Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells. - Wikidata - wikimedia - TriplyDB

Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells.

Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells.

http://www.wikidata.org/entity/Q34554622

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PDE4 inhibitors as potential therapeutic agents in the treatment of COPD-focus on roflumilast Cilomilast: orally active selective phosphodiesterase-4 inhibitor for treatment of chronic obstructive pulmonary disease.Cystic fibrosis transmembrane conductance regulator activation by roflumilast contributes to therapeutic benefit in chronic bronchitis.Psychological factors in asthma Epidermal growth factor receptor copy number in potentially malignant oral disorders and oral squamous cell carcinoma: a short communication and preliminary study.Respiratory syncytial virus inhibits ciliagenesis in differentiated normal human bronchial epithelial cells: effectiveness of N-acetylcysteine.Roflumilast inhibits respiratory syncytial virus infection in human differentiated bronchial epithelial cells Doxofylline: a promising methylxanthine derivative for the treatment of asthma and chronic obstructive pulmonary disease.Caffeine and rolipram affect Smad signalling and TGF-β1 stimulated CTGF and transgelin expression in lung epithelial cells.Role of circulating miRNAs as biomarkers in idiopathic pulmonary arterial hypertension: possible relevance of miR-23a.Systemic inflammatory profile and response to anti-tumor necrosis factor therapy in chronic obstructive pulmonary disease.Use of a soluble epoxide hydrolase inhibitor in smoke-induced chronic obstructive pulmonary disease.Glucagon Like Peptide-1 (GLP-1) Modulates OVA-Induced Airway Inflammation and Mucus Secretion Involving a Protein Kinase A (PKA)-Dependent Nuclear Factor-κB (NF-κB) Signaling Pathway in Mice.PGC-1α induction in pulmonary arterial hypertension Emerging drugs for the treatment of chronic obstructive pulmonary disease.Roflumilast: a phosphodiesterase-4 inhibitor for the treatment of respiratory disease.Anti-inflammatory effects of the novel inhaled phosphodiesterase type 4 inhibitor CHF6001 on virus-inducible cytokines Novel medications for asthma: a look at the future.Autocrine Acetylcholine, Induced by IL-17A via NFκB and ERK1/2 Pathway Activation, Promotes MUC5AC and IL-8 Synthesis in Bronchial Epithelial Cells.Treating COPD with PDE 4 inhibitors.Is 'GOLD' standard for the management of COPD in clinical practice?Roflumilast in chronic obstructive pulmonary disease: evidence from large trials.IL-33 stimulates CXCL8/IL-8 secretion in goblet cells but not normally differentiated airway cells.Apigenin and Wogonin Regulate Epidermal Growth Factor Receptor Signaling Pathway Involved in MUC5AC Mucin Gene Expression and Production from Cultured Airway Epithelial Cells.New therapeutic options in the management of COPD - focus on roflumilast.Genetic basis for personalized medicine in asthma.Phosphodiesterase-4 inhibitor therapy for lung diseases.Multiple facets of cAMP signalling and physiological impact: cAMP compartmentalization in the lung.Targeting mucus hypersecretion: new therapeutic opportunities for COPD?Roles of roflumilast, a selective phosphodiesterase 4 inhibitor, in airway diseases.NPS2143 Inhibits MUC5AC and Proinflammatory Mediators in Cigarette Smoke Extract (CSE)-Stimulated Human Airway Epithelial Cells.The discovery of roflumilast for the treatment of chronic obstructive pulmonary disease.Melatonin inhibits MUC5AC production via suppression of MAPK signaling in human airway epithelial cells.Roflumilast N-oxide prevents cytokine secretion induced by cigarette smoke combined with LPS through JAK/STAT and ERK1/2 inhibition in airway epithelial cells.Roflumilast inhibits leukocyte-endothelial cell interactions, expression of adhesion molecules and microvascular permeability.Piclamilast inhibits the pro-apoptotic and anti-proliferative responses of A549 cells exposed to H(2)O(2) via mechanisms involving AP-1 activation.Roflumilast N-oxide, a PDE4 inhibitor, improves cilia motility and ciliated human bronchial epithelial cells compromised by cigarette smoke in vitro.Aclidinium inhibits human lung fibroblast to myofibroblast transition.Roflumilast, a phosphodiesterase 4 inhibitor, alleviates bleomycin-induced lung injury Roflumilast: systemic therapy for chronic obstructive pulmonary disease.

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P2860

Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells.

http://www.wikidata.org/entity/Q34554622

description

2005 nî lūn-bûn

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2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2005 թվականի փետրվարին հրատարակված գիտական հոդված

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2005年の論文

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Phosphodiesterase 4 inhibition ...... human airway epithelial cells.

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Phosphodiesterase 4 inhibition ...... human airway epithelial cells.

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Phosphodiesterase 4 inhibition ...... human airway epithelial cells.

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Phosphodiesterase 4 inhibition ...... human airway epithelial cells.

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Phosphodiesterase 4 inhibition ...... human airway epithelial cells.

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Buenestado A

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P2860

Bronchodilator and anti-inflammatory activities of glaucine: In vitro studies in human airway smooth muscle and polymorphonuclear leukocytes

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Inhibition of forskolin-induced neurite outgrowth and protein phosphorylation by a newly synthesized selective inhibitor of cyclic AMP-dependent protein kinase, N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinolinesulfonamide (H-89), of PC12D pheochromoc

Oxidative stress causes mucin synthesis via transactivation of epidermal growth factor receptor: role of neutrophils

Inhibition of phosphodiesterase 4 attenuates airway hyperresponsiveness and airway inflammation in a model of secondary allergen challenge.

Development status of second generation PDE4 inhibitors for asthma and COPD: the story so far.

Epidermal growth factor system regulates mucin production in airways.

Increasing cAMP attenuates activation of mitogen-activated protein kinase.

Inhibition of focal adhesion kinase expression or activity disrupts epidermal growth factor-stimulated signaling promoting the migration of invasive human carcinoma cells.

Tobacco smoke control of mucin production in lung cells requires oxygen radicals AP-1 and JNK.

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