Transcriptional repression by the orphan steroid receptor RVR/Rev-erb beta is dependent on the signature motif and helix 5 in the E region: functional evidence for a biological role of RVR in myogenesis.

Transcriptional repression by the orphan steroid receptor RVR/Rev-erb beta is dependent on the signature motif and helix 5 in the E region: functional evidence for a biological role of RVR in myogenesis.

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http://www.wikidata.org/entity/Q34610873

Q34610873

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Exogenous expression of a dominant negative RORalpha1 vector in muscle cells impairs differentiation: RORalpha1 directly interacts with p300 and myoD A zinc finger HIT domain-containing protein, ZNHIT-1, interacts with orphan nuclear hormone receptor Rev-erbbeta and removes Rev-erbbeta-induced inhibition of apoCIII transcription Two receptor interaction domains in the corepressor, N-CoR/RIP13, are required for an efficient interaction with Rev-erbA alpha and RVR: physical association is dependent on the E region of the orphan receptors The orphan nuclear receptor, COUP-TF II, inhibits myogenesis by post-transcriptional regulation of MyoD function: COUP-TF II directly interacts with p300 and myoD Identification of heme as the ligand for the orphan nuclear receptors REV-ERBalpha and REV-ERBbeta.Circadian clock regulation of skeletal muscle growth and repair The Structural Basis of Gas-Responsive Transcription by the Human Nuclear Hormone Receptor REV-ERBβ Nuclear receptor Rev-erbalpha: a heme receptor that coordinates circadian rhythm and metabolism Nuclear hormone receptors for heme: REV-ERBalpha and REV-ERBbeta are ligand-regulated components of the mammalian clock.Rev-Erb co-regulates muscle regeneration via tethered interaction with the NF-Y cistrome Orphan nuclear hormone receptor Rev-erbalpha regulates the human apolipoprotein CIII promoter.RORα suppresses breast tumor invasion by inducing SEMA3F expression The orphan Rev-erb nuclear receptors: a link between metabolism, circadian rhythm and inflammation?The corepressor N-CoR and its variants RIP13a and RIP13Delta1 directly interact with the basal transcription factors TFIIB, TAFII32 and TAFII70.Retinoid-related Orphan Receptors (RORs): Roles in Cellular Differentiation and Development.The REV-ERBs and RORs: molecular links between circadian rhythms and lipid homeostasis.The steroid receptor coactivator, GRIP-1, is necessary for MEF-2C-dependent gene expression and skeletal muscle differentiation.Orphan nuclear receptors: therapeutic opportunities in skeletal muscle.Nuclear receptor Rev-erbα: up, down, and all around.Skeletal muscle functions around the clock.The hepatic circadian clock regulates the choline kinase α gene through the BMAL1-REV-ERBα axis.Domain-Specific Monoclonal Antibodies Against Human Rev-erbβ.Pharmacological inhibition of REV-ERB stimulates differentiation, inhibits turnover and reduces fibrosis in dystrophic muscle.Transcriptional programming of lipid and amino acid metabolism by the skeletal muscle circadian clock

P2860

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P2860

Transcriptional repression by the orphan steroid receptor RVR/Rev-erb beta is dependent on the signature motif and helix 5 in the E region: functional evidence for a biological role of RVR in myogenesis.

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http://www.wikidata.org/entity/Q34610873

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1996 nî lūn-bûn

@nan

1996 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

1996年の論文

@ja

1996年論文

@yue

1996年論文

@zh-hant

1996年論文

@zh-hk

1996年論文

@zh-mo

1996年論文

@zh-tw

1996年论文

@wuu