Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis. - Wikidata - wikimedia - TriplyDB

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

http://www.wikidata.org/entity/Q34626985

about

Staphylococcus aureus α-hemolysin mediates virulence in a murine model of severe pneumonia through activation of the NLRP3 inflammasome In the critically ill patient, diabetes predicts mortality independent of statin therapy but is not associated with acute lung injury: a cohort study The impact of diabetes on the pathogenesis of sepsis Glyburide reduces bacterial dissemination in a mouse model of melioidosis Cell death and autophagy in tuberculosis Sulphonylurea usage in melioidosis is associated with severe disease and suppressed immune response Glibenclamide Prevents Diabetes in NOD Mice Inhibition of the nucleotide-binding domain, leucine-rich containing family, pyrin-domain containing 3 inflammasome reduces the severity of experimentally induced acute pancreatitis in obese mice.Host responses to melioidosis and tuberculosis are both dominated by interferon-mediated signaling.Association of melioidosis incidence with rainfall and humidity, Singapore, 2003-2012.Diabetes-independent increase of factor VII-activating protease activation in patients with Gram-negative sepsis (melioidosis).Glibenclamide decreases ATP-induced intracellular calcium transient elevation via inhibiting reactive oxygen species and mitochondrial activity in macrophages.Elimination of the NLRP3-ASC inflammasome protects against chronic obesity-induced pancreatic damage Pentoxifylline reduces the inflammatory process in diabetic rats: relationship with decreases of pro-inflammatory cytokines and inducible nitric oxide synthase T-Cell Responses Are Associated with Survival in Acute Melioidosis Patients.Glutathione deficiency in type 2 diabetes impairs cytokine responses and control of intracellular bacteria.Burkholderia pseudomallei triggers altered inflammatory profiles in a whole-blood model of type 2 diabetes-melioidosis comorbidity The Blood Transcriptome of Experimental Melioidosis Reflects Disease Severity and Shows Considerable Similarity with the Human Disease Association between Use of Oral Anti-Diabetic Drugs and the Risk of Sepsis: A Nested Case-Control Study Neutrophil extracellular traps in the host defense against sepsis induced by Burkholderia pseudomallei (melioidosis)Bioelectric modulation of macrophage polarization.Mechanisms of disease: inflammasome activation and the development of type 2 diabetes.Short-Term High-Fat Diet (HFD) Induced Anxiety-Like Behaviors and Cognitive Impairment Are Improved with Treatment by Glyburide.Metformin and other glucose-lowering drug initiation and rates of community-based antibiotic use and hospital-treated infections in patients with type 2 diabetes: a Danish nationwide population-based cohort study Glibenclamide impairs responses of neutrophils against Burkholderia pseudomallei by reduction of intracellular glutathione.Therapeutic Administration of a Monoclonal Anti-Il-1β Antibody Protects Against Experimental Melioidosis.Glibenclamide reduces pro-inflammatory cytokine production by neutrophils of diabetes patients in response to bacterial infection The Nlrp3 inflammasome promotes age-related thymic demise and immunosenescence Current status of inflammasome blockers as anti-inflammatory drugs.Mechanisms of antibiotic resistance in Burkholderia pseudomallei: implications for treatment of melioidosis.Regulating against the dysregulation: new treatment options in autoinflammation.Role of Canonical and Non-canonical Inflammasomes During Burkholderia Infection.Programmed death ligand 1 on Burkholderia pseudomallei-infected human polymorphonuclear neutrophils impairs T cell functions.NLRP3 inflammasome activation is essential for paraquat-induced acute lung injury.Interleukin 10 inhibits pro-inflammatory cytokine responses and killing of Burkholderia pseudomallei A Protective Role of Glibenclamide in Inflammation-Associated Injury.Expression and function of transforming growth factor β in melioidosis.Delayed activation of host innate immune pathways in streptozotocin-induced diabetic hosts leads to more severe disease during infection with Burkholderia pseudomallei.Glyburide and retinoic acid synergize to promote wound healing by anti-inflammation and RIP140 degradation.Mitigating the Impact of Antibacterial Drug Resistance through Host-Directed Therapies: Current Progress, Outlook, and Challenges.

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P2860

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

http://www.wikidata.org/entity/Q34626985

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@ast

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@en

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@nl

type

label

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@ast

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@en

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@nl

prefLabel

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@ast

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@en

Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@nl

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Clinical Infectious Diseases

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Glyburide is anti-inflammatory and associated with reduced mortality in melioidosis.

@en

P2093

Gavin C K W Koh

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Gordon Dougan

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M Fernanda Schreiber

http://www.w3.org/2001/XMLSchema#string

Rapeephan R Maude

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Sue J Lee

http://www.w3.org/2001/XMLSchema#string

W Joost Wiersinga

http://www.w3.org/2001/XMLSchema#string

Weera Mahavanakul

http://www.w3.org/2001/XMLSchema#string

Wipada Chaowagul

http://www.w3.org/2001/XMLSchema#string

Wirongrong Chierakul

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P2860

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Role and significance of quantitative urine cultures in diagnosis of melioidosis.

ICAM-1 regulates neutrophil adhesion and transcellular migration of TNF-alpha-activated vascular endothelium under flow

Fallibility in estimating direct effects

Editorial: sulfonylureas differ in effects on ischemic preconditioning--is it time to retire glyburide?

The sulphonylurea glibenclamide inhibits multidrug resistance protein (MRP1) activity in human lung cancer cells

The acute respiratory distress syndrome

The inflammasome recognizes cytosolic microbial and host DNA and triggers an innate immune response

Risk factors for melioidosis and bacteremic melioidosis.

Stimulation of rat erythrocyte P2X7 receptor induces the release of epoxyeicosatrienoic acids.

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717-725

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scholarly article

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10.1093/CID/CIQ192

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6

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52

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Nicholas John White

Nicholas P J Day

Vanaporn Wuthiekanun

Direk Limmathurotsakul

Tom van der Poll

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2011-02-03T00:00:00Z

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49809520

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21293047

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3049341

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