Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers. - Wikidata - wikimedia - TriplyDB

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

http://www.wikidata.org/entity/Q34610562

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Tumor angiogenesis: pericytes and maturation are not to be ignored Direct Interaction between Carcinoma Cells and Cancer Associated Fibroblasts for the Regulation of Cancer Invasion Insights into Orphan Nuclear Receptors as Prognostic Markers and Novel Therapeutic Targets for Breast Cancer Interactions between tumor cells and microenvironment in breast cancer: a new opportunity for targeted therapy Relationship of mammographic density and gene expression: analysis of normal breast tissue surrounding breast cancer.Weight Loss Reversed Obesity-Induced HGF/c-Met Pathway and Basal-Like Breast Cancer Progression Cabozantinib (XL184) Inhibits Growth and Invasion of Preclinical TNBC Models.Distinctive responsiveness to stromal signaling accompanies histologic grade programming of cancer cells Systematic bias in genomic classification due to contaminating non-neoplastic tissue in breast tumor samples.Differential gene expression between African American and European American colorectal cancer patients.Malignant stroma increases luminal breast cancer cell proliferation and angiogenesis through platelet-derived growth factor signaling Probing biological nanotopology via diffusion of weakly constrained plasmonic nanorods with optical coherence tomography Obesity-mediated regulation of HGF/c-Met is associated with reduced basal-like breast cancer latency in parous mice.Longitudinal study of mammary epithelial and fibroblast co-cultures using optical coherence tomography reveals morphological hallmarks of pre-malignancy.Parity-related molecular signatures and breast cancer subtypes by estrogen receptor status Thymic stromal lymphopoietin fosters human breast tumor growth by promoting type 2 inflammation Benign breast tissue composition in breast cancer patients: association with risk factors, clinical variables, and gene expression Systematic assessment of prognostic gene signatures for breast cancer shows distinct influence of time and ER status.Interactions among genes, tumor biology and the environment in cancer health disparities: examining the evidence on a national and global scale Breast cancer subtype-specific interactions with the microenvironment dictate mechanisms of invasion.Choosing the right cell line for breast cancer research Gene expression analysis of in vitro cocultures to study interactions between breast epithelium and stroma.Tumor intrinsic subtype is reflected in cancer-adjacent tissue.Fibroblasts Influence Survival and Therapeutic Response in a 3D Co-Culture Model Extensive rewiring of epithelial-stromal co-expression networks in breast cancer Characteristic Gene Expression Profiles of Human Fibroblasts and Breast Cancer Cells in a Newly Developed Bilateral Coculture System The HSP90 Inhibitor Ganetespib Radiosensitizes Human Lung Adenocarcinoma Cells.Basal breast cancer: a complex and deadly molecular subtype Laser Direct-Write Onto Live Tissues: A Novel Model for Studying Cancer Cell Migration.LIN28A Modulates Splicing and Gene Expression Programs in Breast Cancer Cells.Decreased Expression of TMEM173 Predicts Poor Prognosis in Patients with Hepatocellular Carcinoma Gene expression in extratumoral microenvironment predicts clinical outcome in breast cancer patients.Analysis of terminal duct lobular unit involution in luminal A and basal breast cancers.Blood Genome-Wide Transcriptional Profiles of HER2 Negative Breast Cancers Patients Inverse-power-law behavior of cellular motility reveals stromal-epithelial cell interactions in 3D co-culture by OCT fluctuation spectroscopy cMET inhibitor crizotinib impairs angiogenesis and reduces tumor burden in the C3(1)-Tag model of basal-like breast cancer Basal-like breast cancer cells induce phenotypic and genomic changes in macrophages.Imaging Extracellular Matrix Remodeling In Vitro by Diffusion-Sensitive Optical Coherence Tomography.Impact of tumor microenvironment and epithelial phenotypes on metabolism in breast cancer Fibroblasts as architects of cancer pathogenesis

P2860

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P2860

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

http://www.wikidata.org/entity/Q34610562

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年学术文章

@wuu

2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

name

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@ast

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@en

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@nl

type

label

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@ast

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@en

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@nl

prefLabel

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@ast

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@en

Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers.

@nl

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1541-7786.MCR-10-0372

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Molecular Cancer Research

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Interactions with fibroblasts are distinct in Basal-like and luminal breast cancers

@en

P2093

Delisha A Stewart

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Erick Roman-Perez

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Fathi Elloumi

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J Terese Camp

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Jessica Rein

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Melissa A Troester

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P2860

Significance analysis of microarrays applied to the ionizing radiation response

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

Activation of host wound responses in breast cancer microenvironment

Tumor-endothelial interaction links the CD44(+)/CD24(-) phenotype with poor prognosis in early-stage breast cancer

Tumor self-seeding by circulating cancer cells

Determination of stromal signatures in breast carcinoma.

In vitro analysis of the invasive phenotype of SUM 149, an inflammatory breast cancer cell line

Prediction of toxicant-specific gene expression signatures after chemotherapeutic treatment of breast cell lines

Transient tumor-fibroblast interactions increase tumor cell malignancy by a TGF-Beta mediated mechanism in a mouse xenograft model of breast cancer

Gene expression profiling predicts clinical outcome of breast cancer

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3-13

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scholarly article

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10.1158/1541-7786.MCR-10-0372

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1

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9

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Charles M. Perou

J Chuck Harrell

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2010-12-03T00:00:00Z

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21131600

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3045848

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