Efficient TGF-β/SMAD signaling in human melanoma cells associated with high c-SKI/SnoN expression.
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Other targeted drugs in melanomaCell density sensing alters TGF-β signaling in a cell-type-specific manner, independent from Hippo pathway activationLong-term miR-29b suppression reduces aneurysm formation in a Marfan mouse model.Molecular mechanism of smurf2 in regulating the expression of SnoN in diabetic nephropathy.The Pathogenic Transforming Growth Factor-β Overdrive Hypothesis in Aortic Aneurysms and Dissections: A Mirage?Protective Effect of Triptolide against Glomerular Mesangial Cell Proliferation and Glomerular Fibrosis in Rats Involves the TGF- β 1/Smad Signaling Pathway.Oxymatrine Inhibits Renal Tubular EMT Induced by High Glucose via Upregulation of SnoN and Inhibition of TGF-β1/Smad Signaling Pathway.Differential role of Sloan-Kettering Institute (Ski) protein in Nodal and transforming growth factor-beta (TGF-β)-induced Smad signaling in prostate cancer cells.Insights into the Transforming Growth Factor-β Signaling Pathway in Cutaneous Melanoma.Expression of microphthalmia-associated transcription factor (MITF), which is critical for melanoma progression, is inhibited by both transcription factor GLI2 and transforming growth factor-β.A simple feature construction method for predicting upstream/downstream signal flow in human protein-protein interaction networks.GLI2 cooperates with ZEB1 for transcriptional repression of CDH1 expression in human melanoma cells.Mutational activation of BRAF confers sensitivity to transforming growth factor beta inhibitors in human cancer cells.Combined treatment with a transforming growth factor beta inhibitor (1D11) and bortezomib improves bone architecture in a mouse model of myeloma-induced bone disease.The mechanism of TGF-β/miR-155/c-Ski regulates endothelial-mesenchymal transition in human coronary artery endothelial cells.Herbal compound 861 prevents hepatic fibrosis by inhibiting the TGF-β1/Smad/SnoN pathway in bile duct-ligated rats.Upregulation of ski in fibroblast is implicated in the peroxisome proliferator--activated receptor δ-mediated wound healing.Ski modulate the characteristics of pancreatic cancer stem cells via regulating sonic hedgehog signaling pathway.MEK inhibition prevents tumour-shed transforming growth factor-β-induced T-regulatory cell augmentation in tumour milieu.
P2860
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P2860
Efficient TGF-β/SMAD signaling in human melanoma cells associated with high c-SKI/SnoN expression.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@ast
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@en
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@nl
type
label
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@ast
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@en
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@nl
prefLabel
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@ast
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@en
Efficient TGF-β/SMAD signaling ...... th high c-SKI/SnoN expression.
@nl
P2093
P2860
P356
P1433
P1476
Efficient TGF-β/SMAD signaling ...... ith high c-SKI/SnoN expression
@en
P2093
Erwan Le Scolan
Kunxin Luo
Leon van Kempen
Vasileia I Alexaki
P2860
P2888
P356
10.1186/1476-4598-10-2
P577
2011-01-06T00:00:00Z
P5875
P6179
1020792889