Direct measurement of free radical generation following reperfusion of ischemic myocardium
about
The Role of Mitochondrial Reactive Oxygen Species in Cardiovascular Injury and Protective StrategiesMitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningAtomic structures of wild-type and thermostable mutant recombinant human Cu,Zn superoxide dismutaseAction mechanism and cardiovascular effect of anthocyanins: a systematic review of animal and human studiesUnderstanding and preventing mitochondrial oxidative damagePast and present course of cardioprotection against ischemia-reperfusion injuryMitochondrial dynamics in diabetic cardiomyopathyPotential implication of the chemical properties and bioactivity of nitrone spin traps for therapeuticsApparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxideMeasurement of Reactive Oxygen Species, Reactive Nitrogen Species, and Redox-Dependent Signaling in the Cardiovascular System: A Scientific Statement From the American Heart Association.Subcellular Energetics and Metabolism: A Cross-Species Framework.Coordinated behavior of mitochondria in both space and time: a reactive oxygen species-activated wave of mitochondrial depolarization.Glutathione oxidation as a trigger of mitochondrial depolarization and oscillation in intact hearts.Suppression of re-entrant and multifocal ventricular fibrillation by the late sodium current blocker ranolazineStructure-redox-relaxivity relationships for redox responsive manganese-based magnetic resonance imaging probes.The targeting of cyclophilin D by RNAi as a novel cardioprotective therapy: evidence from two-photon imaging.Signal transduction of nitric oxide donor-induced protection in hydrogen peroxide-mediated apoptosis in H9C2 cardiomyoblasts.Behavior of Ca(2+) waves in multicellular preparations from guinea pig ventricle.Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis.Myocardial ischemia and reperfusion: direct evidence for free radical generation by electron spin resonance spectroscopy.Measurement of endothelial cell free radical generation: evidence for a central mechanism of free radical injury in postischemic tissues.Nonanticoagulant heparin reduces myocyte Na+ and Ca2+ loading during simulated ischemia and decreases reperfusion injury.Transient rise in serum interleukin-8 concentration during acute myocardial infarction.Electron paramagnetic resonance measurements of free radicals in the intact beating heart: a technique for detection and characterization of free radicals in whole biological tissues.Superoxide radical anion adduct of 5,5-dimethyl-1-pyrroline N-oxide. 6. Redox propertiesDirect measurement of nitroxide pharmacokinetics in isolated hearts situated in a low-frequency electron spin resonance spectrometer: implications for spin trapping and in vivo oxymetryCellular stress responses: cell survival and cell deathHighly stable dendritic trityl radicals as oxygen and pH probe.Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion.Impaired intestinal mucosal barrier upon ischemia-reperfusion: "patching holes in the shield with a simple surgical method".Perivascular demyelination and intramyelinic oedema in reperfusion nerve injury.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Cardiac mitochondria and reactive oxygen species generationConformational dynamics of bovine Cu, Zn superoxide dismutase revealed by time-resolved fluorescence spectroscopy of the single tyrosine residueDemonstration of free radical generation in the "stunned" myocardium in the conscious dog and identification of major differences between conscious and open-chest dogs.A myocardial ischemia- and reperfusion-induced injury is mediated by reactive oxygen species released from blood platelets.Nitroxide stable radicals protect beating cardiomyocytes against oxidative damageReactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.Reperfusion injury induces apoptosis in rabbit cardiomyocytes.Glycolytic inhibition and calcium overload as consequences of exogenously generated free radicals in rabbit hearts.
P2860
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P2860
Direct measurement of free radical generation following reperfusion of ischemic myocardium
description
1987 nî lūn-bûn
@nan
1987 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1987 թվականի մարտին հրատարակված գիտական հոդված
@hy
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
name
Direct measurement of free rad ...... rfusion of ischemic myocardium
@ast
Direct measurement of free rad ...... rfusion of ischemic myocardium
@en
Direct measurement of free rad ...... rfusion of ischemic myocardium
@nl
type
label
Direct measurement of free rad ...... rfusion of ischemic myocardium
@ast
Direct measurement of free rad ...... rfusion of ischemic myocardium
@en
Direct measurement of free rad ...... rfusion of ischemic myocardium
@nl
prefLabel
Direct measurement of free rad ...... rfusion of ischemic myocardium
@ast
Direct measurement of free rad ...... rfusion of ischemic myocardium
@en
Direct measurement of free rad ...... rfusion of ischemic myocardium
@nl
P2093
P2860
P356
P1476
Direct measurement of free rad ...... rfusion of ischemic myocardium
@en
P2093
Flaherty JT
Weisfeldt ML
P2860
P304
P356
10.1073/PNAS.84.5.1404
P407
P577
1987-03-01T00:00:00Z