Hemodynamic basis for glomerular injury in rats with desoxycorticosterone-salt hypertension. - Wikidata - wikimedia - TriplyDB

Hemodynamic basis for glomerular injury in rats with desoxycorticosterone-salt hypertension.

Hemodynamic basis for glomerular injury in rats with desoxycorticosterone-salt hypertension.

http://www.wikidata.org/entity/Q34610189

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Modulation of Immunity and Inflammation by the Mineralocorticoid Receptor and Aldosterone Renal autoregulation in health and disease Relationship of and cross-talk between physical and biologic properties of the glomerulus Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass Therapeutic advantage of converting enzyme inhibitors in arresting progressive renal disease associated with systemic hypertension in the rat Biochemical and Cellular Determinants of Renal Glomerular Elasticity Intrarenal hemodynamics in primary aldosteronism before and after treatment.Ontogenetic aspects of hypertension development: analysis in the rat.Glomerular injury in uninephrectomized spontaneously hypertensive rats. A consequence of glomerular capillary hypertension.Dietary protein restriction in established renal injury in the rat. Selective role of glomerular capillary pressure in progressive glomerular dysfunction.Control of glomerular hypertension by insulin administration in diabetic rats.Diseased renal glomeruli are getting soft. Focus on "Biophysical properties of normal and diseased renal glomeruli"Biophysical properties of normal and diseased renal glomeruli.Glomerular sclerosis is prevented during urinary tract obstruction due to podocyte protection Longitudinal study of living kidney donor glomerular dynamics after nephrectomy.Serum phosphorus predicts incident chronic kidney disease and end-stage renal disease Chronic glucocorticoid therapy amplifies glomerular injury in rats with renal ablation.Nitric oxide and angiotensin II. Glomerular and tubular interaction in the rat The association of predonation hypertension with glomerular function and number in older living kidney donors.Prevention of 11-deoxycorticosterone-salt-induced glomerular hypertrophy and glomerulosclerosis by dietary phosphate binder A simple indentation device for measuring micrometer-scale tissue stiffness.Role of aldosterone in the remnant kidney model in the rat.Predominance of hemodynamic rather than metabolic factors in the pathogenesis of diabetic glomerulopathy Cardiovascular and renal damage in primary aldosteronism: outcomes after treatment.The podocyte slit diaphragm--from a thin grey line to a complex signalling hub.A potential role for mechanical forces in the detachment of podocytes and the progression of CKD.Treatment of Primary Aldosteronism and Organ Protection.Stressed podocytes-mechanical forces, sensors, signaling and response.The Gordon Wilson lecture. Why kidneys fail: an unifying hypothesis.The aging kidney: structure, function, mechanisms, and therapeutic implications.The benefits of ACE inhibitors and calcium antagonists in slowing progressive renal failure: focus on fixed-dose combination antihypertensive therapy.Effect of prolonged administration of a urinary kinase inhibitor, ebelactone B on the development of deoxycorticosterone acetate-salt hypertension in rats.Cytoskeletal changes in podocytes associated with foot process effacement in Masugi nephritis.Complex glomerular pathology of thrombotic microangiopathy and focal segmental glomerulosclerosis forms tumor-like mass in a renal transplant donor with severe renovascular hypertension.Applied pressure enhances cell proliferation through mitogen-activated protein kinase activation in mesangial cells.The alpha8 integrin chain affords mechanical stability to the glomerular capillary tuft in hypertensive glomerular disease.Anti-hypertension effects of traditional Chinese medicine ju-ling-tang on renal hypertensive rats.Role of fibrillin-1 in hypertensive and diabetic glomerular disease.Relationship between subtle urinary albumin excretion and risk of incident hypertension: modification by glomerular filtration rate.NO inhibits stretch-induced MAPK activity by cytoskeletal disruption.

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Hemodynamic basis for glomerular injury in rats with desoxycorticosterone-salt hypertension.

http://www.wikidata.org/entity/Q34610189

description

1984 nî lūn-bûn

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1984 թուականի Մայիսին հրատարակուած գիտական յօդուած

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1984 թվականի մայիսին հրատարակված գիտական հոդված

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1984年の論文

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1984年学术文章

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1984年学术文章

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1984年学术文章

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1984年学术文章

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1984年学术文章

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1984年學術文章

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name

Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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prefLabel

Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

@en

Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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Journal of Clinical Investigation

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Hemodynamic basis for glomerul ...... ticosterone-salt hypertension.

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B M Brenner

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H G Rennke

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L D Dworkin

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T H Hostetter

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P2860

Malignant Hypertension Produced by Treatment with Desoxycorticosterone Acetate and Sodium Chloride.

Steroid-induced hypertension in the rat. A microangiographic and histologic study on the pathogenesis of hypertensive vascular and glomerular lesions.

Contraction of cultured rat glomerular cells of apparent mesangial origin after stimulation with angiotensin II and arginine vasopressin

Mechanism of reduced glomerular filtration rate in chronic malnutrition

Determination of serum protein concentration in nanoliter blood samples using fluorescamine or 9-phthalaldehyde.

Protein overload nephropathy in rats with unilateral nephrectomy. A correlative light immunogluorescence and electron microscopical analysis.

A simple method of estimating progression of chronic renal failure.

Dietary restrictions early and late: effects on the nephropathy of the NZB X NZW mouse.

Malignant hypertension resulting from deoxycorticosterone acetate and salt excess: role of renin and sodium in vascular changes.

Renal lesions and proteinuria in the spontaneously hypertensive rat made normotensive by treatment.

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1448-1461

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10.1172/JCI111349

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5

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73

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6715546

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arterial hypertension

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425168

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