Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury. - Wikidata - wikimedia - TriplyDB

Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury.

Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury.

http://www.wikidata.org/entity/Q47202919

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Overexpression of human copper, zinc-superoxide dismutase (SOD1) prevents postischemic injury T-0162, a novel free radical scavenger, reduces myocardial infarct size in rabbits Congestive heart failure. New frontiers Blood cardioplegia.The delivery of superoxide dismutase encapsulated in polyketal microparticles to rat myocardium and protection from myocardial ischemia-reperfusion injury.Cardioprotection against ischemia/reperfusion by licochalcone B in isolated rat hearts Reperfusion injury.Oxygen radicals generated at reflow induce peroxidation of membrane lipids in reperfused hearts.Direct measurement of free radical generation following reperfusion of ischemic myocardium 3',4'-Dihydroxyflavonol reduces infarct size and injury associated with myocardial ischaemia and reperfusion in sheep.Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury An inhibitor of poly (ADP-ribose) synthetase activity reduces contractile dysfunction and preserves high energy phosphate levels during reperfusion of the ischaemic rat heart.Assessment of myocardial viability using coronary zero flow pressure after successful angioplasty in patients with acute anterior myocardial infarction Recombinant superoxide dismutase reduces oxygen free radical concentrations in reperfused myocardium.Ischemia and reperfusion: effect of fructose-1,6-bisphosphate.Cardioprotection against ischemia/reperfusion injury by QiShenYiQi Pill® via ameliorate of multiple mitochondrial dysfunctions.Allopurinol and amlodipine improve coronary vasodilatation after myocardial ischaemia and reperfusion in anaesthetized dogs.Impaired endothelium-dependent relaxation of dog coronary arteries after myocardial ischaemia and reperfusion: prevention by amlodipine, propranolol and allopurinol Fibrin(ogen) and its fragments in the pathophysiology and treatment of myocardial infarction.The no-reflow phenomenon: A basic mechanism of myocardial ischemia and reperfusion.Inhibition of NADPH Oxidase Mediates Protective Effect of Cardiotonic Pills against Rat Heart Ischemia/Reperfusion Injury.Oxygen-derived free radicals and myocardial reperfusion injury: an overview.Effect of citrate phosphate dextrose solution on reperfusion injury in coronary artery bypass surgical patients undergoing cardiopulmonary bypass To prevent, protect and save the ischemic heart: antioxidants revisited.Tetracyclines: a pleitropic family of compounds with promising therapeutic properties. Review of the literature.Role of complement and perspectives for intervention in ischemia-reperfusion damage.Oxygen-derived free radicals and postischemic myocardial reperfusion: therapeutic implications.Biochemical events associated with rapid cellular damage during the oxygen- and calcium-paradoxes of the mammalian heart.Myocardial ischemia and reperfusion: the role of oxygen radicals in tissue injury.Oxygen and reperfusion damage: an overview.Relationships between structure and effects of ACE inhibitors: comparative effects in myocardial ischaemic/reperfusion injury.Oxygen free radical-mediated heart injury in animal models and during bypass surgery in humans. Effects of alpha-tocopherol.The role of HIFs in ischemia-reperfusion injury.Activated neutrophils release mediators that may contribute to myocardial injury and dysfunction associated with ischemia and reperfusion.Coronary reperfusion following experimental myocardial infarction.Hyperoxia during early reperfusion does not increase ischemia/reperfusion injury.Leukocyte CD11a expression and granulocyte activation during experimental myocardial ischemia and long lasting reperfusion.Is thrombolysis alone the best therapy for acute myocardial infarction? Current status and emerging strategies Targeted intracellular catalase delivery protects neonatal rat myocytes from hypoxia-reoxygenation and ischemia-reperfusion injury.Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes.

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P2860

Reduction in experimental infarct size by recombinant human superoxide dismutase: insights into the pathophysiology of reperfusion injury.

http://www.wikidata.org/entity/Q47202919

description

1986 nî lūn-bûn

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1986年の論文

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1986年学术文章

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1986年学术文章

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1986年学术文章

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1986年学术文章

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1986年学术文章

@zh-my

1986年学术文章

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1986年學術文章

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1986年學術文章

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name

Reduction in experimental infa ...... siology of reperfusion injury.

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Reduction in experimental infa ...... siology of reperfusion injury.

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type

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Reduction in experimental infa ...... siology of reperfusion injury.

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Reduction in experimental infa ...... siology of reperfusion injury.

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Reduction in experimental infa ...... siology of reperfusion injury.

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Reduction in experimental infa ...... siology of reperfusion injury.

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Ambrosio G

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pathophysiology

reperfusion injury