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Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxiaCardiac hypertrophy involves both myocyte hypertrophy and hyperplasia in anemic zebrafishHexokinase II knockdown results in exaggerated cardiac hypertrophy via increased ROS productionGender and post-ischemic recovery of hypertrophied rat heartsEfficacy and safety of a metabolic modulator drug in chronic stable angina: review of evidence from clinical trials.Astragaloside IV alleviates heart failure via activating PPARα to switch glycolysis to fatty acid β-oxidation.Responses of hypertrophied myocytes to reactive species: implications for glycolysis and electrophile metabolismMetabolomic analysis of pressure-overloaded and infarcted mouse hearts.Substrate-enzyme competition attenuates upregulated anaplerotic flux through malic enzyme in hypertrophied rat heart and restores triacylglyceride content: attenuating upregulated anaplerosis in hypertrophy.PPARγ-induced cardiolipotoxicity in mice is ameliorated by PPARα deficiency despite increases in fatty acid oxidation.Cardiac-specific inhibition of kinase activity in calcium/calmodulin-dependent protein kinase kinase-β leads to accelerated left ventricular remodeling and heart failure after transverse aortic constriction in mice.Constitutively active calcineurin induces cardiac endoplasmic reticulum stress and protects against apoptosis that is mediated by alpha-crystallin-B.Transverse aortic constriction leads to accelerated heart failure in mice lacking PPAR-gamma coactivator 1alpha.Myocardial hypertrophy and the maturation of fatty acid oxidation in the newborn human heart.Mitochondrial adaptations to physiological vs. pathological cardiac hypertrophy.Maternal treatment with agonistic autoantibodies against type-1 angiotensin II receptor in late pregnancy increases apoptosis of myocardial cells and myocardial susceptibility to ischemia-reperfusion injury in offspring rats.PGC-1β deficiency accelerates the transition to heart failure in pressure overload hypertrophy.Endurance training or beta-blockade can partially block the energy metabolism remodeling taking place in experimental chronic left ventricle volume overloadInhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload.Impaired mitochondrial biogenesis precedes heart failure in right ventricular hypertrophy in congenital heart disease.Mouse SIRT3 attenuates hypertrophy-related lipid accumulation in the heart through the deacetylation of LCADCardioplegic strategies to protect the hypertrophic heart during cardiac surgery.Recruitment of NADH shuttling in pressure-overloaded and hypertrophic rat hearts.Hyperinsulinemic Normoglycemia Does Not Meaningfully Improve Myocardial Performance during Cardiac Surgery: A Randomized Trial.Male-Specific Cardiac Dysfunction in CTP:Phosphoethanolamine Cytidylyltransferase (Pcyt2)-Deficient MiceMitochondrial energy metabolism in heart failure: a question of balance.Integrated glycoprotein immobilization method for glycopeptide and glycan analysis of cardiac hypertrophy.Trimetazidine attenuates pressure overload-induced early cardiac energy dysfunction via regulation of neuropeptide Y system in a rat model of abdominal aortic constriction.Role of monosaccharide transport proteins in carbohydrate assimilation, distribution, metabolism, and homeostasis.Comparison of 18F-FDG uptake by right ventricular myocardium in idiopathic pulmonary arterial hypertension and pulmonary arterial hypertension associated with congenital heart disease.Cardiac hypertrophy, substrate utilization and metabolic remodelling: cause or effect?Ranolazine, a novel agent for chronic stable angina.AMPK alterations in cardiac physiology and pathology: enemy or ally?AMP-Activated Protein Kinase Signalling in Cancer and Cardiac Hypertrophy.The absence of endogenous lipid oxidation in early stage heart failure exposes limits in lipid storage and turnoverCardiomyocyte autophagy: metabolic profit and loss.The Failing Heart Relies on Ketone Bodies as a Fuel.Potential impact of carbohydrate and fat intake on pathological left ventricular hypertrophy.Mitochondrial protein hyperacetylation in the failing heartCardiac metabolic compensation to hypertension requires lipoprotein lipase
P2860
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P2860
description
2002 nî lūn-bûn
@nan
2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Energy metabolism in the hypertrophied heart.
@ast
Energy metabolism in the hypertrophied heart.
@en
Energy metabolism in the hypertrophied heart.
@nl
type
label
Energy metabolism in the hypertrophied heart.
@ast
Energy metabolism in the hypertrophied heart.
@en
Energy metabolism in the hypertrophied heart.
@nl
prefLabel
Energy metabolism in the hypertrophied heart.
@ast
Energy metabolism in the hypertrophied heart.
@en
Energy metabolism in the hypertrophied heart.
@nl
P2093
P356
P1476
Energy metabolism in the hypertrophied heart.
@en
P2093
Gary D Lopaschuk
Michael F Allard
Nandakumar Sambandam
Roger W Brownsey
P2888
P304
P356
10.1023/A:1015380609464
P577
2002-04-01T00:00:00Z
P6179
1029742382