Granulovacuolar degeneration (GVD) bodies of Alzheimer's disease (AD) resemble late-stage autophagic organelles.
about
Neuropathological alterations in Alzheimer diseaseTangles, Toxicity, and Tau Secretion in AD - New Approaches to a Vexing ProblemEarly etiology of Alzheimer's disease: tipping the balance toward autophagy or endosomal dysfunction?Proteomics of protein post-translational modifications implicated in neurodegenerationMARK4 and MARK3 associate with early tau phosphorylation in Alzheimer's disease granulovacuolar degeneration bodies.TMEM106B expression is reduced in Alzheimer's disease brainsGranulovacuolar degenerations appear in relation to hippocampal phosphorylated tau accumulation in various neurodegenerative disorders.Phosphatidylinositol-4,5-bisphosphate is enriched in granulovacuolar degeneration bodies and neurofibrillary tanglesMolecular markers for granulovacuolar degeneration are present in rimmed vacuoles.Accumulation of amyloid-like Aβ1-42 in AEL (autophagy-endosomal-lysosomal) vesicles: potential implications for plaque biogenesis.Dual modification of Alzheimer's disease PHF-tau protein by lysine methylation and ubiquitylation: a mass spectrometry approach.Hydroxynonenal-generated crosslinking fluorophore accumulation in Alzheimer disease reveals a dichotomy of protein turnover.Potential contribution of exosomes to the prion-like propagation of lesions in Alzheimer's disease.A novel origin for granulovacuolar degeneration in aging and Alzheimer's disease: parallels to stress granules.Antimicrobial peptide β-defensin-1 expression is upregulated in Alzheimer's brainLysosomal fusion dysfunction as a unifying hypothesis for Alzheimer's disease pathology.Brain pathology in myotonic dystrophy: when tauopathy meets spliceopathy and RNAopathy.Granulovacuolar degeneration: a neurodegenerative change that accompanies tau pathology.Phosphorylation of the amyloid β-peptide at Ser26 stabilizes oligomeric assembly and increases neurotoxicity.Granulovacuolar Degeneration in Hippocampus of Neurodegenerative Diseases: Quantitative Study.Aβ accumulation causes MVB enlargement and is modelled by dominant negative VPS4A.Pathology of clinical and preclinical Alzheimer's disease.Antioxidative and Neuroprotective Effects of Curcumin in an Alzheimer's Disease Rat Model Co-Treated with Intracerebroventricular Streptozotocin and Subcutaneous D-Galactose.mTOR Hyperactivation in down syndrome hippocampus appears early during development.Acute spinal cord injury could cause activation of autophagy in dorsal root ganglia.Expression of neurodegenerative disease-related proteins and caspase-3 in glioneuronal tumours.Rare autosomal copy number variations in early-onset familial Alzheimer's disease.TMEM230 Accumulation in Granulovacuolar Degeneration Bodies and Dystrophic Neurites of Alzheimer's Disease.Rab10 Phosphorylation is a Prominent Pathological Feature in Alzheimer's Disease.The identification of raft-derived tau-associated vesicles that are incorporated into immature tangles and paired helical filaments.: An Etiologic Agent for Late-Onset Dementia
P2860
Q27003314-E5BC796D-9D89-4C8F-A33C-30D13441386CQ27004724-EDC708BF-4934-4899-9990-FE06DC1409F6Q27026907-4FD9484B-C6F1-4ED8-9096-D50CB9D72674Q28088631-9AD35CF0-7D54-4B6C-835F-9C88DE14A5E3Q33715568-7D6A5D28-EFBB-44EA-BE33-AD8F0B608AE1Q33746240-92AB4BB2-38E8-4008-B506-7B5213F52EB8Q34071708-EC38814B-A91A-4869-90B5-C90B653C0A23Q34983893-02E5A7E4-1125-430F-B37F-E4B4A6C9355DQ35060401-8C388328-EE54-4BE9-B1DA-B313CAF19982Q35233549-912D38C5-7AD1-467E-8DB9-AFA5F17D5894Q35643978-3A272BF0-9FF7-4768-B8CB-2A490317DCF5Q35711947-D4FF99FB-0BC7-4125-B6E2-0A1492DA8E8DQ36076343-B2AE7CB6-2040-4795-92DB-F630F32E7B0EQ36190490-BF962E3C-1A78-423F-91AD-53D14FCDBD48Q37280709-99D4A738-F026-47C4-BEE3-1A0C870B8C32Q38042636-A21A8AAF-9317-40C8-8122-B24150322522Q38177257-2CDA3F69-8D2F-4645-9922-9D8190C25DF8Q38803351-36D95619-71E2-49AD-9EA5-FF5D16EB8727Q38959054-5AF203EF-A395-4986-AFF9-8DC6FB296200Q39008745-3332AA25-B8D5-448E-80B6-7AC55994EB71Q41506935-52ADC87A-6171-4A6C-87BA-79B0C4A1FAE4Q45353164-8D1075FD-1CFF-4A5D-8097-6B554B08B03AQ46707452-9DB5625B-D7FF-459B-9070-097498B964C4Q48721008-1AD7C84B-6845-4616-A0CC-C38BCDD1DEADQ50231758-2D2742DD-84F4-41CB-B033-B5DE7A379EC8Q50669172-E107FED5-BA5D-45B6-8903-D0A22C616600Q50920611-7BDC26A0-F5F3-4A81-A212-EA5565D99B66Q50990939-093E6900-AD73-490B-A4F7-D5D2EB098694Q52644610-61F32517-7541-429E-AE2E-629114F80ACFQ53481840-44A75343-9F5D-44F4-8B35-ED519F0BB323Q57816571-98A1CE16-D90F-409F-921A-0CF885C67F8D
P2860
Granulovacuolar degeneration (GVD) bodies of Alzheimer's disease (AD) resemble late-stage autophagic organelles.
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@ast
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@en
Granulovacuolar degeneration
@nl
type
label
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@ast
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@en
Granulovacuolar degeneration
@nl
prefLabel
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@ast
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@en
Granulovacuolar degeneration
@nl
P2093
P2860
P1476
Granulovacuolar degeneration ( ...... e-stage autophagic organelles.
@en
P2093
P2860
P304
P356
10.1111/J.1365-2990.2010.01135.X
P577
2011-04-01T00:00:00Z