DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition - Wikidata - wikimedia - TriplyDB

DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition

DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition

http://www.wikidata.org/entity/Q34619321

about

DCLK1 is a broadly dysregulated target against epithelial-mesenchymal transition, focal adhesion, and stemness in clear cell renal carcinoma Epigenetic changes and alternate promoter usage by human colon cancers for expressing DCLK1-isoforms: Clinical Implications Inflammatory and oncogenic roles of a tumor stem cell marker doublecortin-like kinase (DCLK1) in virus-induced chronic liver diseases Dclk1+ small intestinal epithelial tuft cells display the hallmarks of quiescence and self-renewal Doublecortin-like kinase 1 expression associates with breast cancer with neuroendocrine differentiation.Regulatory Roles of Dclk1 in Epithelial Mesenchymal Transition and Cancer Stem Cells Survival of Patients with Gastrointestinal Cancers Can Be Predicted by a Surrogate microRNA Signature for Cancer Stem-like Cells Marked by DCLK1 Kinase.Dclk1, a tumor stem cell marker, regulates pro-survival signaling and self-renewal of intestinal tumor cells.Plasma DCLK1 is a marker of hepatocellular carcinoma (HCC): Targeting DCLK1 prevents HCC tumor xenograft growth via a microRNA-dependent mechanism.Roles of Wnt Target Genes in the Journey of Cancer Stem Cells Characterizing the molecular features of ERG-positive tumors in primary and castration resistant prostate cancer.Doublecortin and CaM kinase-like-1 as an independent prognostic factor in patients with resected pancreatic carcinoma.Pancreatic DCLK1 marks quiescent but oncogenic progenitors: a possible link to neuroendocrine tumors.Commensal-infected macrophages induce dedifferentiation and reprogramming of epithelial cells during colorectal carcinogenesis.DCLK1 Expression in Colorectal Polyps Increases with the Severity of Dysplasia.Lgr5High/DCLK1High phenotype is more common in early stage and intestinal subtypes of gastric carcinomas.Association of doublecortin-like kinase 1 with tumor aggressiveness and poor biochemical recurrence-free survival in prostate cancer.Colorectal cancer: genetic abnormalities, tumor progression, tumor heterogeneity, clonal evolution and tumor-initiating cells.Doublecortin-like kinase 1 compromises DNA repair and induces chromosomal instability

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DCLK1 facilitates intestinal tumor growth via enhancing pluripotency and epithelial mesenchymal transition

http://www.wikidata.org/entity/Q34619321

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@ast

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@en

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@nl

type

label

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@ast

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@en

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@nl

prefLabel

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@ast

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@en

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@nl

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ONCOTARGET.2393

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P1476

DCLK1 facilitates intestinal t ...... thelial mesenchymal transition

@en

P2093

Courtney W Houchen

http://www.w3.org/2001/XMLSchema#string

Dongfeng Qu

http://www.w3.org/2001/XMLSchema#string

Harisha R Chinthalapally

http://www.w3.org/2001/XMLSchema#string

Naushad Ali

http://www.w3.org/2001/XMLSchema#string

Randal May

http://www.w3.org/2001/XMLSchema#string

Shahid Umar

http://www.w3.org/2001/XMLSchema#string

Stan A Lightfoot

http://www.w3.org/2001/XMLSchema#string

P2860

The epithelial-mesenchymal transition generates cells with properties of stem cells

MicroRNAs as regulators of differentiation and cell fate decisions.

Novel changes in NF-{kappa}B activity during progression and regression phases of hyperplasia: role of MEK, ERK, and p38.

Long-lived intestinal tuft cells serve as colon cancer-initiating cells.

Identification of a novel putative gastrointestinal stem cell and adenoma stem cell marker, doublecortin and CaM kinase-like-1, following radiation injury and in adenomatous polyposis coli/multiple intestinal neoplasia mice.

DCAMKL-1 regulates epithelial-mesenchymal transition in human pancreatic cells through a miR-200a-dependent mechanism.

p53 regulates epithelial-mesenchymal transition and stem cell properties through modulating miRNAs.

Fluvastatin interferes with hepatitis C virus replication via microtubule bundling and a doublecortin-like kinase-mediated mechanism.

Differential effects of β-catenin and NF-κB interplay in the regulation of cell proliferation, inflammation and tumorigenesis in response to bacterial infection

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9269-9280

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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10.18632/ONCOTARGET.2393

http://www.w3.org/2001/XMLSchema#string

P407

P433

19

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P478

5

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Sripathi Sureban

Parthasarathy Chandrakesan

Nathaniel Weygant

P577

2014-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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265559707

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25211188

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4253433

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