A molecular basis for hereditary motor and sensory neuropathy disorders.
about
Pharmacological induction of the heat shock response improves myelination in a neuropathic modelMyelin protein zero/P0 phosphorylation and function require an adaptor protein linking it to RACK1 and PKC alpha.Mutations in the cytoplasmic domain of P0 reveal a role for PKC-mediated phosphorylation in adhesion and myelinationRapamycin activates autophagy and improves myelination in explant cultures from neuropathic mice.Conduction block in PMP22 deficiency.The Gdap1 knockout mouse mechanistically links redox control to Charcot-Marie-Tooth disease.
P2860
A molecular basis for hereditary motor and sensory neuropathy disorders.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
A molecular basis for hereditary motor and sensory neuropathy disorders.
@ast
A molecular basis for hereditary motor and sensory neuropathy disorders.
@en
A molecular basis for hereditary motor and sensory neuropathy disorders.
@nl
type
label
A molecular basis for hereditary motor and sensory neuropathy disorders.
@ast
A molecular basis for hereditary motor and sensory neuropathy disorders.
@en
A molecular basis for hereditary motor and sensory neuropathy disorders.
@nl
prefLabel
A molecular basis for hereditary motor and sensory neuropathy disorders.
@ast
A molecular basis for hereditary motor and sensory neuropathy disorders.
@en
A molecular basis for hereditary motor and sensory neuropathy disorders.
@nl
P2093
P2860
P1476
A molecular basis for hereditary motor and sensory neuropathy disorders
@en
P2093
P2860
P2888
P356
10.1007/S11910-001-0079-6
P50
P577
2001-01-01T00:00:00Z
P6179
1022655244