Modulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injury - Wikidata - wikimedia - TriplyDB

Modulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injury

Modulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injury

http://www.wikidata.org/entity/Q34569187

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The liver X receptor agonist GW3965 improves recovery from mild repetitive traumatic brain injury in mice partly through apolipoprotein E Molecular mechanisms of cognitive dysfunction following traumatic brain injury Effects of low-level blast exposure on the nervous system: is there really a controversy?Chronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.Cerebral blood flow changes after brain injury in human amyloid-beta knock-in mice Acute blast injury reduces brain abeta in two rodent species.A new avenue for lithium: intervention in traumatic brain injury.Juvenile traumatic brain injury induces long-term perivascular matrix changes alongside amyloid-beta accumulation.A neurovascular perspective for long-term changes after brain trauma.Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury.Apolipoprotein E Regulates Injury-Induced Activation of Hippocampal Neural Stem and Progenitor Cells.Regulation of cerebral cholesterol metabolism in Alzheimer disease.Dissociated sterol-based liver X receptor agonists as therapeutics for chronic inflammatory diseases.Subacute Changes in Cleavage Processing of Amyloid Precursor Protein and Tau following Penetrating Traumatic Brain Injury Controlled cortical impact results in an extensive loss of dendritic spines that is not mediated by injury-induced amyloid-beta accumulation.Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model.Blood-brain barrier pathophysiology in traumatic brain injury.Liver X receptor modulators: a review of recently patented compounds (2009 - 2012).Common Pesticide, Dichlorodiphenyltrichloroethane (DDT), Increases Amyloid-β Levels by Impairing the Function of ABCA1 and IDE: Implication for Alzheimer's Disease.Differential regulation of ABCA1 and ABCG1 gene expressions in the remodeling mouse hippocampus after entorhinal cortex lesion and liver-X receptor agonist treatment Traumatic brain injury, microglia, and Beta amyloid.Frontiers of Complex Disease Mechanisms: Membrane Surface Tension May Link Genotype to Phenotype in Glaucoma.Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury.

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P2860

Modulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injury

http://www.wikidata.org/entity/Q34569187

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

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Journal of Neurotrauma

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Modulation of ABCA1 by an LXR ...... e after traumatic brain injury

@en

P2093

Alan I Faden

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Ana Pocivavsek

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G William Rebeck

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Hyang-Sook Hoe

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Ibolja Cernak

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Karen E Duff

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Lilit Vardanian

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Patricia M Washington

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P2860

ApoE promotes the proteolytic degradation of Abeta

The cell death-promoting gene DP5, which interacts with the BCL2 family, is induced during neuronal apoptosis following exposure to amyloid beta protein

Reciprocal regulation of inflammation and lipid metabolism by liver X receptors

The liver X receptor ligand T0901317 decreases amyloid beta production in vitro and in a mouse model of Alzheimer's disease

Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury.

beta-Amyloid stimulation of microglia and monocytes results in TNFalpha-dependent expression of inducible nitric oxide synthase and neuronal apoptosis.

Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.

Liver X receptor agonist treatment ameliorates amyloid pathology and memory deficits caused by high-fat diet in APP23 mice.

Interleukin 1 regulates synthesis of amyloid beta-protein precursor mRNA in human endothelial cells.

Neurotoxicity of a fragment of the amyloid precursor associated with Alzheimer's disease.

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225-236

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10.1089/NEU.2010.1595

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2

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28

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21175399

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traumatic brain injury

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3037807

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