Predominance of hemodynamic rather than metabolic factors in the pathogenesis of diabetic glomerulopathy
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An Update on the Use of Animal Models in Diabetic Nephropathy ResearchRenal autoregulation in health and diseaseTherapeutic advantage of converting enzyme inhibitors in arresting progressive renal disease associated with systemic hypertension in the ratSpironolactone ameliorates podocytic adhesive capacity via restoring integrin alpha 3 expression in streptozotocin-induced diabetic ratsAnemia lessens and its prevention with recombinant human erythropoietin worsens glomerular injury and hypertension in rats with reduced renal mass.Dyslipoproteinemia and impairment of renal function in diabetic kidney disease: an analysis of animal studies, observational studies, and clinical trials.Towards understanding the kallikrein-kinin system: insights from measurement of kinin peptides.Glomerular function in Pima Indians with noninsulin-dependent diabetes mellitus of recent onsetAngiotensin converting enzyme inhibition ameliorates glomerular filtration of macromolecules and water and lessens glomerular injury in the ratHaemodynamics in microvascular complications in type 1 diabetes.Effect of a low-protein diet supplemented with ketoacids on skeletal muscle atrophy and autophagy in rats with type 2 diabetic nephropathyEstablishment of a diabetic mouse model with progressive diabetic nephropathy.Paradoxes of nitric oxide in the diabetic kidney.Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice.SOD1, but not SOD3, deficiency accelerates diabetic renal injury in C57BL/6-Ins2(Akita) diabetic miceElevated plasma atrial natriuretic peptide levels in diabetic rats. Potential mediator of hyperfiltrationChronic glucocorticoid therapy amplifies glomerular injury in rats with renal ablation.Functional response of healthy and diseased glomeruli to a large, protein-rich meal.Kidney function in early diabetes: the tubular hypothesis of glomerular filtration.Nitric oxide and angiotensin II. Glomerular and tubular interaction in the ratIntraglomerular pressure and mesangial stretching stimulate extracellular matrix formation in the rat.Glucose-induced downregulation of angiotensin II and arginine vasopressin receptors in cultured rat aortic vascular smooth muscle cells. Role of protein kinase C.Spontaneously reduced blood pressure load in the rat streptozotocin-induced diabetes model: potential pathogenetic relevance.Renal hyperfiltration related to diabetes mellitus and obesity in human disease.Prevention of diabetic nephropathy with enalapril in normotensive diabetics with microalbuminuria.Transient impairment of dynamic renal autoregulation in early diabetes mellitus in rats.Deleterious effects of calcium channel blockade on pressure transmission and glomerular injury in rat remnant kidneys.Cyclic stretching force selectively up-regulates transforming growth factor-beta isoforms in cultured rat mesangial cellsDifferential proteome profiling using iTRAQ in microalbuminuric and normoalbuminuric type 2 diabetic patientsInfluence of Acute High Glucose on Protein Abundance Changes in Murine Glomerular Mesangial Cells.Renal function and hemodynamic study in obese Zucker rats.Urinary markers of glomerular injury in diabetic nephropathyExpression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy.Increased urinary Smad3 is significantly correlated with glomerular hyperfiltration and a reduced glomerular filtration rate and is a new urinary biomarker for diabetic nephropathy.Differential urinary glycoproteome analysis of type 2 diabetic nephropathy using 2D-LC-MS/MS and iTRAQ quantification.High risk of ESRD in type 1 diabetes: new strategies are needed to retard progressive renal function declineOrgan-based response to exercise in type 1 diabetes.Fractalkine and its receptor mediate extracellular matrix accumulation in diabetic nephropathy in miceThe diabetic patient with hypertension.Prevention of diabetic glomerulopathy by pharmacological amelioration of glomerular capillary hypertension.
P2860
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P2860
Predominance of hemodynamic rather than metabolic factors in the pathogenesis of diabetic glomerulopathy
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 1985
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Predominance of hemodynamic ra ...... sis of diabetic glomerulopathy
@en
Predominance of hemodynamic ra ...... is of diabetic glomerulopathy.
@nl
type
label
Predominance of hemodynamic ra ...... sis of diabetic glomerulopathy
@en
Predominance of hemodynamic ra ...... is of diabetic glomerulopathy.
@nl
prefLabel
Predominance of hemodynamic ra ...... sis of diabetic glomerulopathy
@en
Predominance of hemodynamic ra ...... is of diabetic glomerulopathy.
@nl
P2093
P2860
P356
P1476
Predominance of hemodynamic ra ...... sis of diabetic glomerulopathy
@en
P2093
P2860
P304
P356
10.1073/PNAS.82.17.5963
P407
P577
1985-09-01T00:00:00Z