Thrombospondin-1 inhibition of vascular smooth muscle cell responses occurs via modulation of both cAMP and cGMP.
about
Therapeutic opportunities for targeting the ubiquitous cell surface receptor CD47Activated CD47 regulates multiple vascular and stress responses: implications for acute kidney injury and its managementCD47 update: a multifaceted actor in the tumour microenvironment of potential therapeutic interestPADPIN: protein-protein interaction networks of angiogenesis, arteriogenesis, and inflammation in peripheral arterial disease.Age-dependent regulation of skeletal muscle mitochondria by the thrombospondin-1 receptor CD47.CD47 deficiency protects mice from diet-induced obesity and improves whole body glucose tolerance and insulin sensitivity.The matricellular protein thrombospondin-1 globally regulates cardiovascular function and responses to stress via CD47.Loss-of-function thrombospondin-1 mutations in familial pulmonary hypertensionCD47 blockade reduces ischemia/reperfusion injury and improves survival in a rat liver transplantation model.Transcriptional and Post-Transcriptional Regulation of Thrombospondin-1 Expression: A Computational Model.CD47 signaling pathways controlling cellular differentiation and responses to stress.Valproic acid shows a potent antitumor effect with alteration of DNA methylation in neuroblastomaThe role of circulating thrombospondin-1 in patients with precapillary pulmonary hypertensionRegulation of soluble guanylate cyclase by matricellular thrombospondins: implications for blood flow.Thrombospondin-1 and CD47 regulation of cardiac, pulmonary and vascular responses in health and disease.Thrombospondin-1, free radicals, and the coronary microcirculation: the aging conundrum.Matricellular protein thrombospondin-1 in pulmonary hypertension: multiple pathways to disease.Thrombospondins: A Role in Cardiovascular Disease.HIF-2α-mediated induction of pulmonary thrombospondin-1 contributes to hypoxia-driven vascular remodelling and vasoconstrictionThe effect of cyclic nucleotide analog drugs on the mediators release from basophils.Physiological levels of thrombospondin-1 decrease NO-dependent vasodilation in coronary microvessels from aged rats.GalT-KO pig lungs are highly susceptible to acute vascular rejection in baboons, which may be mitigated by transgenic expression of hCD47 on porcine blood vessels.
P2860
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P2860
Thrombospondin-1 inhibition of vascular smooth muscle cell responses occurs via modulation of both cAMP and cGMP.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@ast
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@en
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@nl
type
label
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@ast
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@en
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@nl
prefLabel
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@ast
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@en
Thrombospondin-1 inhibition of ...... ulation of both cAMP and cGMP.
@nl
P2860
P1476
Thrombospondin-1 inhibition of ...... dulation of both cAMP and cGMP
@en
P2093
Jeff S Isenberg
Mingyi Yao
P2860
P356
10.1016/J.PHRS.2010.10.014
P577
2010-10-29T00:00:00Z