Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.

Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.

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http://www.wikidata.org/entity/Q34598816
Gammaherpesviruses and pulmonary fibrosis: evidence from humans, horses, and rodentsViruses in Idiopathic Pulmonary Fibrosis. Etiology and ExacerbationFollowing the path of CCL2 from prostaglandins to periostin in lung fibrosisγ-Herpes virus-68, but not Pseudomonas aeruginosa or influenza A (H1N1), exacerbates established murine lung fibrosisExperimental induction of pulmonary fibrosis in horses with the gammaherpesvirus equine herpesvirus 5.Severe gammaherpesvirus-induced pneumonitis and fibrosis in syngeneic bone marrow transplant mice is related to effects of transforming growth factor-β.CD8+ T Cell Response to Gammaherpesvirus Infection Mediates Inflammation and Fibrosis in Interferon Gamma Receptor-Deficient MiceRight place, right time: the evolving role of herpesvirus infection as a "second hit" in idiopathic pulmonary fibrosisPulmonary fibrosis induced by γ-herpesvirus in aged mice is associated with increased fibroblast responsiveness to transforming growth factor-β.Current and novel drug therapies for idiopathic pulmonary fibrosis.Nanoparticle exposure reactivates latent herpesvirus and restores a signature of acute infection.TLR9-induced interferon β is associated with protection from gammaherpesvirus-induced exacerbation of lung fibrosis.Epstein-Barr virus infection induces aberrant TLR activation pathway and fibroblast-myofibroblast conversion in sclerodermaPlasma membrane wounding and repair in pulmonary diseases.Loss of CCR2 signaling alters leukocyte recruitment and exacerbates γ-herpesvirus-induced pneumonitis and fibrosis following bone marrow transplantation.The anti-fibrotic effect of inhibition of TGFβ-ALK5 signalling in experimental pulmonary fibrosis in mice is attenuated in the presence of concurrent γ-herpesvirus infection.Efferocytosis of apoptotic alveolar epithelial cells is sufficient to initiate lung fibrosis
P2860

Latent infection by γherpesvirus stimulates profibrotic mediator release from multiple cell types.

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http://www.wikidata.org/entity/Q34598816
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2010 nî lūn-bûn
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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
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2010年の論文
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2010年学术文章
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2010年学术文章
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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Latent infection by γherpesvir ...... ease from multiple cell types.
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10.1152/AJPLUNG.00028.2010
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