Genetic analysis of Caenorhabditis elegans glp-1 mutants suggests receptor interaction or competition.
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The Tumor Suppressor BCL7B Functions in the Wnt Signaling PathwayGLD-4-mediated translational activation regulates the size of the proliferative germ cell pool in the adult C. elegans germ lineIncreasing Notch signaling antagonizes PRC2-mediated silencing to promote reprograming of germ cells into neuronsInsulin signaling promotes germline proliferation in C. elegans.A conserved tetraspanin subfamily promotes Notch signaling in Caenorhabditis elegans and in human cells.The C. elegans VAPB homolog VPR-1 is a permissive signal for gonad development.Cancer models in Caenorhabditis elegans.Piwi and piRNAs act upstream of an endogenous siRNA pathway to suppress Tc3 transposon mobility in the Caenorhabditis elegans germline.Basic Caenorhabditis elegans methods: synchronization and observation.New positive regulators of lin-12 activity in Caenorhabditis elegans include the BRE-5/Brainiac glycosphingolipid biosynthesis enzyme.Caenorhabditis elegans atx-2 promotes germline proliferation and the oocyte fate.Autosomal genes of autosomal/X-linked duplicated gene pairs and germ-line proliferation in Caenorhabditis elegans.sel-7, a positive regulator of lin-12 activity, encodes a novel nuclear protein in Caenorhabditis elegansPRP-17 and the pre-mRNA splicing pathway are preferentially required for the proliferation versus meiotic development decision and germline sex determination in Caenorhabditis elegans.C. elegans CEP-1/p53 and BEC-1 are involved in DNA repair.hecd-1 modulates notch activity in Caenorhabditis elegans.EMB-4: a predicted ATPase that facilitates lin-12 activity in Caenorhabditis elegansA model of stem cell population dynamics: in silico analysis and in vivo validation.Non-autonomous DAF-16/FOXO activity antagonizes age-related loss of C. elegans germline stem/progenitor cellsEvidence for functional redundancy between C. elegans ADAM proteins SUP-17/Kuzbanian and ADM-4/TACETargeting Homologous Recombination in Notch-Driven C. elegans Stem Cell and Human Tumors.S6K links cell fate, cell cycle and nutrient response in C. elegans germline stem/progenitor cellsA role for Caenorhabditis elegans chromatin-associated protein HIM-17 in the proliferation vs. meiotic entry decisionNotch signaling is antagonized by SAO-1, a novel GYF-domain protein that interacts with the E3 ubiquitin ligase SEL-10 in Caenorhabditis elegansSoma-germline interactions that influence germline proliferation in Caenorhabditis elegans.lin-35/Rb and the CoREST ortholog spr-1 coordinately regulate vulval morphogenesis and gonad development in C. elegansPUF-8 Functions Redundantly with GLD-1 to Promote the Meiotic Progression of Spermatocytes in Caenorhabditis elegansTEG-1 CD2BP2 regulates stem cell proliferation and sex determination in the C. elegans germ line and physically interacts with the UAF-1 U2AF65 splicing factorGenetic Screen Reveals Link between the Maternal Effect Sterile Gene mes-1 and Pseudomonas aeruginosa-induced Neurodegeneration in Caenorhabditis elegans.Mechano-logical model of C. elegans germ line suggests feedback on the cell cycle.Nucleotide levels regulate germline proliferation through modulating GLP-1/Notch signaling in C. elegans.Cell cycle features of C. elegans germline stem/progenitor cells vary temporally and spatially.Sensory regulation of the C. elegans germline through TGF-β-dependent signaling in the niche.Proteasomal regulation of the proliferation vs. meiotic entry decision in the Caenorhabditis elegans germ lineUBR-5, a Conserved HECT-Type E3 Ubiquitin Ligase, Negatively Regulates Notch-Type Signaling in Caenorhabditis elegansC. elegans RNA-binding proteins PUF-8 and MEX-3 function redundantly to promote germline stem cell mitosis.A "latent niche" mechanism for tumor initiationCharacterization of the Caenorhabditis elegans Islet LIM-homeodomain ortholog, lim-7.METT-10, a putative methyltransferase, inhibits germ cell proliferative fate in Caenorhabditis elegans.Fat metabolism links germline stem cells and longevity in C. elegans.
P2860
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P2860
Genetic analysis of Caenorhabditis elegans glp-1 mutants suggests receptor interaction or competition.
description
2003 nî lūn-bûn
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2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Genetic analysis of Caenorhabd ...... or interaction or competition.
@ast
Genetic analysis of Caenorhabd ...... or interaction or competition.
@en
Genetic analysis of Caenorhabd ...... or interaction or competition.
@nl
type
label
Genetic analysis of Caenorhabd ...... or interaction or competition.
@ast
Genetic analysis of Caenorhabd ...... or interaction or competition.
@en
Genetic analysis of Caenorhabd ...... or interaction or competition.
@nl
prefLabel
Genetic analysis of Caenorhabd ...... or interaction or competition.
@ast
Genetic analysis of Caenorhabd ...... or interaction or competition.
@en
Genetic analysis of Caenorhabd ...... or interaction or competition.
@nl
P2860
P1433
P1476
Genetic analysis of Caenorhabd ...... tor interaction or competition
@en
P2093
Anita S-R Pepper
Darrell J Killian
P2860
P304
P407
P577
2003-01-01T00:00:00Z