Increased natural CD4+CD25+ regulatory T cells and their suppressor activity do not contribute to mortality in murine polymicrobial sepsis. - Wikidata - wikimedia - TriplyDB

Increased natural CD4+CD25+ regulatory T cells and their suppressor activity do not contribute to mortality in murine polymicrobial sepsis.

Increased natural CD4+CD25+ regulatory T cells and their suppressor activity do not contribute to mortality in murine polymicrobial sepsis.

http://www.wikidata.org/entity/Q34572664

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Trauma equals danger--damage control by the immune system The role of regulatory T cells in the pathogenesis of sepsis and its clinical implication The immunopathology of sepsis: pathogen recognition, systemic inflammation, the compensatory anti-inflammatory response, and regulatory T cells Foxp3+ regulatory T cells are required for recovery from severe sepsis Neutrophil mobilization from the bone marrow during polymicrobial sepsis is dependent on CXCL12 signaling.Type I interferon signaling in hematopoietic cells is required for survival in mouse polymicrobial sepsis by regulating CXCL10.The relationship between CD4+CD25+CD127- regulatory T cells and inflammatory response and outcome during shock states.The post sepsis-induced expansion and enhanced function of regulatory T cells create an environment to potentiate tumor growth.Systemic treatment with CpG-B after sublethal rickettsial infection induces mouse death through indoleamine 2,3-dioxygenase (IDO).Baicalin improves survival in a murine model of polymicrobial sepsis via suppressing inflammatory response and lymphocyte apoptosis.Increased percentages of T helper cells producing IL-17 and monocytes expressing markers of alternative activation in patients with sepsis Quantification of regulatory T cells in septic patients by real-time PCR-based methylation assay and flow cytometry Treatment with GITR agonistic antibody corrects adaptive immune dysfunction in sepsis.Monitoring immune dysfunctions in the septic patient: a new skin for the old ceremony.Increased circulating regulatory T cells (CD4(+)CD25 (+)CD127 (-)) contribute to lymphocyte anergy in septic shock patients.Potential role of myeloid cell/eosinophil-derived IL-17 in LPS-induced endotoxin shock Heterotrimeric Gα(i) proteins are regulated by lipopolysaccharide and are anti-inflammatory in endotoxemia and polymicrobial sepsis Adoptive transfer of bone marrow-derived dendritic cells decreases inhibitory and regulatory T-cell differentiation and improves survival in murine polymicrobial sepsis.Cecal ligation and puncture.Regulatory T cells are protective in systemic inflammation response syndrome induced by zymosan in mice Epigenetic regulation of immune cell functions during post-septic immunosuppression.Full activation of CD4+ T cells early during sepsis requires specific antigen.Cholinergic regulatory lymphocytes re-establish neuromodulation of innate immune responses in sepsis B cells enhance early innate immune responses during bacterial sepsis.The role of regulatory T cells in immune dysfunction during sepsis.Critical role for CXC ligand 10/CXC receptor 3 signaling in the murine neonatal response to sepsis.CCR4 Controls the Suppressive Effects of Regulatory T Cells on Early and Late Events during Severe Sepsis.A Detailed Characterization of the Dysfunctional Immunity and Abnormal Myelopoiesis Induced by Severe Shock and Trauma in the Aged.Genome-level longitudinal expression of signaling pathways and gene networks in pediatric septic shock.MyD88-dependent expansion of an immature GR-1(+)CD11b(+) population induces T cell suppression and Th2 polarization in sepsis Differential alterations of tissue T-cell subsets after sepsis.STAT3-mediated IL-17 production by postseptic T cells exacerbates viral immunopathology of the lung.Sepsis-induced immune dysfunction: can immune therapies reduce mortality?A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.Innate lymphocyte subsets and their immunoregulatory roles in burn injury and sepsis.Adenosine promotes Foxp3 expression in Treg cells in sepsis model by activating JNK/AP-1 pathway.Persistent inflammation and immunosuppression: a common syndrome and new horizon for surgical intensive care Differential lymphopenia-induced homeostatic proliferation for CD4+ and CD8+ T cells following septic injury Sepsis induces early alterations in innate immunity that impact mortality to secondary infection.CD4+ lymphocytes control gut epithelial apoptosis and mediate survival in sepsis.

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P2860

Increased natural CD4+CD25+ regulatory T cells and their suppressor activity do not contribute to mortality in murine polymicrobial sepsis.

http://www.wikidata.org/entity/Q34572664

description

2006 nî lūn-bûn

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2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... n murine polymicrobial sepsis.

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Increased natural CD4+CD25+ re ...... in murine polymicrobial sepsis

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Kerri A O'Malley

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Kindra M Kelly

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Lyle L Moldawer

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Mark A Atkinson

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Matthew J Delano

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Michael J Clare Salzler

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Philip A Efron

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Philip O Scumpia

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Priscilla F McAuliffe

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Ricardo Ungaro

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