Synovial membrane cytokine expression is predictive of joint damage progression in rheumatoid arthritis: a two-year prospective study (the DAMAGE study cohort).
about
IL-17RC: a partner in IL-17 signaling and beyondThe Interplay Between Monocytes/Macrophages and CD4(+) T Cell Subsets in Rheumatoid Arthritisβ-catenin/TCF-1 pathway in T cell development and differentiationSkewed Helper T-Cell Responses to IL-12 Family Cytokines Produced by Antigen-Presenting Cells and the Genetic Background in Behcet's DiseaseA role for altered phagosome maturation in the long-term persistence of Helicobacter pylori infectionEffect of IL-17 receptor A blockade with brodalumab in inflammatory diseasesThe IL-17 pathway as a major therapeutic target in autoimmune diseasesRNA plasticity and selectivity applicable to therapeutics and novel biosensor developmentMyeloid-derived suppressor cells are proinflammatory and regulate collagen-induced arthritis through manipulating Th17 cell differentiation.Location of CD4+ T cell priming regulates the differentiation of Th1 and Th17 cells and their contribution to arthritis.Increase in circulating Th17 cells during anti-TNF therapy is associated with ultrasonographic improvement of synovitis in rheumatoid arthritisIL-17 induces monocyte migration in rheumatoid arthritisLinking power Doppler ultrasound to the presence of th17 cells in the rheumatoid arthritis jointTherapeutic potential of anti-interleukin-17A aptamer: suppression of interleukin-17A signaling and attenuation of autoimmunity in two mouse models.Leucine-rich alpha 2 glycoprotein promotes Th17 differentiation and collagen-induced arthritis in mice through enhancement of TGF-β-Smad2 signaling in naïve helper T cells.Th17 cells and activated dendritic cells are increased in vitiligo lesionsCritical role of TCF-1 in repression of the IL-17 gene.Interleukins and interleukin receptors in rheumatoid arthritis: Research, diagnostics and clinical implications.A genetic variant in the IL-17 promoter is functionally associated with acute graft-versus-host disease after unrelated bone marrow transplantation.IL-17RA signaling amplifies antibody-induced arthritis.IRF4 and its regulators: evolving insights into the pathogenesis of inflammatory arthritis?Interleukin-17+CD8+ T cells are enriched in the joints of patients with psoriatic arthritis and correlate with disease activity and joint damage progressionTofacitinib for the treatment of rheumatoid arthritis.Human CCR4+ CCR6+ Th17 cells suppress autologous CD8+ T cell responses.Rheumatoid synovial fluid interleukin-17-producing CD4 T cells have abundant tumor necrosis factor-alpha co-expression, but little interleukin-22 and interleukin-23R expression.Identification of a cytokine network sustaining neutrophil and Th17 activation in untreated early rheumatoid arthritis.IL-17A is increased in the serum and in spinal cord CD8 and mast cells of ALS patients.Tissue specific CD4+ T cell priming determines the requirement for interleukin-23 in experimental arthritisTh17 cells can provide B cell help in autoantibody induced arthritis.Development of a nanoparticulate formulation of retinoic acid that suppresses Th17 cells and upregulates regulatory T cellsHeterogeneous expression pattern of interleukin 17A (IL-17A), IL-17F and their receptors in synovium of rheumatoid arthritis, psoriatic arthritis and osteoarthritis: possible explanation for nonresponse to anti-IL-17 therapy?The role of interleukin-17 in the pathogenesis of rheumatoid arthritis.IL-23 receptor regulation by Let-7f in human CD4+ memory T cells.Percentages of CD4+CD161+ and CD4-CD8-CD161+ T cells in the synovial fluid are correlated with disease activity in rheumatoid arthritisAntigen-specific blocking of CD4-specific immunological synapse formation using BPI and current therapies for autoimmune diseases.Cells of the synovium in rheumatoid arthritis. T lymphocytes.Th17 cells in human disease.Exacerbation of collagen induced arthritis by Fcγ receptor targeted collagen peptide due to enhanced inflammatory chemokine and cytokine productionFrequency of Th17 CD20+ cells in the peripheral blood of rheumatoid arthritis patients is higher compared to healthy subjects.Kinsenoside inhibits the inflammatory mediator release in a type-II collagen induced arthritis mouse model by regulating the T cells responses.
P2860
Q24629783-43F0D41D-4B9F-44C6-8687-28D4C5DA4B46Q26774681-C8DD44BD-8E90-4AC1-9D49-93DDA6A42948Q26821842-473B6950-B226-4214-8F7D-6330209411E8Q26866098-9CFFA2FC-AF0F-452A-A0AB-C6C64C4CD8FDQ26996022-A88700AF-2462-46BA-999E-57D9061F8847Q28076496-803F4F30-D055-4B72-9E1E-E1287BF18AF7Q28300826-E5CCCFCB-B5CD-451E-9F73-54192906B043Q28727743-0DC3071D-0DF7-40F3-9817-FB02F9A20971Q30300780-D77416FE-CCD4-4EE6-ACB8-E7C0FBD9BD6CQ30412824-E15534BA-45FF-4369-87C5-3B3D2E400BDEQ30832987-2C868329-7CA1-4D36-A0E7-1F0880132EA1Q33610883-49A93A58-63CA-466C-829D-09E4C84FFEE6Q33687116-9EBEBE83-CC32-4CEC-92F2-BD697169CADCQ33725862-A09F7422-B5E0-4776-B20C-A037276BB91CQ33802290-B8E733DF-777E-436D-AB6B-748BABDE2FB9Q33889471-BDED2137-80E4-4D4D-9731-0FFD42074840Q34026243-E22A5DB4-063F-466F-831C-DD7F2F8AC8DFQ34047753-BDA15A32-6181-40F9-9207-49356A53AED2Q34058468-48192DA4-2910-46EB-8E57-32119630F47DQ34058535-CD4E7F42-0B3F-4924-910A-8B0899534C37Q34062484-F8DCC84A-94A3-423C-9A4E-4153EB23B742Q34154244-75DCA0C8-9BBA-4383-8982-17E929445737Q34275878-D1BB8A33-8BEE-4808-A0E7-B6A736277F4DQ34276831-971F8C6E-5722-41C5-870B-F247F064CE9DQ34346316-A5FF55F0-8C23-45FC-8045-9C92AFA20A54Q34346409-7774101D-2CF7-43A7-B5A1-4CE17D85C662Q34351001-2CBF1BE2-D898-433F-9E48-44FC9E3E4259Q34375258-7E4949C1-715E-490E-ACC2-80CDB315D7E2Q34565713-7BFA9825-BFFB-4174-88CC-544ADD7C1BDAQ34707563-CB1F3144-E2BF-445C-A1D4-E10E6E662271Q34937925-C6E7B463-E626-4A88-9718-B49172677A84Q35004526-AAA1FD02-6ED9-496B-A91A-918B308C5993Q35098317-8F39728C-016A-4239-B3E2-A8F32ACFE9ADQ35557267-845D9C3E-6EF4-4F73-B849-0E0C7B21489BQ35637379-647DDC66-CA63-4984-B698-3DA6FA18676EQ35769049-8C6FA1B2-AF1C-4E9F-AF4F-BFA053F8278AQ35820734-D21DFAAC-63A5-476A-B55F-11C06A365037Q35905956-3BFA4599-D378-4DB1-8EFA-A1A177EC58ECQ35907830-07935766-039F-4541-BDFB-EEC299AFCE3EQ35936285-97A3FC27-82C0-4B37-90F6-4021305F3129
P2860
Synovial membrane cytokine expression is predictive of joint damage progression in rheumatoid arthritis: a two-year prospective study (the DAMAGE study cohort).
description
2006 nî lūn-bûn
@nan
2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
name
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@ast
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@en
Synovial membrane cytokine exp ...... : a two-year prospective study
@nl
type
label
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@ast
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@en
Synovial membrane cytokine exp ...... : a two-year prospective study
@nl
prefLabel
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@ast
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@en
Synovial membrane cytokine exp ...... : a two-year prospective study
@nl
P2093
P356
P1476
Synovial membrane cytokine exp ...... udy (the DAMAGE study cohort).
@en
P2093
Bruce W Kirkham
C Soon Lee
Ian J Portek
John P Edmonds
Katherine M Juhasz
Marissa N Lassere
Paul A Bird
Ron Shnier
P304
P356
10.1002/ART.21749
P577
2006-04-01T00:00:00Z