Hypersusceptibility of cystic fibrosis mice to chronic Pseudomonas aeruginosa oropharyngeal colonization and lung infection. - Wikidata - wikimedia - TriplyDB

Hypersusceptibility of cystic fibrosis mice to chronic Pseudomonas aeruginosa oropharyngeal colonization and lung infection.

Hypersusceptibility of cystic fibrosis mice to chronic Pseudomonas aeruginosa oropharyngeal colonization and lung infection.

http://www.wikidata.org/entity/Q34531184

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Pseudomonas aeruginosa-plant root interactions. Pathogenicity, biofilm formation, and root exudation Trehalose biosynthesis promotes Pseudomonas aeruginosa pathogenicity in plants Proteolytic regulation of alginate overproduction in Pseudomonas aeruginosa.The galU Gene of Pseudomonas aeruginosa is required for corneal infection and efficient systemic spread following pneumonia but not for infection confined to the lung.Genes required for and effects of alginate overproduction induced by growth of Pseudomonas aeruginosa on Pseudomonas isolation agar supplemented with ammonium metavanadate.Vanadate and triclosan synergistically induce alginate production by Pseudomonas aeruginosa strain PAO1.Cross-sectional analysis of clinical and environmental isolates of Pseudomonas aeruginosa: biofilm formation, virulence, and genome diversity Novel mouse model of chronic Pseudomonas aeruginosa lung infection mimicking cystic fibrosis Caveolin-1 modifies the immunity to Pseudomonas aeruginosa The Pseudomonas aeruginosa reference strain PA14 displays increased virulence due to a mutation in ladS.Nonmucoid Pseudomonas aeruginosa expresses alginate in the lungs of patients with cystic fibrosis and in a mouse model.ClpXP proteases positively regulate alginate overexpression and mucoid conversion in Pseudomonas aeruginosa Expression of S100A8 correlates with inflammatory lung disease in congenic mice deficient of the cystic fibrosis transmembrane conductance regulator.Protection of Cftr knockout mice from acute lung infection by a helper-dependent adenoviral vector expressing Cftr in airway epithelia.Azithromycin inhibits expression of the GacA-dependent small RNAs RsmY and RsmZ in Pseudomonas aeruginosa Construction and characterization of a Pseudomonas aeruginosa mucoid exopolysaccharide-alginate conjugate vaccine.Azithromycin reduces spontaneous and induced inflammation in DeltaF508 cystic fibrosis mice.Emerging drug treatments for cystic fibrosis.Glycosylation of Pseudomonas aeruginosa strain Pa5196 type IV pilins with mycobacterium-like alpha-1,5-linked d-Araf oligosaccharides.Pseudomonas aeruginosa lipopolysaccharide: a major virulence factor, initiator of inflammation and target for effective immunity.The Hybrid Histidine Kinase LadS Forms a Multicomponent Signal Transduction System with the GacS/GacA Two-Component System in Pseudomonas aeruginosa Application of vaccine technology to prevention of Pseudomonas aeruginosa infections.A dynamic and intricate regulatory network determines Pseudomonas aeruginosa virulence.Modification of Pseudomonas aeruginosa Pa5196 type IV Pilins at multiple sites with D-Araf by a novel GT-C family Arabinosyltransferase, TfpW.Airway epithelial control of Pseudomonas aeruginosa infection in cystic fibrosis.Rational design of engineered cationic antimicrobial peptides consisting exclusively of arginine and tryptophan, and their activity against multidrug-resistant pathogens.IL1B polymorphisms modulate cystic fibrosis lung disease.Principles of bone marrow transplantation (BMT): providing optimal veterinary and husbandry care to irradiated mice in BMT studies.Animal models of chronic lung infection with Pseudomonas aeruginosa: useful tools for cystic fibrosis studies.Regulation of virulence and antibiotic resistance by two-component regulatory systems in Pseudomonas aeruginosa.Novel experimental Pseudomonas aeruginosa lung infection model mimicking long-term host-pathogen interactions in cystic fibrosis.Inescapable need for neutrophils as mediators of cellular innate immunity to acute Pseudomonas aeruginosa pneumonia.Analysis of acquisition of Pseudomonas aeruginosa gastrointestinal mucosal colonization and horizontal transmission in a murine model.Lipids in cystic fibrosis.Immune responses in cystic fibrosis: are they intrinsically defective?Cell-surface signaling in Pseudomonas: stress responses, iron transport, and pathogenicity.N-glycosylation augmentation of the cystic fibrosis epithelium improves Pseudomonas aeruginosa clearance.Resistance to Pseudomonas aeruginosa chronic lung infection requires cystic fibrosis transmembrane conductance regulator-modulated interleukin-1 (IL-1) release and signaling through the IL-1 receptor.Plasmid carriage can limit bacteria-phage coevolution.Comparison of haematopoietic stem cell engraftment through the retro-orbital venous sinus and the lateral vein: alternative routes for bone marrow transplantation in mice.

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Hypersusceptibility of cystic fibrosis mice to chronic Pseudomonas aeruginosa oropharyngeal colonization and lung infection.

http://www.wikidata.org/entity/Q34531184

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2003 nî lūn-bûn

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Hypersusceptibility of cystic ...... lonization and lung infection.

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Carolyn L Cannon

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Christopher Ray

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Fadie T Coleman

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Simone Mueschenborn

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Vincent J Carey

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P2860

Use of model plant hosts to identify Pseudomonas aeruginosa virulence factors

Pathophysiology of gene-targeted mouse models for cystic fibrosis

Pseudomonas aeruginosa GacA, a factor in multihost virulence, is also essential for biofilm formation

CFTR is a pattern recognition molecule that extracts Pseudomonas aeruginosa LPS from the outer membrane into epithelial cells and activates NF-kappa B translocation

CFTR mutations and host susceptibility to Pseudomonas aeruginosa lung infection.

A mouse model for the delta F508 allele of cystic fibrosis.

Longitudinal study of antibody response to lipopolysaccharides during chronic Pseudomonas aeruginosa lung infection in cystic fibrosis

Longitudinal study of immune response to Pseudomonas aeruginosa antigens in cystic fibrosis.

Polysaccharide surface antigens expressed by nonmucoid isolates of Pseudomonas aeruginosa from cystic fibrosis patients

Excessive inflammatory response of cystic fibrosis mice to bronchopulmonary infection with Pseudomonas aeruginosa

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