The absence of endogenous lipid oxidation in early stage heart failure exposes limits in lipid storage and turnover
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Cardiac energy metabolic alterations in pressure overload-induced left and right heart failure (2013 Grover Conference Series)Optimization of cardiac metabolism in heart failureMultiphasic triacylglycerol dynamics in the intact heart during acute in vivo overexpression of CD36Imaging myocardial metabolic remodeling.Metabolic Remodeling in Diabetic Cardiomyopathy.SERCA1 expression enhances the metabolic efficiency of improved contractility in post-ischemic heart.Assessment of myocardial triglyceride oxidation with PET and 11C-palmitateSubstrate-enzyme competition attenuates upregulated anaplerotic flux through malic enzyme in hypertrophied rat heart and restores triacylglyceride content: attenuating upregulated anaplerosis in hypertrophy.Dietary fat supply to failing hearts determines dynamic lipid signaling for nuclear receptor activation and oxidation of stored triglyceride.Delineation of substrate selection and anaplerosis in tricarboxylic acid cycle of the heart by 13C NMR spectroscopy and mass spectrometry.Metabolic efficiency promotes protection from pressure overload in hearts expressing slow skeletal troponin I.Resolving confounding enrichment kinetics due to overlapping resonance signals from 13C-enriched long chain fatty acid oxidation and uptake within intact hearts.Myocardial ATGL overexpression decreases the reliance on fatty acid oxidation and protects against pressure overload-induced cardiac dysfunctionSexual dimorphism in cardiac triacylglyceride dynamics in mice on long term caloric restriction.Enhancing Cardiac Triacylglycerol Metabolism Improves Recovery From Ischemic Stress.Ventricular assist device implantation corrects myocardial lipotoxicity, reverses insulin resistance, and normalizes cardiac metabolism in patients with advanced heart failureAcute liver carnitine palmitoyltransferase I overexpression recapitulates reduced palmitate oxidation of cardiac hypertrophyNovel proteins associated with human dilated cardiomyopathy: selective reduction in α(1A)-adrenergic receptors and increased desensitization proteins.The transcriptional coactivator PGC-1alpha is essential for maximal and efficient cardiac mitochondrial fatty acid oxidation and lipid homeostasisCardiac lipotoxicity: molecular pathways and therapeutic implications.Cardiac metabolic compensation to hypertension requires lipoprotein lipaseAcyl CoA synthetase-1 links facilitated long chain fatty acid uptake to intracellular metabolic trafficking differently in hearts of male versus female mice.Limited functional and metabolic improvements in hypertrophic and healthy rat heart overexpressing the skeletal muscle isoform of SERCA1 by adenoviral gene transfer in vivo.Energy metabolism in heart failure and remodelling.Assessing Cardiac Metabolism: A Scientific Statement From the American Heart Association.Lipid Use and Misuse by the Heart.Metabolic therapy at the crossroad: how to optimize myocardial substrate utilization?Metabolic crosstalk between the heart and liver impacts familial hypertrophic cardiomyopathy.Metabolomics as a tool for cardiac research.Alterations in mitochondrial function in cardiac hypertrophy and heart failure.Cardiac metabolism and its interactions with contraction, growth, and survival of cardiomyocytes.Recent advances in metabolic imaging.Matrix revisited: mechanisms linking energy substrate metabolism to the function of the heart.Dietary management of heart failure: room for improvement?Peroxisome proliferator-activated receptors in cardiac energy metabolism and cardiovascular disease.Peroxisome proliferator-activated receptor-α expression induces alterations in cardiac myofilaments in a pressure-overload model of hypertrophy.Metabolism in cardiomyopathy: every substrate matters.Sucrose Nonfermenting-Related Kinase Enzyme-Mediated Rho-Associated Kinase Signaling is Responsible for Cardiac Function.Metabolic remodelling in hypertrophied and failing myocardium: a review.Effects of dietary phosphate on glucose and lipid metabolism.
P2860
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P2860
The absence of endogenous lipid oxidation in early stage heart failure exposes limits in lipid storage and turnover
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
The absence of endogenous lipi ...... in lipid storage and turnover
@ast
The absence of endogenous lipi ...... in lipid storage and turnover
@en
type
label
The absence of endogenous lipi ...... in lipid storage and turnover
@ast
The absence of endogenous lipi ...... in lipid storage and turnover
@en
prefLabel
The absence of endogenous lipi ...... in lipid storage and turnover
@ast
The absence of endogenous lipi ...... in lipid storage and turnover
@en
P2093
P2860
P1476
The absence of endogenous lipi ...... in lipid storage and turnover
@en
P2093
Aaron D Fields
E Douglas Lewandowski
J Michael O'Donnell
Natalia Sorokina
P2860
P304
P356
10.1016/J.YJMCC.2007.11.006
P577
2007-11-24T00:00:00Z