Entry of newly synthesized GLUT4 into the insulin-responsive storage compartment is GGA dependent.
about
GDI-1 preferably interacts with Rab10 in insulin-stimulated GLUT4 translocationGapex-5, a Rab31 guanine nucleotide exchange factor that regulates Glut4 trafficking in adipocytes.Modulation of muscle regeneration, myogenesis, and adipogenesis by the Rho family guanine nucleotide exchange factor GEFT.The glucose transporter 4-regulating protein TUG is essential for highly insulin-responsive glucose uptake in 3T3-L1 adipocytesThe F-BAR protein CIP4 promotes GLUT4 endocytosis through bidirectional interactions with N-WASp and Dynamin-2Mapping of R-SNARE function at distinct intracellular GLUT4 trafficking steps in adipocytesSelf-assembly of Glut4 storage vesicles during differentiation of 3T3-L1 adipocytesInsulin stimulates membrane fusion and GLUT4 accumulation in clathrin coats on adipocyte plasma membranes.The GLUT4 codeHigh basal cell surface levels of fish GLUT4 are related to reduced sensitivity of insulin-induced translocation toward GGA and AS160 inhibition in adipocytes.Insulin-stimulated plasma membrane fusion of Glut4 glucose transporter-containing vesicles is regulated by phospholipase D1.Intracellular trafficking and secretion of adiponectin is dependent on GGA-coated vesicles.Tumor necrosis factor (TNF)-α-induced repression of GKAP42 protein levels through cGMP-dependent kinase (cGK)-Iα causes insulin resistance in 3T3-L1 adipocytesInsulin-regulated trafficking of GLUT4 requires ubiquitinationGolgin-160 is required for the Golgi membrane sorting of the insulin-responsive glucose transporter GLUT4 in adipocytes.The role of insulin-regulated aminopeptidase in MHC class I antigen presentation.Endoproteolytic cleavage of TUG protein regulates GLUT4 glucose transporter translocationInsulin stimulates the halting, tethering, and fusion of mobile GLUT4 vesicles in rat adipose cells.Glucose transporter 4: cycling, compartments and controversies.Ins (endocytosis) and outs (exocytosis) of GLUT4 traffickingPostreceptoral adipocyte insulin resistance induced by nelfinavir is caused by insensitivity of PKB/Akt to phosphatidylinositol-3,4,5-trisphosphate.Comparative prion disease gene expression profiling using the prion disease mimetic, cuprizone.A novel GGA-binding site is required for intracellular sorting mediated by stabilin-1.Biogenesis and regulation of insulin-responsive vesicles containing GLUT4.SNARE proteins underpin insulin-regulated GLUT4 traffic.Endosomal transport via ubiquitinationPosttranslational modifications of GLUT4 affect its subcellular localization and translocation.Trans, trans-farnesol as a mevalonate-derived inducer of murine 3T3-F442A pre-adipocyte differentiation.Insulin-stimulated exocytosis of GLUT4 is enhanced by IRAP and its partner tankyrase.Endosomal sorting of GLUT4 and Gap1 is conserved between yeast and insulin-sensitive cells.GGA function is required for maturation of neuroendocrine secretory granules.Recycling of IRAP from the plasma membrane back to the insulin-responsive compartment requires the Q-SNARE syntaxin 6 but not the GGA clathrin adaptors.Initial entry of IRAP into the insulin-responsive storage compartment occurs prior to basal or insulin-stimulated plasma membrane recycling.Adipocytes support cAMP-dependent translocation of aquaporin-2 from intracellular sites distinct from the insulin-responsive GLUT4 storage compartment.A specific dileucine motif is required for the GGA-dependent entry of newly synthesized insulin-responsive aminopeptidase into the insulin-responsive compartment.Microtubule network is required for insulin signaling through activation of Akt/protein kinase B: evidence that insulin stimulates vesicle docking/fusion but not intracellular mobility.Evidence that electrostatic interactions between vesicle-associated membrane protein 2 and acidic phospholipids may modulate the fusion of transport vesicles with the plasma membraneMolecular basis of insulin-responsive GLUT4 trafficking systems revealed by single molecule imaging.Entry of newly synthesized GLUT4 into the insulin-responsive storage compartment is dependent upon both the amino terminus and the large cytoplasmic loop.Phosphatidylinositol 4,5-bisphosphate regulates adipocyte actin dynamics and GLUT4 vesicle recycling.
P2860
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P2860
Entry of newly synthesized GLUT4 into the insulin-responsive storage compartment is GGA dependent.
description
2004 nî lūn-bûn
@nan
2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@ast
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@en
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@nl
type
label
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@ast
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@en
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@nl
prefLabel
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@ast
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@en
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@nl
P2093
P2860
P356
P1433
P1476
Entry of newly synthesized GLU ...... compartment is GGA dependent.
@en
P2093
Ahmir H Khan
Jeffrey E Pessin
June Chunqiu Hou
Konstantin V Kandror
Makoto Kanzaki
Megumi Furukawa
Robert T Watson
Shuichi Okada
P2860
P304
P356
10.1038/SJ.EMBOJ.7600159
P407
P577
2004-04-29T00:00:00Z