRP3 Restriction of a Protective Early Neutrophil Response to Pulmonary Tularemia.Calcium oxalate crystals induce renal inflammation by NLRP3-mediated IL-1β secretion The Hepatitis C Virus-induced NLRP3 Inflammasome Activates the Sterol Regulatory Element-binding Protein (SREBP) and Regulates Lipid Metabolism.Involvement of endoplasmic reticulum stress in angiotensin II-induced NLRP3 inflammasome activation in human renal proximal tubular cells in vitro Characterization and Activation of NLRP3 Inflammasomes in the Renal Medulla in Mice.NLRP3 Deficiency Reduces Macrophage Interleukin-10 Production and Enhances the Susceptibility to Doxorubicin-induced Cardiotoxicity.The NLRP3 Inflammasome as a novel player of the intercellular crosstalk in metabolic disorders.Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.NLRP3 protects alveolar barrier integrity by an inflammasome-independent increase of epithelial cell adherence.NALP3-mediated inflammation is a principal cause of progressive renal failure in oxalate nephropathy.Histones activate the NLRP3 inflammasome in Kupffer cells during sterile inflammatory liver injury.Divergence of IL-1, IL-18, and cell death in NLRP3 inflammasomopathies.Endoplasmic Reticulum Chaperon Tauroursodeoxycholic Acid Attenuates Aldosterone-Infused Renal Injury.NLRP3 inflammasome mediates contrast media-induced acute kidney injury by regulating cell apoptosis.Anti-inflammatory interventions-what has worked, not worked, and what may work in the future.Danggui Buxue Tang Attenuates Tubulointerstitial Fibrosis via Suppressing NLRP3 Inflammasome in a Rat Model of Unilateral Ureteral Obstruction.NLRP3 inflammasome knockout mice are protected against ischemic but not cisplatin-induced acute kidney injury.Nlrp3 prevents early renal interstitial edema and vascular permeability in unilateral ureteral obstruction.Activation of inflammasomes in podocyte injury of mice on the high fat diet: Effects of ASC gene deletion and silencing The role of innate immunity in donor organ procurement.Cross-regulation between the IL-1β/IL-18 processing inflammasome and other inflammatory cytokines.

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P2860

An inflammasome-independent role for epithelial-expressed Nlrp3 in renal ischemia-reperfusion injury

http://www.wikidata.org/entity/Q34485850

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-sg

2010年學術文章

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name

An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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JIMMUNOL.1002330

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Journal of Immunology

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An inflammasome-independent ro ...... al ischemia-reperfusion injury

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Alana A Shigeoka

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Amanpreet Kambo

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Andrew J King

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Dianne B McKay

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Hal M Hoffman

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James L Mueller

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Jean da Silva Correia

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John C Mathison

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Richard J Ulevitch

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Wesley F Hall

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P2860

Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome

Chronic infantile neurological cutaneous and articular syndrome is caused by mutations in CIAS1, a gene highly expressed in polymorphonuclear cells and chondrocytes

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Hearing improvement in a patient with variant Muckle-Wells syndrome in response to interleukin 1 receptor antagonism

Mechanism and repertoire of ASC-mediated gene expression

NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder

Inflammasome components NALP 1 and 3 show distinct but separate expression profiles in human tissues suggesting a site-specific role in the inflammatory response

De novo CIAS1 mutations, cytokine activation, and evidence for genetic heterogeneity in patients with neonatal-onset multisystem inflammatory disease (NOMID): a new member of the expanding family of pyrin-associated autoinflammatory diseases

Gout-associated uric acid crystals activate the NALP3 inflammasome

Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf

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6277-6285

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10.4049/JIMMUNOL.1002330

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10

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185

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2010-10-20T00:00:00Z

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47509977

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20962258

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reperfusion injury

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3020135

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