Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.
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The Nucleotide Oligomerization Domain-Like Receptors in Kidney InjuryInflammasomes in the Pathophysiology of Kidney DiseasesSecondary amyloidosis in autoinflammatory diseases and the role of inflammation in renal damageInflammation and hypertension: new understandings and potential therapeutic targetsChrysophanol inhibits NALP3 inflammasome activation and ameliorates cerebral ischemia/reperfusion in miceEndothelial NLRP3 inflammasome activation and arterial neointima formation associated with acid sphingomyelinase during hypercholesterolemiaNod-like receptor protein 3 (NLRP3) inflammasome activation and podocyte injury via thioredoxin-interacting protein (TXNIP) during hyperhomocysteinemiaThe emerging role of the inflammasome in kidney diseasesControl of autophagy maturation by acid sphingomyelinase in mouse coronary arterial smooth muscle cells: protective role in atherosclerosisThe podocyte power-plant disaster and its contribution to glomerulopathy.Nlrp3-inflammasome activation in non-myeloid-derived cells aggravates diabetic nephropathyEndothelial Nlrp3 inflammasome activation associated with lysosomal destabilization during coronary arteritis.Expression pattern of NLRP3 and its related cytokines in the lung and brain of avian influenza virus H9N2 infected BALB/c mice.NADPH oxidase-induced NALP3 inflammasome activation is driven by thioredoxin-interacting protein which contributes to podocyte injury in hyperglycemiaRedox regulation of NLRP3 inflammasomes: ROS as trigger or effector?Involvement of the NLRC4-Inflammasome in Diabetic Nephropathy.Instigation of endothelial Nlrp3 inflammasome by adipokine visfatin promotes inter-endothelial junction disruption: role of HMGB1.NADPH oxidase-mediated triggering of inflammasome activation in mouse podocytes and glomeruli during hyperhomocysteinemia.Mesenchymal stem cell transplantation inhibited high salt-induced activation of the NLRP3 inflammasome in the renal medulla in Dahl S ratsCharacterization and Activation of NLRP3 Inflammasomes in the Renal Medulla in Mice.Inhibition of hyperhomocysteinemia-induced inflammasome activation and glomerular sclerosis by NLRP3 gene deletion.Protective Action of Anandamide and Its COX-2 Metabolite against l-Homocysteine-Induced NLRP3 Inflammasome Activation and Injury in Podocytes.Defective homocysteine metabolism: potential implications for skeletal muscle malfunctionInstigation of NLRP3 inflammasome activation and glomerular injury in mice on the high fat diet: role of acid sphingomyelinase geneNLRP3 inflammasome mediates contrast media-induced acute kidney injury by regulating cell apoptosis.Role of NADPH oxidase-mediated reactive oxygen species in podocyte injury.NLRP3 inflammasome: Pathogenic role and potential therapeutic target for IgA nephropathy.Contribution of endogenously produced reactive oxygen species to the activation of podocyte NLRP3 inflammasomes in hyperhomocysteinemia.Innate Immune Activity in Glomerular PodocytesDefective autophagosome trafficking contributes to impaired autophagic flux in coronary arterial myocytes lacking CD38 gene.Expression of specific inflammasome gene modules stratifies older individuals into two extreme clinical and immunological states.Enhancement of endothelial permeability by free fatty acid through lysosomal cathepsin B-mediated Nlrp3 inflammasome activation.Activation of inflammasomes in podocyte injury of mice on the high fat diet: Effects of ASC gene deletion and silencingCanonical and non-canonical effects of the NLRP3 inflammasome in kidney inflammation and fibrosis.Hyperuricemia-induced NLRP3 activation of macrophages contributes to the progression of diabetic nephropathy.Activation of NLRP3 inflammasomes contributes to hyperhomocysteinemia-aggravated inflammation and atherosclerosis in apoE-deficient mice.NLRP3 inflammasome activation contributes to aldosterone-induced podocyte injury.The NLRP3 inflammasome in kidney disease and autoimmunity.Inflammasome Activation in Chronic Glomerular Diseases.NLRP3 Localizes to the Tubular Epithelium in Human Kidney and Correlates With Outcome in IgA Nephropathy
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Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 29 May 2012
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@en
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@nl
type
label
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@en
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@nl
prefLabel
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@en
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@nl
P2093
P2860
P1433
P1476
Activation of Nod-like recepto ...... rosis in hyperhomocysteinemia.
@en
P2093
Chun Zhang
Justine M Abais
Krishna M Boini
Pin-Lan Li
Qinglian Liu
P2860
P304
P356
10.1161/HYPERTENSIONAHA.111.189688
P407
P577
2012-05-29T00:00:00Z